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Items: 1 to 20 of 105


Nitric oxide attenuates IGF-I-induced aortic smooth muscle cell motility by decreasing Rac1 activity: essential role of PTP-PEST and p130cas.

Ceacareanu AC, Ceacareanu B, Zhuang D, Chang Y, Ray RM, Desai L, Chapman KE, Waters CM, Hassid A.

Am J Physiol Cell Physiol. 2006 Apr;290(4):C1263-70. Epub 2005 Dec 14.


Nitric oxide-induced inhibition of aortic smooth muscle cell motility: role of PTP-PEST and adaptor proteins p130cas and Crk.

Lin Y, Ceacareanu AC, Hassid A.

Am J Physiol Heart Circ Physiol. 2003 Aug;285(2):H710-21. Epub 2003 Apr 24.


Essential role of protein kinase G and decreased cytoplasmic Ca2+ levels in NO-induced inhibition of rat aortic smooth muscle cell motility.

Zhuang D, Ceacareanu AC, Ceacareanu B, Hassid A.

Am J Physiol Heart Circ Physiol. 2005 Apr;288(4):H1859-66. Epub 2004 Dec 2.


Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H2O2 levels: essential role of cGMP.

Zhuang D, Ceacareanu AC, Lin Y, Ceacareanu B, Dixit M, Chapman KE, Waters CM, Rao GN, Hassid A.

Am J Physiol Heart Circ Physiol. 2004 Jun;286(6):H2103-12. Epub 2004 Jan 29. Erratum in: Am J Physiol Heart Circ Physiol. 2004 Aug;287(2):H983.


Regulation of fibroblast motility by the protein tyrosine phosphatase PTP-PEST.

Garton AJ, Tonks NK.

J Biol Chem. 1999 Feb 5;274(6):3811-8.


Combination of gene targeting and substrate trapping to identify substrates of protein tyrosine phosphatases using PTP-PEST as a model.

Côté JF, Charest A, Wagner J, Tremblay ML.

Biochemistry. 1998 Sep 22;37(38):13128-37.


Insulin-like growth factor-I induces reactive oxygen species production and cell migration through Nox4 and Rac1 in vascular smooth muscle cells.

Meng D, Lv DD, Fang J.

Cardiovasc Res. 2008 Nov 1;80(2):299-308. doi: 10.1093/cvr/cvn173. Epub 2008 Jun 20.


PTP-PEST controls motility through regulation of Rac1.

Sastry SK, Lyons PD, Schaller MD, Burridge K.

J Cell Sci. 2002 Nov 15;115(Pt 22):4305-16.


Increase of PTP levels in vascular injury and in cultured aortic smooth muscle cells treated with specific growth factors.

Chang Y, Zhuang D, Zhang C, Hassid A.

Am J Physiol Heart Circ Physiol. 2004 Nov;287(5):H2201-8. Epub 2004 Jul 22. Erratum in: Am J Physiol Heart Circ Physiol. 2006 Apr;290(4):H1727.


Mechanisms related to NO-induced motility in differentiated rat aortic smooth muscle cells.

Pu Q, Zhuang D, Thakran S, Hassid A.

Am J Physiol Heart Circ Physiol. 2011 Jan;300(1):H101-8. doi: 10.1152/ajpheart.00342.2010. Epub 2010 Oct 29.


Regulatory effects of nitric oxide on Src kinase, FAK, p130Cas, and receptor protein tyrosine phosphatase alpha (PTP-alpha): a role for the cellular redox environment.

Curcio MF, Batista WL, Linares E, Nascimento FD, Moraes MS, Borges RE, Sap J, Stern A, Monteiro HP.

Antioxid Redox Signal. 2010 Jul 15;13(2):109-25. doi: 10.1089/ars.2009.2534.


Inhibition of the catalytic activity of cell adhesion kinase beta by protein-tyrosine phosphatase-PEST-mediated dephosphorylation.

Lyons PD, Dunty JM, Schaefer EM, Schaller MD.

J Biol Chem. 2001 Jun 29;276(26):24422-31. Epub 2001 May 3.


Protein tyrosine phosphatase-PEST regulates focal adhesion disassembly, migration, and cytokinesis in fibroblasts.

Angers-Loustau A, Côté JF, Charest A, Dowbenko D, Spencer S, Lasky LA, Tremblay ML.

J Cell Biol. 1999 Mar 8;144(5):1019-31.

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