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Hemodynamic profile, responsiveness to anandamide, and baroreflex sensitivity of mice lacking fatty acid amide hydrolase.

Pacher P, Bátkai S, Osei-Hyiaman D, Offertáler L, Liu J, Harvey-White J, Brassai A, Járai Z, Cravatt BF, Kunos G.

Am J Physiol Heart Circ Physiol. 2005 Aug;289(2):H533-41. Epub 2005 Apr 8.


Assessment of anandamide's pharmacological effects in mice deficient of both fatty acid amide hydrolase and cannabinoid CB1 receptors.

Wise LE, Shelton CC, Cravatt BF, Martin BR, Lichtman AH.

Eur J Pharmacol. 2007 Feb 14;557(1):44-8. Epub 2006 Nov 10.


Phenotypic assessment of THC discriminative stimulus properties in fatty acid amide hydrolase knockout and wildtype mice.

Walentiny DM, Vann RE, Wiley JL.

Neuropharmacology. 2015 Jun;93:237-42. doi: 10.1016/j.neuropharm.2015.02.004. Epub 2015 Feb 16.


Decreased age-related cardiac dysfunction, myocardial nitrative stress, inflammatory gene expression, and apoptosis in mice lacking fatty acid amide hydrolase.

Bátkai S, Rajesh M, Mukhopadhyay P, Haskó G, Liaudet L, Cravatt BF, Csiszár A, Ungvári Z, Pacher P.

Am J Physiol Heart Circ Physiol. 2007 Aug;293(2):H909-18. Epub 2007 Apr 13.


Haemodynamic profile and responsiveness to anandamide of TRPV1 receptor knock-out mice.

Pacher P, Bátkai S, Kunos G.

J Physiol. 2004 Jul 15;558(Pt 2):647-57. Epub 2004 Apr 30.


FAAH-/- mice display differential tolerance, dependence, and cannabinoid receptor adaptation after delta 9-tetrahydrocannabinol and anandamide administration.

Falenski KW, Thorpe AJ, Schlosburg JE, Cravatt BF, Abdullah RA, Smith TH, Selley DE, Lichtman AH, Sim-Selley LJ.

Neuropsychopharmacology. 2010 Jul;35(8):1775-87. doi: 10.1038/npp.2010.44. Epub 2010 Mar 31.


Inhibition of fatty acid amide hydrolase unmasks CB1 receptor and TRPV1 channel-mediated modulation of glutamatergic synaptic transmission in midbrain periaqueductal grey.

Kawahara H, Drew GM, Christie MJ, Vaughan CW.

Br J Pharmacol. 2011 Jul;163(6):1214-22. doi: 10.1111/j.1476-5381.2010.01157.x.


Modulation of opioids via protection of anandamide degradation by fatty acid amide hydrolase.

Haller VL, Stevens DL, Welch SP.

Eur J Pharmacol. 2008 Dec 14;600(1-3):50-8. doi: 10.1016/j.ejphar.2008.08.005. Epub 2008 Aug 20.


Cocaine- and amphetamine-related transcript is involved in the orexigenic effect of endogenous anandamide.

Osei-Hyiaman D, Depetrillo M, Harvey-White J, Bannon AW, Cravatt BF, Kuhar MJ, Mackie K, Palkovits M, Kunos G.

Neuroendocrinology. 2005;81(4):273-82. Epub 2005 Aug 29.


Anandamide transport is independent of fatty-acid amide hydrolase activity and is blocked by the hydrolysis-resistant inhibitor AM1172.

Fegley D, Kathuria S, Mercier R, Li C, Goutopoulos A, Makriyannis A, Piomelli D.

Proc Natl Acad Sci U S A. 2004 Jun 8;101(23):8756-61. Epub 2004 May 11.


The fatty acid amide hydrolase (FAAH) inhibitor PF-3845 acts in the nervous system to reverse LPS-induced tactile allodynia in mice.

Booker L, Kinsey SG, Abdullah RA, Blankman JL, Long JZ, Ezzili C, Boger DL, Cravatt BF, Lichtman AH.

Br J Pharmacol. 2012 Apr;165(8):2485-96. doi: 10.1111/j.1476-5381.2011.01445.x.


Inhibition of fatty-acid amide hydrolase accelerates acquisition and extinction rates in a spatial memory task.

Varvel SA, Wise LE, Niyuhire F, Cravatt BF, Lichtman AH.

Neuropsychopharmacology. 2007 May;32(5):1032-41. Epub 2006 Oct 18.


Mice lacking fatty acid amide hydrolase exhibit a cannabinoid receptor-mediated phenotypic hypoalgesia.

Lichtman AH, Shelton CC, Advani T, Cravatt BF.

Pain. 2004 Jun;109(3):319-27.


Endocannabinoids acting at cannabinoid-1 receptors regulate cardiovascular function in hypertension.

Bátkai S, Pacher P, Osei-Hyiaman D, Radaeva S, Liu J, Harvey-White J, Offertáler L, Mackie K, Rudd MA, Bukoski RD, Kunos G.

Circulation. 2004 Oct 5;110(14):1996-2002. Epub 2004 Sep 27.


Comparison of anandamide transport in FAAH wild-type and knockout neurons: evidence for contributions by both FAAH and the CB1 receptor to anandamide uptake.

Ortega-Gutiérrez S, Hawkins EG, Viso A, López-Rodríguez ML, Cravatt BF.

Biochemistry. 2004 Jun 29;43(25):8184-90.


Pharmacological activity of fatty acid amides is regulated, but not mediated, by fatty acid amide hydrolase in vivo.

Lichtman AH, Hawkins EG, Griffin G, Cravatt BF.

J Pharmacol Exp Ther. 2002 Jul;302(1):73-9.


Fatty acid amide hydrolase: an emerging therapeutic target in the endocannabinoid system.

Cravatt BF, Lichtman AH.

Curr Opin Chem Biol. 2003 Aug;7(4):469-75. Review.


Attenuation of experimental autoimmune hepatitis by exogenous and endogenous cannabinoids: involvement of regulatory T cells.

Hegde VL, Hegde S, Cravatt BF, Hofseth LJ, Nagarkatti M, Nagarkatti PS.

Mol Pharmacol. 2008 Jul;74(1):20-33. doi: 10.1124/mol.108.047035. Epub 2008 Apr 3.

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