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Items: 1 to 20 of 141

1.

Advances in understanding the molecular causes of diabetes-induced birth defects.

Loeken MR.

J Soc Gynecol Investig. 2006 Jan;13(1):2-10. Epub 2005 Nov 21.

PMID:
16303321
2.

Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects.

Chang TI, Horal M, Jain SK, Wang F, Patel R, Loeken MR.

Diabetologia. 2003 Apr;46(4):538-45. Epub 2003 Mar 26.

PMID:
12739027
3.

Hypoxic stress in diabetic pregnancy contributes to impaired embryo gene expression and defective development by inducing oxidative stress.

Li R, Chase M, Jung SK, Smith PJ, Loeken MR.

Am J Physiol Endocrinol Metab. 2005 Oct;289(4):E591-9. Epub 2005 May 31.

4.

Current perspectives on the causes of neural tube defects resulting from diabetic pregnancy.

Loeken MR.

Am J Med Genet C Semin Med Genet. 2005 May 15;135C(1):77-87. Review.

PMID:
15800853
5.

AMP-activated protein kinase mediates effects of oxidative stress on embryo gene expression in a mouse model of diabetic embryopathy.

Wu Y, Viana M, Thirumangalathu S, Loeken MR.

Diabetologia. 2012 Jan;55(1):245-54. doi: 10.1007/s00125-011-2326-y. Epub 2011 Oct 13.

6.

Oxidative stress during diabetic pregnancy disrupts cardiac neural crest migration and causes outflow tract defects.

Morgan SC, Relaix F, Sandell LL, Loeken MR.

Birth Defects Res A Clin Mol Teratol. 2008 Jun;82(6):453-63. doi: 10.1002/bdra.20457.

7.

Neural tube defects in embryos of diabetic mice: role of the Pax-3 gene and apoptosis.

Phelan SA, Ito M, Loeken MR.

Diabetes. 1997 Jul;46(7):1189-97.

PMID:
9200655
8.

Lack of metformin effect on mouse embryo AMPK activity: implications for metformin treatment during pregnancy.

Lee HY, Wei D, Loeken MR.

Diabetes Metab Res Rev. 2014 Jan;30(1):23-30. doi: 10.1002/dmrr.2451.

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12.

[Expression of Pax3 and Cx43 in NTD embryos induced by hyperglycemia].

Mao DW, Zhang YJ, Li QM, Li SR.

Zhonghua Yi Xue Za Zhi. 2003 Apr 10;83(7):593-7. Chinese.

PMID:
12887752
13.

Activation of the hexosamine pathway causes oxidative stress and abnormal embryo gene expression: involvement in diabetic teratogenesis.

Horal M, Zhang Z, Stanton R, Virkamäki A, Loeken MR.

Birth Defects Res A Clin Mol Teratol. 2004 Aug;70(8):519-27.

PMID:
15329829
14.

The green tea polyphenol EGCG alleviates maternal diabetes-induced neural tube defects by inhibiting DNA hypermethylation.

Zhong J, Xu C, Reece EA, Yang P.

Am J Obstet Gynecol. 2016 Sep;215(3):368.e1-368.e10. doi: 10.1016/j.ajog.2016.03.009. Epub 2016 Mar 12.

15.

[Study of teratogenicity of hyperglycemia on neural tube defects and antagonistic effect of taurine].

Mao DW, Zhao YP, Li SR, Che JH, Tan WH.

Zhonghua Fu Chan Ke Za Zhi. 2004 Mar;39(3):169-72. Chinese.

PMID:
15130376
16.

Use of a murine embryonic stem cell line that is sensitive to high glucose environment to model neural tube development in diabetic pregnancy.

Sanders K, Jung JH, Loeken MR.

Birth Defects Res A Clin Mol Teratol. 2014 Aug;100(8):584-91. doi: 10.1002/bdra.23281. Epub 2014 Aug 13.

17.

Transgenic mice expressing PAX3-FKHR have multiple defects in muscle development, including ectopic skeletal myogenesis in the developing neural tube.

Finckenstein FG, Davicioni E, Osborn KG, Cavenee WK, Arden KC, Anderson MJ.

Transgenic Res. 2006 Oct;15(5):595-614. Epub 2006 Sep 2.

PMID:
16952014
18.

Pax3 stimulates p53 ubiquitination and degradation independent of transcription.

Wang XD, Morgan SC, Loeken MR.

PLoS One. 2011;6(12):e29379. doi: 10.1371/journal.pone.0029379. Epub 2011 Dec 28.

19.

Free radicals and birth defects.

Loeken MR.

J Matern Fetal Neonatal Med. 2004 Jan;15(1):6-14. Review.

PMID:
15101606
20.

Global gene expression analysis of cranial neural tubes in embryos of diabetic mice.

Jiang B, Kumar SD, Loh WT, Manikandan J, Ling EA, Tay SS, Dheen ST.

J Neurosci Res. 2008 Dec;86(16):3481-93. doi: 10.1002/jnr.21800.

PMID:
18655203

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