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Items: 1 to 20 of 109

1.
2.

Downregulation of an astrocyte-derived inflammatory protein, S100B, reduces vascular inflammatory responses in brains persistently infected with Borna disease virus.

Ohtaki N, Kamitani W, Watanabe Y, Hayashi Y, Yanai H, Ikuta K, Tomonaga K.

J Virol. 2007 Jun;81(11):5940-8. Epub 2007 Mar 21.

3.

Borna disease virus-induced accumulation of macrophage migration inhibitory factor in rat brain astrocytes is associated with inhibition of macrophage infiltration.

Bacher M, Weihe E, Dietzschold B, Meinhardt A, Vedder H, Gemsa D, Bette M.

Glia. 2002 Mar 15;37(4):291-306.

PMID:
11870869
4.

Distribution of Borna disease virus in the brain of rats infected with an obesity-inducing virus strain.

Herden C, Herzog S, Richt JA, Nesseler A, Christ M, Failing K, Frese K.

Brain Pathol. 2000 Jan;10(1):39-48.

PMID:
10668894
5.

Astrocytes and Schwann cells are virus-host cells in the nervous system of rats with Borna disease.

Carbone KM, Trapp BD, Griffin JW, Duchala CS, Narayan O.

J Neuropathol Exp Neurol. 1989 Nov;48(6):631-44.

PMID:
2507750
6.

Cytokine expression in the rat central nervous system following perinatal Borna disease virus infection.

Sauder C, de la Torre JC.

J Neuroimmunol. 1999 Apr 1;96(1):29-45.

PMID:
10227422
7.

The central nervous system inflammatory response to neurotropic virus infection is peroxynitrite dependent.

Hooper DC, Kean RB, Scott GS, Spitsin SV, Mikheeva T, Morimoto K, Bette M, Röhrenbeck AM, Dietzschold B, Weihe E.

J Immunol. 2001 Sep 15;167(6):3470-7.

8.

Astrocytes play a key role in activation of microglia by persistent Borna disease virus infection.

Ovanesov MV, Ayhan Y, Wolbert C, Moldovan K, Sauder C, Pletnikov MV.

J Neuroinflammation. 2008 Nov 11;5:50. doi: 10.1186/1742-2094-5-50.

10.
11.

Restricted expression of Borna disease virus glycoprotein in brains of experimentally infected Lewis rats.

Werner-Keiss N, Garten W, Richt JA, Porombka D, Algermissen D, Herzog S, Baumgärtner W, Herden C.

Neuropathol Appl Neurobiol. 2008 Dec;34(6):590-602. doi: 10.1111/j.1365-2990.2008.00940.x. Epub 2008 Feb 14.

PMID:
18282160
12.

Absence of a robust innate immune response in rat neurons facilitates persistent infection of Borna disease virus in neuronal tissue.

Lin CC, Wu YJ, Heimrich B, Schwemmle M.

Cell Mol Life Sci. 2013 Nov;70(22):4399-410. doi: 10.1007/s00018-013-1402-5. Epub 2013 Jun 23.

PMID:
23793543
13.

Implications for a regulated replication of Borna disease virus in brains of experimentally infected Lewis rats.

Porombka D, Baumgärtner W, Eickmann M, Herden C.

Virus Genes. 2008 Apr;36(2):415-20. doi: 10.1007/s11262-008-0210-8. Epub 2008 Feb 14.

PMID:
18273698
14.

Expression of C1q, a subcomponent of the rat complement system, is dramatically enhanced in brains of rats with either Borna disease or experimental allergic encephalomyelitis.

Dietzschold B, Schwaeble W, Schäfer MK, Hooper DC, Zehng YM, Petry F, Sheng H, Fink T, Loos M, Koprowski H, Weihe E.

J Neurol Sci. 1995 May;130(1):11-6.

PMID:
7544401
15.

Intrinsic responses to Borna disease virus infection of the central nervous system.

Morimoto K, Hooper DC, Bornhorst A, Corisdeo S, Bette M, Fu ZF, Schäfer MK, Koprowski H, Weihe E, Dietzschold B.

Proc Natl Acad Sci U S A. 1996 Nov 12;93(23):13345-50.

16.

Changes of the organotypic retinal organization in Borna virus-infected Lewis rats.

Kacza J, Mohr C, Pannicke T, Kuhrt H, Dietzel J, Flüss M, Richt JA, Vahlenkamp TW, Stahl T, Reichenbach A, Seeger J.

J Neurocytol. 2001 Sep-Oct;30(9-10):801-20.

PMID:
12165671
20.

Chemokine gene expression in astrocytes of Borna disease virus-infected rats and mice in the absence of inflammation.

Sauder C, Hallensleben W, Pagenstecher A, Schneckenburger S, Biro L, Pertlik D, Hausmann J, Suter M, Staeheli P.

J Virol. 2000 Oct;74(19):9267-80.

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