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Items: 1 to 20 of 126

1.

Chromatin remodeling is a key mechanism underlying cocaine-induced plasticity in striatum.

Kumar A, Choi KH, Renthal W, Tsankova NM, Theobald DE, Truong HT, Russo SJ, Laplant Q, Sasaki TS, Whistler KN, Neve RL, Self DW, Nestler EJ.

Neuron. 2005 Oct 20;48(2):303-14.

2.

Drug-induced activation of dopamine D(1) receptor signaling and inhibition of class I/II histone deacetylase induce chromatin remodeling in reward circuitry and modulate cocaine-related behaviors.

Schroeder FA, Penta KL, Matevossian A, Jones SR, Konradi C, Tapper AR, Akbarian S.

Neuropsychopharmacology. 2008 Nov;33(12):2981-92. doi: 10.1038/npp.2008.15. Epub 2008 Feb 20.

3.

Increased brain-derived neurotrophic factor (BDNF) expression in the ventral tegmental area during cocaine abstinence is associated with increased histone acetylation at BDNF exon I-containing promoters.

Schmidt HD, Sangrey GR, Darnell SB, Schassburger RL, Cha JH, Pierce RC, Sadri-Vakili G.

J Neurochem. 2012 Jan;120(2):202-9. doi: 10.1111/j.1471-4159.2011.07571.x. Epub 2011 Nov 28.

4.

Delta FosB mediates epigenetic desensitization of the c-fos gene after chronic amphetamine exposure.

Renthal W, Carle TL, Maze I, Covington HE 3rd, Truong HT, Alibhai I, Kumar A, Montgomery RL, Olson EN, Nestler EJ.

J Neurosci. 2008 Jul 16;28(29):7344-9. doi: 10.1523/JNEUROSCI.1043-08.2008.

5.
6.

CREB-binding protein controls response to cocaine by acetylating histones at the fosB promoter in the mouse striatum.

Levine AA, Guan Z, Barco A, Xu S, Kandel ER, Schwartz JH.

Proc Natl Acad Sci U S A. 2005 Dec 27;102(52):19186-91.

7.

Additive effects of histone deacetylase inhibitors and amphetamine on histone H4 acetylation, cAMP responsive element binding protein phosphorylation and DeltaFosB expression in the striatum and locomotor sensitization in mice.

Shen HY, Kalda A, Yu L, Ferrara J, Zhu J, Chen JF.

Neuroscience. 2008 Dec 2;157(3):644-55. doi: 10.1016/j.neuroscience.2008.09.019. Epub 2008 Sep 16.

PMID:
18848971
8.

Activity-Regulated Cytoskeleton-Associated Protein Accumulates in the Nucleus in Response to Cocaine and Acts as a Brake on Chromatin Remodeling and Long-Term Behavioral Alterations.

Salery M, Dos Santos M, Saint-Jour E, Moumné L, Pagès C, Kappès V, Parnaudeau S, Caboche J, Vanhoutte P.

Biol Psychiatry. 2017 Apr 1;81(7):573-584. doi: 10.1016/j.biopsych.2016.05.025. Epub 2016 Jun 16.

9.

Chronic cocaine-induced H3 acetylation and transcriptional activation of CaMKIIalpha in the nucleus accumbens is critical for motivation for drug reinforcement.

Wang L, Lv Z, Hu Z, Sheng J, Hui B, Sun J, Ma L.

Neuropsychopharmacology. 2010 Mar;35(4):913-28. doi: 10.1038/npp.2009.193. Epub 2009 Dec 9.

10.

Dopamine D2-like antagonists induce chromatin remodeling in striatal neurons through cyclic AMP-protein kinase A and NMDA receptor signaling.

Li J, Guo Y, Schroeder FA, Youngs RM, Schmidt TW, Ferris C, Konradi C, Akbarian S.

J Neurochem. 2004 Sep;90(5):1117-31.

11.

Maturation of coordinated immediate early gene expression by cocaine during adolescence.

Caster JM, Kuhn CM.

Neuroscience. 2009 Apr 21;160(1):13-31. doi: 10.1016/j.neuroscience.2009.01.001. Epub 2009 Jan 7.

12.

Histone deacetylase 5 modulates the effects of social adversity in early life on cocaine-induced behavior.

Valzania A, Catale C, Viscomi MT, Puglisi-Allegra S, Carola V.

Physiol Behav. 2017 Mar 15;171:7-12. doi: 10.1016/j.physbeh.2016.12.027. Epub 2016 Dec 23.

PMID:
28025089
13.

Dynamic chromatin remodeling events in hippocampal neurons are associated with NMDA receptor-mediated activation of Bdnf gene promoter 1.

Tian F, Hu XZ, Wu X, Jiang H, Pan H, Marini AM, Lipsky RH.

J Neurochem. 2009 Jun;109(5):1375-88. doi: 10.1111/j.1471-4159.2009.06058.x. Epub 2009 Mar 23.

14.

Plasticity-associated gene Krox24/Zif268 is required for long-lasting behavioral effects of cocaine.

Valjent E, Aubier B, Corbillé AG, Brami-Cherrier K, Caboche J, Topilko P, Girault JA, Hervé D.

J Neurosci. 2006 May 3;26(18):4956-60. Erratum in: J Neurosci. 2006 May 17;26(20):table of contents.

15.

Targeted Epigenetic Remodeling of the Cdk5 Gene in Nucleus Accumbens Regulates Cocaine- and Stress-Evoked Behavior.

Heller EA, Hamilton PJ, Burek DD, Lombroso SI, Peña CJ, Neve RL, Nestler EJ.

J Neurosci. 2016 Apr 27;36(17):4690-7. doi: 10.1523/JNEUROSCI.0013-16.2016.

16.

Cocaine-induced chromatin remodeling increases brain-derived neurotrophic factor transcription in the rat medial prefrontal cortex, which alters the reinforcing efficacy of cocaine.

Sadri-Vakili G, Kumaresan V, Schmidt HD, Famous KR, Chawla P, Vassoler FM, Overland RP, Xia E, Bass CE, Terwilliger EF, Pierce RC, Cha JH.

J Neurosci. 2010 Sep 1;30(35):11735-44. doi: 10.1523/JNEUROSCI.2328-10.2010.

17.

Effects of chronic exposure to cocaine are regulated by the neuronal protein Cdk5.

Bibb JA, Chen J, Taylor JR, Svenningsson P, Nishi A, Snyder GL, Yan Z, Sagawa ZK, Ouimet CC, Nairn AC, Nestler EJ, Greengard P.

Nature. 2001 Mar 15;410(6826):376-80.

PMID:
11268215
18.

Shear stress-mediated chromatin remodeling provides molecular basis for flow-dependent regulation of gene expression.

Illi B, Nanni S, Scopece A, Farsetti A, Biglioli P, Capogrossi MC, Gaetano C.

Circ Res. 2003 Jul 25;93(2):155-61. Epub 2003 Jun 12.

20.

Repeated cocaine administration induces gene expression changes through the dopamine D1 receptors.

Zhang D, Zhang L, Tang Y, Zhang Q, Lou D, Sharp FR, Zhang J, Xu M.

Neuropsychopharmacology. 2005 Aug;30(8):1443-54.

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