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Items: 1 to 20 of 143

1.

Cytosolic NADH redox and thiol oxidation regulate pulmonary arterial force through ERK MAP kinase.

Oeckler RA, Arcuino E, Ahmad M, Olson SC, Wolin MS.

Am J Physiol Lung Cell Mol Physiol. 2005 Jun;288(6):L1017-25. Epub 2005 Jan 21.

3.

Roles for cytosolic NADPH redox in regulating pulmonary artery relaxation by thiol oxidation-elicited subunit dimerization of protein kinase G1α.

Neo BH, Patel D, Kandhi S, Wolin MS.

Am J Physiol Heart Circ Physiol. 2013 Aug 1;305(3):H330-43. doi: 10.1152/ajpheart.01010.2011. Epub 2013 May 24.

4.

Regulation of NO-elicited pulmonary artery relaxation and guanylate cyclase activation by NADH oxidase and SOD.

Gupte SA, Rupawalla T, Mohazzab-H KM, Wolin MS.

Am J Physiol. 1999 May;276(5 Pt 2):H1535-42.

5.

Mitochondrial-derived hydrogen peroxide inhibits relaxation of bovine coronary arterial smooth muscle to hypoxia through stimulation of ERK MAP kinase.

Gao Q, Zhao X, Ahmad M, Wolin MS.

Am J Physiol Heart Circ Physiol. 2009 Dec;297(6):H2262-9. doi: 10.1152/ajpheart.00817.2009. Epub 2009 Oct 23.

6.
7.

Thiol oxidation inhibits nitric oxide-mediated pulmonary artery relaxation and guanylate cyclase stimulation.

Mingone CJ, Gupte SA, Ali N, Oeckler RA, Wolin MS.

Am J Physiol Lung Cell Mol Physiol. 2006 Mar;290(3):L549-57. Epub 2005 Nov 4.

8.

Depolarization-induced ERK phosphorylation depends on the cytosolic Ca2+ level rather than on the Ca2+ channel subtype of chromaffin cells.

Mendoza IE, Schmachtenberg O, Tonk E, Fuentealba J, Díaz-Raya P, Lagos VL, García AG, Cárdenas AM.

J Neurochem. 2003 Sep;86(6):1477-86.

9.
10.

Mechanism of fluoride-induced MAP kinase activation in pulmonary artery endothelial cells.

Bogatcheva NV, Wang P, Birukova AA, Verin AD, Garcia JG.

Am J Physiol Lung Cell Mol Physiol. 2006 Jun;290(6):L1139-45. Epub 2006 Jan 13.

11.
12.

Stretch-induced phosphorylation of focal adhesion kinase in endothelial cells: role of mitochondrial oxidants.

Ali MH, Mungai PT, Schumacker PT.

Am J Physiol Lung Cell Mol Physiol. 2006 Jul;291(1):L38-45. Epub 2006 Mar 1.

13.

Involvement of endogenous nitric oxide in angiotensin II-induced activation of vascular mitogen-activated protein kinases.

Zhang GX, Nagai Y, Nakagawa T, Miyanaka H, Fujisawa Y, Nishiyama A, Izuishi K, Ohmori K, Kimura S.

Am J Physiol Heart Circ Physiol. 2007 Oct;293(4):H2403-8. Epub 2007 Jul 6.

14.

Oxidative stress and adenosine A1 receptor activation differentially modulate subcellular cardiomyocyte MAPKs.

Ballard-Croft C, Locklar AC, Keith BJ, Mentzer RM Jr, Lasley RD.

Am J Physiol Heart Circ Physiol. 2008 Jan;294(1):H263-71. Epub 2007 Oct 26.

15.

Enhanced survival effect of pyruvate correlates MAPK and NF-kappaB activation in hydrogen peroxide-treated human endothelial cells.

Lee YJ, Kang IJ, Bünger R, Kang YH.

J Appl Physiol (1985). 2004 Feb;96(2):793-801; discussion 792. Epub 2003 Oct 24.

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18.

Roles for redox mechanisms controlling protein kinase G in pulmonary and coronary artery responses to hypoxia.

Neo BH, Kandhi S, Wolin MS.

Am J Physiol Heart Circ Physiol. 2011 Dec;301(6):H2295-304. doi: 10.1152/ajpheart.00624.2011. Epub 2011 Sep 16.

19.

O2-dependent modulation of calf pulmonary artery tone by lactate: potential role of H2O2 and cGMP.

Omar HA, Mohazzab KM, Mortelliti MP, Wolin MS.

Am J Physiol. 1993 Feb;264(2 Pt 1):L141-5.

PMID:
8383445
20.

Nitric oxide inhibits pulmonary artery catalase and H2O2-associated relaxation.

Mohazzab-H KM, Fayngersh RP, Wolin MS.

Am J Physiol. 1996 Nov;271(5 Pt 2):H1900-6.

PMID:
8945907

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