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Items: 1 to 20 of 125

1.

The cerebellar transcriptome during postnatal development of the Ts1Cje mouse, a segmental trisomy model for Down syndrome.

Dauphinot L, Lyle R, Rivals I, Dang MT, Moldrich RX, Golfier G, Ettwiller L, Toyama K, Rossier J, Personnaz L, Antonarakis SE, Epstein CJ, Sinet PM, Potier MC.

Hum Mol Genet. 2005 Feb 1;14(3):373-84. Epub 2004 Dec 8.

PMID:
15590701
2.

Transcriptional disruptions in Down syndrome: a case study in the Ts1Cje mouse cerebellum during post-natal development.

Potier MC, Rivals I, Mercier G, Ettwiller L, Moldrich RX, Laffaire J, Personnaz L, Rossier J, Dauphinot L.

J Neurochem. 2006 Apr;97 Suppl 1:104-9.

3.

Dosage-dependent over-expression of genes in the trisomic region of Ts1Cje mouse model for Down syndrome.

Amano K, Sago H, Uchikawa C, Suzuki T, Kotliarova SE, Nukina N, Epstein CJ, Yamakawa K.

Hum Mol Genet. 2004 Jul 1;13(13):1333-40. Epub 2004 May 11.

PMID:
15138197
4.

Gene expression signature of cerebellar hypoplasia in a mouse model of Down syndrome during postnatal development.

Laffaire J, Rivals I, Dauphinot L, Pasteau F, Wehrle R, Larrat B, Vitalis T, Moldrich RX, Rossier J, Sinkus R, Herault Y, Dusart I, Potier MC.

BMC Genomics. 2009 Mar 30;10:138. doi: 10.1186/1471-2164-10-138.

5.

Down syndrome gene dosage imbalance on cerebellum development.

Moldrich RX, Dauphinot L, Laffaire J, Rossier J, Potier MC.

Prog Neurobiol. 2007 Jun;82(2):87-94. Epub 2007 Feb 23. Review.

PMID:
17408845
6.

Global disruption of the cerebellar transcriptome in a Down syndrome mouse model.

Saran NG, Pletcher MT, Natale JE, Cheng Y, Reeves RH.

Hum Mol Genet. 2003 Aug 15;12(16):2013-9.

PMID:
12913072
7.

Functional transcriptome analysis of the postnatal brain of the Ts1Cje mouse model for Down syndrome reveals global disruption of interferon-related molecular networks.

Ling KH, Hewitt CA, Tan KL, Cheah PS, Vidyadaran S, Lai MI, Lee HC, Simpson K, Hyde L, Pritchard MA, Smyth GK, Thomas T, Scott HS.

BMC Genomics. 2014 Jul 22;15:624. doi: 10.1186/1471-2164-15-624.

8.

Transmembrane protein 50b (C21orf4), a candidate for Down syndrome neurophenotypes, encodes an intracellular membrane protein expressed in the rodent brain.

Moldrich RX, Lainé J, Visel A, Beart PM, Laffaire J, Rossier J, Potier MC.

Neuroscience. 2008 Jul 17;154(4):1255-66. doi: 10.1016/j.neuroscience.2008.01.089. Epub 2008 Mar 4.

PMID:
18541381
9.

Proliferation deficits and gene expression dysregulation in Down's syndrome (Ts1Cje) neural progenitor cells cultured from neurospheres.

Moldrich RX, Dauphinot L, Laffaire J, Vitalis T, Hérault Y, Beart PM, Rossier J, Vivien D, Gehrig C, Antonarakis SE, Lyle R, Potier MC.

J Neurosci Res. 2009 Nov 1;87(14):3143-52. doi: 10.1002/jnr.22131.

PMID:
19472221
10.

Cerebellar development transcriptome database (CDT-DB): profiling of spatio-temporal gene expression during the postnatal development of mouse cerebellum.

Sato A, Sekine Y, Saruta C, Nishibe H, Morita N, Sato Y, Sadakata T, Shinoda Y, Kojima T, Furuichi T.

Neural Netw. 2008 Oct;21(8):1056-69. doi: 10.1016/j.neunet.2008.05.004. Epub 2008 Jun 4.

PMID:
18603407
11.

Developmental instability of the cerebellum and its relevance to Down syndrome.

Shapiro BL.

J Neural Transm Suppl. 2001;(61):11-34. Review.

PMID:
11771737
12.

Gene expression profiling of mouse postnatal cerebellar development using cDNA microarrays.

Lim CR, Fukakusa A, Matsubara K.

Gene. 2004 May 26;333:3-13.

PMID:
15177675
13.

Overexpression of mSim2 gene in the zona limitans of the diencephalon of segmental trisomy 16 Ts1Cje fetuses, a mouse model for trisomy 21: a novel whole-mount based RNA hybridization study.

Vialard F, Toyama K, Vernoux S, Carlson EJ, Epstein CJ, Sinet PM, Rahmani Z.

Brain Res Dev Brain Res. 2000 May 11;121(1):73-8.

PMID:
10837894
14.

Mental retardation in Down syndrome: from gene dosage imbalance to molecular and cellular mechanisms.

Rachidi M, Lopes C.

Neurosci Res. 2007 Dec;59(4):349-69. Epub 2007 Aug 15. Review.

PMID:
17897742
15.

Specific age-related molecular alterations in the cerebellum of Down syndrome mouse models.

Créau N, Cabet E, Daubigney F, Souchet B, Bennaï S, Delabar J.

Brain Res. 2016 Sep 1;1646:342-353. doi: 10.1016/j.brainres.2016.06.003. Epub 2016 Jun 11.

PMID:
27297494
16.
17.

Gene expression profiles following exposure to a developmental neurotoxicant, Aroclor 1254: pathway analysis for possible mode(s) of action.

Royland JE, Kodavanti PR.

Toxicol Appl Pharmacol. 2008 Sep 1;231(2):179-96. doi: 10.1016/j.taap.2008.04.023. Epub 2008 May 6.

PMID:
18602130
18.

Abnormal synaptic plasticity in the Ts1Cje segmental trisomy 16 mouse model of Down syndrome.

Siarey RJ, Villar AJ, Epstein CJ, Galdzicki Z.

Neuropharmacology. 2005 Jul;49(1):122-8.

PMID:
15992587
19.

Trisomy for the Down syndrome 'critical region' is necessary but not sufficient for brain phenotypes of trisomic mice.

Olson LE, Roper RJ, Sengstaken CL, Peterson EA, Aquino V, Galdzicki Z, Siarey R, Pletnikov M, Moran TH, Reeves RH.

Hum Mol Genet. 2007 Apr 1;16(7):774-82. Epub 2007 Mar 5.

PMID:
17339268
20.

The fetal brain transcriptome and neonatal behavioral phenotype in the Ts1Cje mouse model of Down syndrome.

Guedj F, Pennings JL, Ferres MA, Graham LC, Wick HC, Miczek KA, Slonim DK, Bianchi DW.

Am J Med Genet A. 2015 Sep;167A(9):1993-2008. doi: 10.1002/ajmg.a.37156. Epub 2015 May 14.

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