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Items: 1 to 20 of 123

1.

High frequency of partial DIIIa and DAR alleles found in sickle cell disease patients suggests increased risk of alloimmunization to RhD.

Castilho L, Rios M, Rodrigues A, Pellegrino J Jr, Saad ST, Costa FF.

Transfus Med. 2005 Feb;15(1):49-55.

PMID:
15713129
2.

Clinically relevant RHD-CE genotypes in patients with sickle cell disease and in African Brazilian donors.

Gaspardi AC, Sippert EA, De Macedo MD, Pellegrino J Jr, Costa FF, Castilho L.

Blood Transfus. 2016 Sep;14(5):449-54. doi: 10.2450/2016.0275-15.

3.

Partial D, weak D types, and novel RHD alleles among 33,864 multiethnic patients: implications for anti-D alloimmunization and prevention.

Denomme GA, Wagner FF, Fernandes BJ, Li W, Flegel WA.

Transfusion. 2005 Oct;45(10):1554-60.

PMID:
16181204
4.

DIIIa and DIII Type 5 are encoded by the same allele and are associated with altered RHCE*ce alleles: clinical implications.

Westhoff CM, Vege S, Halter-Hipsky C, Whorley T, Hue-Roye K, Lomas-Francis C, Reid ME.

Transfusion. 2010 Jun;50(6):1303-11. doi: 10.1111/j.1537-2995.2009.02573.x.

5.

Partial C antigen in sickle cell disease patients: clinical relevance and prevention of alloimmunization.

Tournamille C, Meunier-Costes N, Costes B, Martret J, Barrault A, Gauthier P, Galactéros F, Nzouékou R, Bierling P, Noizat-Pirenne F.

Transfusion. 2010 Jan;50(1):13-9. doi: 10.1111/j.1537-2995.2009.02382.x.

PMID:
19778340
6.

Weakened expression of 'e' owing to concomitant occurrence of Cys16 and Val245 (VS antigen).

Rodrigues A, Rios M, Costa FF, Saad ST, Pellegrino J Jr, Castilho L.

Vox Sang. 2004 Feb;86(2):136-40.

PMID:
15023184
7.

DNA-based typing of blood groups for the management of multiply-transfused sickle cell disease patients.

Castilho L, Rios M, Bianco C, Pellegrino J Jr, Alberto FL, Saad ST, Costa FF.

Transfusion. 2002 Feb;42(2):232-8.

PMID:
11896340
8.

Identification of RHCE and KEL alleles in large cohorts of Afro-Caribbean and Comorian donors by multiplex SNaPshot and fragment assays: a transfusion support for sickle cell disease patients.

Silvy M, Di Cristofaro J, Beley S, Papa K, Rits M, Richard P, Chiaroni J, Bailly P.

Br J Haematol. 2011 Jul;154(2):260-70. doi: 10.1111/j.1365-2141.2011.08691.x.

PMID:
21623766
9.

Rh discrepancies caused by variable reactivity of partial and weak D types with different serologic techniques.

Denomme GA, Dake LR, Vilensky D, Ramyar L, Judd WJ.

Transfusion. 2008 Mar;48(3):473-8.

10.

DAR, a new RhD variant involving exons 4, 5, and 7, often in linkage with ceAR, a new Rhce variant frequently found in African blacks.

Hemker MB, Ligthart PC, Berger L, van Rhenen DJ, van der Schoot CE, Wijk PA.

Blood. 1999 Dec 15;94(12):4337-42.

11.

Rare RHCE phenotypes in black individuals of Afro-Caribbean origin: identification and transfusion safety.

Noizat-Pirenne F, Lee K, Pennec PY, Simon P, Kazup P, Bachir D, Rouzaud AM, Roussel M, Juszczak G, Ménanteau C, Rouger P, Kotb R, Cartron JP, Ansart-Pirenne H.

Blood. 2002 Dec 1;100(12):4223-31.

12.

Identification of RHD alleles with the potential of anti-D immunization among seemingly D- blood donors in Upper Austria.

Polin H, Danzer M, Gaszner W, Broda D, St-Louis M, Pröll J, Hofer K, Gabriel C.

Transfusion. 2009 Apr;49(4):676-81. doi: 10.1111/j.1537-2995.2008.02046.x.

PMID:
19170995
13.

Serologic and molecular investigations of DAR1 (weak D Type 4.2), DAR1.2, DAR1.3, DAR2 (DARE), and DARA.

Lejon Crottet S, Haer-Wigman L, Gowland P, Fontana S, Niederhauser C, Hustinx H.

Transfusion. 2013 Nov;53(11 Suppl 2):3000-8. doi: 10.1111/trf.12363.

PMID:
23902153
14.

Random survey for RHD alleles among D+ European persons.

Chen Q, Flegel WA.

Transfusion. 2005 Jul;45(7):1183-91.

PMID:
15987365
15.

RHD(T201R, F223V) cluster analysis in five different ethnic groups and serologic characterization of a new Ethiopian variant DARE, the DIII type 6, and the RHD(F223V).

Grootkerk-Tax MG, van Wintershoven JD, Ligthart PC, van Rhenen DJ, van der Schoot CE, Maaskant-van Wijk PA.

Transfusion. 2006 Apr;46(4):606-15.

PMID:
16584437
16.

Isolation, characterization, and family study of DTI, a novel partial D phenotype affecting the fourth external loop of D polypeptides.

Omi T, Takahashi J, Seno T, Tanaka M, Hirayama F, Matsuo M, Ueda N, Obara K, Okuda H, Iwamoto S, Tani Y, Kajii E.

Transfusion. 2002 Apr;42(4):481-9.

PMID:
12076297
17.

Risk of alloimmunization and delayed hemolytic transfusion reactions in patients with sickle cell disease.

Cox JV, Steane E, Cunningham G, Frenkel EP.

Arch Intern Med. 1988 Nov;148(11):2485-9.

PMID:
3142382
18.

Genomic analyses of RH alleles to improve transfusion therapy in patients with sickle cell disease.

Reid ME, Halter Hipsky C, Hue-Roye K, Hoppe C.

Blood Cells Mol Dis. 2014 Apr;52(4):195-202. doi: 10.1016/j.bcmd.2013.11.003.

19.

Thrombophilic mutations among Southern Iranian patients with sickle cell disease: high prevalence of factor V Leiden.

Rahimi Z, Vaisi-Raygani A, Nagel RL, Muniz A.

J Thromb Thrombolysis. 2008 Jun;25(3):288-92.

PMID:
17619828
20.

Six years' experience performing RHD genotyping to confirm D- red blood cell units in Germany for preventing anti-D immunizations.

Flegel WA, von Zabern I, Wagner FF.

Transfusion. 2009 Mar;49(3):465-71. doi: 10.1111/j.1537-2995.2008.01975.x.

PMID:
19243542

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