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Items: 1 to 20 of 230

1.
2.

Exacerbation of intestinal permeability in rats after a two-hit injury: burn and Enterococcus faecalis infection.

Samonte VA, Goto M, Ravindranath TM, Fazal N, Holloway VM, Goyal A, Gamelli RL, Sayeed MM.

Crit Care Med. 2004 Nov;32(11):2267-73.

PMID:
15640640
3.

Enterococcus faecalis exacerbates burn injury-induced host responses in rats.

Goto M, Samonte V, Khan M, Haque F, Goyal A, Al-Ghoul W, Raziuddin S, Fazal N, Ravindranath T, Reed RL, Gamelli RL, Sayeed MM.

Shock. 2002 Dec;18(6):523-8.

PMID:
12462560
4.

Differential effector responses by circulating/blood and tissue/peritoneal neutrophils following burn combined with Enterococcus faecalis infection.

Fazal N, Shelip A, Siddiqui E, Ali A, Azim AC, Al-Ghoul WM.

FEMS Immunol Med Microbiol. 2012 Mar;64(2):191-204. doi: 10.1111/j.1574-695X.2011.00881.x.

5.

Effects of burn with and without Escherichia coli infection in rats on intestinal vs. splenic T-cell responses.

Ravindranath T, Al-Ghoul W, Namak S, Fazal N, Durazo-Arvizu R, Choudhry M, Sayeed MM.

Crit Care Med. 2001 Dec;29(12):2245-50.

PMID:
11801815
6.
7.

Burn injury exacerbates hemodynamic and metabolic responses in rats with polymicrobial sepsis.

Goto M, Samonte V, Ravindranath T, Sayeed MM, Gamelli RL.

J Burn Care Res. 2006 Jan-Feb;27(1):50-9.

PMID:
16566537
8.
9.

Increased susceptibility to infection related to extent of burn injury.

Yurt RW, McManus AT, Mason AD Jr, Pruitt BA Jr.

Arch Surg. 1984 Feb;119(2):183-8.

PMID:
6421264
10.

Signaling mechanisms of elevated neutrophil O2- generation after burn injury.

Sabeh F, Hockberger P, Sayeed MM.

Am J Physiol. 1998 Feb;274(2 Pt 2):R476-85.

11.

[Typical bacteria in an intensive care burn unit in severely burned patients and their importance with regard to mortality: retrospective study 1995 - 2004].

Steinsträsser L, Thies AH, Rabstein S, Steinau HU.

Handchir Mikrochir Plast Chir. 2007 Oct;39(5):338-44. German.

PMID:
17985278
12.
13.

Aspiration pneumonia-induced sepsis increases cardiac dysfunction after burn trauma.

Sheeran PW, Maass DL, White DJ, Turbeville TD, Giroir BP, Horton JW.

J Surg Res. 1998 May;76(2):192-9.

PMID:
9698522
14.

Neutrophil depletion in rats reduces burn-injury induced intestinal bacterial translocation.

Fazal N, Shamim M, Khan SS, Gamelli RL, Sayeed MM.

Crit Care Med. 2000 May;28(5):1550-5.

PMID:
10834710
15.

[Reproduction of a rat model of burn with infection].

Zhang DW, Gong ZY, Peng YZ.

Zhonghua Shao Shang Za Zhi. 2011 Apr;27(2):104-8. Chinese.

PMID:
21651846
16.

The effects of early excision and grafting on myocardial inflammation and function after burn injury.

Horton JW, Sanders B, White DJ, Maass DL.

J Trauma. 2006 Nov;61(5):1069-77.

PMID:
17099511
17.

Evidence of the proinflammatory role of Enterococcus faecalis in polymicrobial peritonitis in rats.

Montravers P, Mohler J, Saint Julien L, Carbon C.

Infect Immun. 1997 Jan;65(1):144-9.

18.

Insulin attenuates the cytokine response in a burn wound infection model.

Przkora R, Herndon DN, Finnerty CC, Jeschke MG.

Shock. 2007 Feb;27(2):205-8.

PMID:
17224797
19.

Burn plasma transfer induces burn edema in healthy rats.

Kremer T, Abé D, Weihrauch M, Peters C, Gebhardt MM, Germann G, Heitmann C, Walther A.

Shock. 2008 Oct;30(4):394-400. doi: 10.1097/SHK.0b013e3181673908.

PMID:
18323747
20.

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