Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 94


Gastric cancer originating from bone marrow-derived cells.

Houghton J, Stoicov C, Nomura S, Rogers AB, Carlson J, Li H, Cai X, Fox JG, Goldenring JR, Wang TC.

Science. 2004 Nov 26;306(5701):1568-71.


Medicine. Bone marrow cells: the source of gastric cancer?

Marx J.

Science. 2004 Nov 26;306(5701):1455-7. No abstract available.


K-ras mutation targeted to gastric tissue progenitor cells results in chronic inflammation, an altered microenvironment, and progression to intraepithelial neoplasia.

Okumura T, Ericksen RE, Takaishi S, Wang SS, Dubeykovskiy Z, Shibata W, Betz KS, Muthupalani S, Rogers AB, Fox JG, Rustgi AK, Wang TC.

Cancer Res. 2010 Nov 1;70(21):8435-45. doi: 10.1158/0008-5472.CAN-10-1506.


Helicobacter pylori infection recruits bone marrow-derived cells that participate in gastric preneoplasia in mice.

Varon C, Dubus P, Mazurier F, Asencio C, Chambonnier L, Ferrand J, Giese A, Senant-Dugot N, Carlotti M, Mégraud F.

Gastroenterology. 2012 Feb;142(2):281-91. doi: 10.1053/j.gastro.2011.10.036.


Mature chief cells are cryptic progenitors for metaplasia in the stomach.

Nam KT, Lee HJ, Sousa JF, Weis VG, O'Neal RL, Finke PE, Romero-Gallo J, Shi G, Mills JC, Peek RM Jr, Konieczny SF, Goldenring JR.

Gastroenterology. 2010 Dec;139(6):2028-2037.e9. doi: 10.1053/j.gastro.2010.09.005.


Fibroblastic colony-forming unit bone marrow cells delay progression to gastric dysplasia in a helicobacter model of gastric tumorigenesis.

Wang SS, Asfaha S, Okumura T, Betz KS, Muthupalani S, Rogers AB, Tu S, Takaishi S, Jin G, Yang X, Wu DC, Fox JG, Wang TC.

Stem Cells. 2009 Sep;27(9):2301-11. doi: 10.1002/stem.165.


Identification of a bone marrow-derived mesenchymal progenitor cell subset that can contribute to the gastric epithelium.

Okumura T, Wang SS, Takaishi S, Tu SP, Ng V, Ericksen RE, Rustgi AK, Wang TC.

Lab Invest. 2009 Dec;89(12):1410-22. doi: 10.1038/labinvest.2009.88.


Spasmolytic polypeptide expressing metaplasia to preneoplasia in H. felis-infected mice.

Nomura S, Baxter T, Yamaguchi H, Leys C, Vartapetian AB, Fox JG, Lee JR, Wang TC, Goldenring JR.

Gastroenterology. 2004 Aug;127(2):582-94.


Mice lacking secretory phospholipase A2 show altered apoptosis and differentiation with Helicobacter felis infection.

Wang TC, Goldenring JR, Dangler C, Ito S, Mueller A, Jeon WK, Koh TJ, Fox JG.

Gastroenterology. 1998 Apr;114(4):675-89.


Gastric mucus alterations associated with murine Helicobacter infection.

Schmitz JM, Durham CG, Ho SB, Lorenz RG.

J Histochem Cytochem. 2009 May;57(5):457-67. doi: 10.1369/jhc.2009.952473.


Augmented intestinal trefoil factor (TFF3) and loss of pS2 (TFF1) expression precedes metaplastic differentiation of gastric epithelium.

Taupin D, Pedersen J, Familari M, Cook G, Yeomans N, Giraud AS.

Lab Invest. 2001 Mar;81(3):397-408.


Activation of EGFR and ERBB2 by Helicobacter pylori results in survival of gastric epithelial cells with DNA damage.

Chaturvedi R, Asim M, Piazuelo MB, Yan F, Barry DP, Sierra JC, Delgado AG, Hill S, Casero RA Jr, Bravo LE, Dominguez RL, Correa P, Polk DB, Washington MK, Rose KL, Schey KL, Morgan DR, Peek RM Jr, Wilson KT.

Gastroenterology. 2014 Jun;146(7):1739-51.e14. doi: 10.1053/j.gastro.2014.02.005.


Gastritis promotes an activated bone marrow-derived mesenchymal stem cell with a phenotype reminiscent of a cancer-promoting cell.

Donnelly JM, Engevik AC, Engevik M, Schumacher MA, Xiao C, Yang L, Worrell RT, Zavros Y.

Dig Dis Sci. 2014 Mar;59(3):569-82. doi: 10.1007/s10620-013-2927-z.


Mesenchymal stem cells utilize CXCR4-SDF-1 signaling for acute, but not chronic, trafficking to gastric mucosal inflammation.

Stoicov C, Li H, Liu JH, Houghton J.

Dig Dis Sci. 2013 Sep;58(9):2466-77. doi: 10.1007/s10620-013-2782-y.


A molecular signature of gastric metaplasia arising in response to acute parietal cell loss.

Nozaki K, Ogawa M, Williams JA, Lafleur BJ, Ng V, Drapkin RI, Mills JC, Konieczny SF, Nomura S, Goldenring JR.

Gastroenterology. 2008 Feb;134(2):511-22. doi: 10.1053/j.gastro.2007.11.058.


Metaplasia, intraepithelial neoplasia and early cancer of the stomach are related to dedifferentiated epithelial cells defined by cytokeratin-7 expression in gastritis.

Kirchner T, Müller S, Hattori T, Mukaisyo K, Papadopoulos T, Brabletz T, Jung A.

Virchows Arch. 2001 Oct;439(4):512-22.


Gli1 deletion prevents Helicobacter-induced gastric metaplasia and expansion of myeloid cell subsets.

El-Zaatari M, Kao JY, Tessier A, Bai L, Hayes MM, Fontaine C, Eaton KA, Merchant JL.

PLoS One. 2013;8(3):e58935. doi: 10.1371/journal.pone.0058935.


Metaplasia of the duodenum shows a Helicobacter pylori-correlated differentiation into gastric-type protein expression.

Van De Bovenkamp JH, Korteland-Van Male AM, Büller HA, Einerhand AW, Dekker J.

Hum Pathol. 2003 Feb;34(2):156-65.


Conditional deletion of IkappaB-kinase-beta accelerates helicobacter-dependent gastric apoptosis, proliferation, and preneoplasia.

Shibata W, Takaishi S, Muthupalani S, Pritchard DM, Whary MT, Rogers AB, Fox JG, Betz KS, Kaestner KH, Karin M, Wang TC.

Gastroenterology. 2010 Mar;138(3):1022-34.e1-10. doi: 10.1053/j.gastro.2009.11.054.


Stromal cell-derived factor-1 overexpression induces gastric dysplasia through expansion of stromal myofibroblasts and epithelial progenitors.

Shibata W, Ariyama H, Westphalen CB, Worthley DL, Muthupalani S, Asfaha S, Dubeykovskaya Z, Quante M, Fox JG, Wang TC.

Gut. 2013 Feb;62(2):192-200. doi: 10.1136/gutjnl-2011-301824.

Supplemental Content

Support Center