Similar articles for PMID: 15657432

Year	Number of Results
1994	1
1995	2
1996	7
1997	7
1998	4
1999	5
2000	9
2001	5
2002	3
2003	4
2004	5
2005	5
2006	5
2007	9
2008	3
2009	8
2010	9
2011	13
2012	6
2013	4
2014	5
2015	5
2016	6
2017	4
2018	5
2019	3
2020	1
2021	6
2022	2
2023	1
2024	0

1

c-Jun N-terminal kinase contributes to aberrant retinoid signaling in lung cancer cells by phosphorylating and inducing proteasomal degradation of retinoic acid receptor alpha.

Srinivas H, Juroske DM, Kalyankrishna S, Cody DD, Price RE, Xu XC, Narayanan R, Weigel NL, Kurie JM. Srinivas H, et al. Mol Cell Biol. 2005 Feb;25(3):1054-69. doi: 10.1128/MCB.25.3.1054-1069.2005. Mol Cell Biol. 2005. PMID: 15657432 Free PMC article.

2

c-Jun N-terminal kinase activation by oxidative stress suppresses retinoid signaling through proteasomal degradation of retinoic acid receptor α protein in hepatic cells.

Hoshikawa Y, Kanki K, Ashla AA, Arakaki Y, Azumi J, Yasui T, Tezuka Y, Matsumi Y, Tsuchiya H, Kurimasa A, Hisatome I, Hirano T, Fujimoto J, Kagechika H, Shomori K, Ito H, Shiota G. Hoshikawa Y, et al. Cancer Sci. 2011 May;102(5):934-41. doi: 10.1111/j.1349-7006.2011.01889.x. Epub 2011 Feb 24. Cancer Sci. 2011. PMID: 21272161 Free article.

3

High glucose-induced repression of RAR/RXR in cardiomyocytes is mediated through oxidative stress/JNK signaling.

Singh AB, Guleria RS, Nizamutdinova IT, Baker KM, Pan J. Singh AB, et al. J Cell Physiol. 2012 Jun;227(6):2632-44. doi: 10.1002/jcp.23005. J Cell Physiol. 2012. PMID: 21882190 Free PMC article.

4

Phosphorylation of the retinoid x receptor at the omega loop, modulates the expression of retinoic-acid-target genes with a promoter context specificity.

Bruck N, Bastien J, Bour G, Tarrade A, Plassat JL, Bauer A, Adam-Stitah S, Rochette-Egly C. Bruck N, et al. Cell Signal. 2005 Oct;17(10):1229-39. doi: 10.1016/j.cellsig.2004.12.006. Epub 2005 Jan 20. Cell Signal. 2005. PMID: 16038797

5

All-trans-retinoic acid inhibits Jun N-terminal kinase by increasing dual-specificity phosphatase activity.

Lee HY, Sueoka N, Hong WK, Mangelsdorf DJ, Claret FX, Kurie JM. Lee HY, et al. Mol Cell Biol. 1999 Mar;19(3):1973-80. doi: 10.1128/MCB.19.3.1973. Mol Cell Biol. 1999. PMID: 10022884 Free PMC article.

6

Retinoic acid-induced blr1 expression requires RARalpha, RXR, and MAPK activation and uses ERK2 but not JNK/SAPK to accelerate cell differentiation.

Battle TE, Roberson MS, Zhang T, Varvayanis S, Yen A. Battle TE, et al. Eur J Cell Biol. 2001 Jan;80(1):59-67. doi: 10.1078/0171-9335-00141. Eur J Cell Biol. 2001. PMID: 11211936

7

Retinoic acids acting through retinoid receptors protect hippocampal neurons from oxygen-glucose deprivation-mediated cell death by inhibition of c-jun-N-terminal kinase and p38 mitogen-activated protein kinase.

Shinozaki Y, Sato Y, Koizumi S, Ohno Y, Nagao T, Inoue K. Shinozaki Y, et al. Neuroscience. 2007 Jun 15;147(1):153-63. doi: 10.1016/j.neuroscience.2007.04.032. Epub 2007 May 22. Neuroscience. 2007. PMID: 17521827

8

Increased retinoic acid responsiveness in lung carcinoma cells that are nonresponsive despite the presence of endogenous retinoic acid receptor (RAR) beta by expression of exogenous retinoid receptors retinoid X receptor alpha, RAR alpha, and RAR gamma.

Wan H, Hong WK, Lotan R. Wan H, et al. Cancer Res. 2001 Jan 15;61(2):556-64. Cancer Res. 2001. PMID: 11212249

9

All-trans-retinoic acid inhibits Jun N-terminal kinase-dependent signaling pathways.

Lee HY, Walsh GL, Dawson MI, Hong WK, Kurie JM. Lee HY, et al. J Biol Chem. 1998 Mar 20;273(12):7066-71. doi: 10.1074/jbc.273.12.7066. J Biol Chem. 1998. PMID: 9507016 Free article.

10

Retinoid refractoriness occurs during lung carcinogenesis despite functional retinoid receptors.

Kim YH, Dohi DF, Han GR, Zou CP, Oridate N, Walsh GL, Nesbitt JC, Xu XC, Hong WK, Lotan R, et al. Kim YH, et al. Cancer Res. 1995 Dec 1;55(23):5603-10. Cancer Res. 1995. PMID: 7585641

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