Similar articles for PMID: 15781338

Year	Number of Results
1990	1
1993	1
1994	4
1995	4
1996	8
1997	1
1998	3
1999	8
2000	9
2001	6
2002	14
2003	19
2004	28
2005	26
2006	9
2007	13
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2009	7
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2011	7
2012	11
2013	13
2014	4
2015	9
2016	9
2017	6
2018	10
2019	9
2020	7
2021	4
2022	4
2023	9
2024	0

1

Src family tyrosine kinases are activated by Flt3 and are involved in the proliferative effects of leukemia-associated Flt3 mutations.

Robinson LJ, Xue J, Corey SJ. Robinson LJ, et al. Exp Hematol. 2005 Apr;33(4):469-79. doi: 10.1016/j.exphem.2005.01.004. Exp Hematol. 2005. PMID: 15781338 Free article.

2

Overexpression and constitutive activation of FLT3 induces STAT5 activation in primary acute myeloid leukemia blast cells.

Spiekermann K, Bagrintseva K, Schwab R, Schmieja K, Hiddemann W. Spiekermann K, et al. Clin Cancer Res. 2003 Jun;9(6):2140-50. Clin Cancer Res. 2003. PMID: 12796379

3

[Mutations of growth factor receptor Flt3 in acute myeloid leukemia: transformation of myeloid cells by Ras-dependent and Ras-independent mechanisms].

Müller-Tidow C, Steur C, Mizuki M, Schwäble J, Brandts C, Berdel WE, Serve H. Müller-Tidow C, et al. Dtsch Med Wochenschr. 2002 Oct 18;127(42):2195-200. doi: 10.1055/s-2002-34942. Dtsch Med Wochenschr. 2002. PMID: 12397548 German.

4

FLT3 mutations in acute myeloid leukemia cell lines.

Quentmeier H, Reinhardt J, Zaborski M, Drexler HG. Quentmeier H, et al. Leukemia. 2003 Jan;17(1):120-4. doi: 10.1038/sj.leu.2402740. Leukemia. 2003. PMID: 12529668

5

Lyn is an important component of the signal transduction pathway specific to FLT3/ITD and can be a therapeutic target in the treatment of AML with FLT3/ITD.

Okamoto M, Hayakawa F, Miyata Y, Watamoto K, Emi N, Abe A, Kiyoi H, Towatari M, Naoe T. Okamoto M, et al. Leukemia. 2007 Mar;21(3):403-10. doi: 10.1038/sj.leu.2404547. Epub 2007 Jan 18. Leukemia. 2007. PMID: 17230226

6

Inhibition of the transforming activity of FLT3 internal tandem duplication mutants from AML patients by a tyrosine kinase inhibitor.

Tse KF, Allebach J, Levis M, Smith BD, Bohmer FD, Small D. Tse KF, et al. Leukemia. 2002 Oct;16(10):2027-36. doi: 10.1038/sj.leu.2402674. Leukemia. 2002. PMID: 12357354

7

Flt3-mediated signaling in human acute myelogenous leukemia (AML) blasts: a functional characterization of Flt3-ligand effects in AML cell populations with and without genetic Flt3 abnormalities.

Bruserud Ø, Hovland R, Wergeland L, Huang TS, Gjertsen BT. Bruserud Ø, et al. Haematologica. 2003 Apr;88(4):416-28. Haematologica. 2003. PMID: 12681969

8

Effects of SU5416, a small molecule tyrosine kinase receptor inhibitor, on FLT3 expression and phosphorylation in patients with refractory acute myeloid leukemia.

O'Farrell AM, Yuen HA, Smolich B, Hannah AL, Louie SG, Hong W, Stopeck AT, Silverman LR, Lancet JE, Karp JE, Albitar M, Cherrington JM, Giles FJ. O'Farrell AM, et al. Leuk Res. 2004 Jul;28(7):679-89. doi: 10.1016/j.leukres.2003.11.004. Leuk Res. 2004. PMID: 15158089 Clinical Trial.

9

AEE788 is a vascular endothelial growth factor receptor tyrosine kinase inhibitor with antiproliferative and proapoptotic effects in acute myeloid leukemia.

Barbarroja N, Torres LA, Rodriguez-Ariza A, Valverde-Estepa A, Lopez-Sanchez LM, Ruiz-Limon P, Perez-Sanchez C, Carretero RM, Velasco F, López-Pedrera C. Barbarroja N, et al. Exp Hematol. 2010 Aug;38(8):641-52. doi: 10.1016/j.exphem.2010.03.017. Epub 2010 Apr 7. Exp Hematol. 2010. PMID: 20380868 Free article.

10

Inhibition of Flt3-activating mutations does not prevent constitutive activation of ERK/Akt/STAT pathways in some AML cells: a possible cause for the limited effectiveness of monotherapy with small-molecule inhibitors.

Siendones E, Barbarroja N, Torres LA, Buendía P, Velasco F, Dorado G, Torres A, López-Pedrera C. Siendones E, et al. Hematol Oncol. 2007 Mar;25(1):30-7. doi: 10.1002/hon.805. Hematol Oncol. 2007. PMID: 17128418

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