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Balance of MafB and PU.1 specifies alternative macrophage or dendritic cell fate.

Bakri Y, Sarrazin S, Mayer UP, Tillmanns S, Nerlov C, Boned A, Sieweke MH.

Blood. 2005 Apr 1;105(7):2707-16.


MafB is an inducer of monocytic differentiation.

Kelly LM, Englmeier U, Lafon I, Sieweke MH, Graf T.

EMBO J. 2000 May 2;19(9):1987-97.


Transient expression of PU.1 commits multipotent progenitors to a myeloid fate whereas continued expression favors macrophage over granulocyte differentiation.

McIvor Z, Hein S, Fiegler H, Schroeder T, Stocking C, Just U, Cross M.

Exp Hematol. 2003 Jan;31(1):39-47.


MafB is an interaction partner and repressor of Ets-1 that inhibits erythroid differentiation.

Sieweke MH, Tekotte H, Frampton J, Graf T.

Cell. 1996 Apr 5;85(1):49-60.


PU.1 regulates the tissue-specific expression of dendritic cell-specific intercellular adhesion molecule (ICAM)-3-grabbing nonintegrin.

Domínguez-Soto A, Puig-Kröger A, Vega MA, Corbí AL.

J Biol Chem. 2005 Sep 30;280(39):33123-31.


The transcriptional repressor Gfi1 controls STAT3-dependent dendritic cell development and function.

Rathinam C, Geffers R, Yücel R, Buer J, Welte K, Möröy T, Klein C.

Immunity. 2005 Jun;22(6):717-28.


Neutrophils and monocytes express high levels of PU.1 (Spi-1) but not Spi-B.

Chen HM, Zhang P, Voso MT, Hohaus S, Gonzalez DA, Glass CK, Zhang DE, Tenen DG.

Blood. 1995 May 15;85(10):2918-28.


PU.1 is essential for CD11c expression in CD8(+)/CD8(-) lymphoid and monocyte-derived dendritic cells during GM-CSF or FLT3L-induced differentiation.

Zhu XJ, Yang ZF, Chen Y, Wang J, Rosmarin AG.

PLoS One. 2012;7(12):e52141. doi: 10.1371/journal.pone.0052141.


PU.1 and the granulocyte- and macrophage colony-stimulating factor receptors play distinct roles in late-stage myeloid cell differentiation.

Anderson KL, Smith KA, Perkin H, Hermanson G, Anderson CG, Jolly DJ, Maki RA, Torbett BE.

Blood. 1999 Oct 1;94(7):2310-8.


Transcription factor PU.1 is necessary for development of thymic and myeloid progenitor-derived dendritic cells.

Anderson KL, Perkin H, Surh CD, Venturini S, Maki RA, Torbett BE.

J Immunol. 2000 Feb 15;164(4):1855-61.


Reprogramming of committed T cell progenitors to macrophages and dendritic cells by C/EBP alpha and PU.1 transcription factors.

Laiosa CV, Stadtfeld M, Xie H, de Andres-Aguayo L, Graf T.

Immunity. 2006 Nov;25(5):731-44.


Differential expression of IFN regulatory factor 4 gene in human monocyte-derived dendritic cells and macrophages.

Lehtonen A, Veckman V, Nikula T, Lahesmaa R, Kinnunen L, Matikainen S, Julkunen I.

J Immunol. 2005 Nov 15;175(10):6570-9.


Regulation of macrophage and neutrophil cell fates by the PU.1:C/EBPalpha ratio and granulocyte colony-stimulating factor.

Dahl R, Walsh JC, Lancki D, Laslo P, Iyer SR, Singh H, Simon MC.

Nat Immunol. 2003 Oct;4(10):1029-36.


Downregulation of mafB expression in T-helper cells during early differentiation in vitro.

Hashizume H, Hamalainen H, Sun Q, Sucharczuk A, Lahesmaa R.

Scand J Immunol. 2003 Jan;57(1):28-34.


MicroRNA-155 modulates the pathogen binding ability of dendritic cells (DCs) by down-regulation of DC-specific intercellular adhesion molecule-3 grabbing non-integrin (DC-SIGN).

Martinez-Nunez RT, Louafi F, Friedmann PS, Sanchez-Elsner T.

J Biol Chem. 2009 Jun 12;284(24):16334-42. doi: 10.1074/jbc.M109.011601.


Distinct signals control the hematopoiesis of lymphoid-related dendritic cells.

Galy A, Christopherson I, Ferlazzo G, Liu G, Spits H, Georgopoulos K.

Blood. 2000 Jan 1;95(1):128-37.


The expression pattern of the mafB/kr gene in birds and mice reveals that the kreisler phenotype does not represent a null mutant.

Eichmann A, Grapin-Botton A, Kelly L, Graf T, Le Douarin NM, Sieweke M.

Mech Dev. 1997 Jul;65(1-2):111-22.


Multilineage transcriptional priming and determination of alternate hematopoietic cell fates.

Laslo P, Spooner CJ, Warmflash A, Lancki DW, Lee HJ, Sciammas R, Gantner BN, Dinner AR, Singh H.

Cell. 2006 Aug 25;126(4):755-66.


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