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Items: 1 to 20 of 158

1.

The function of RecQ helicase gene family (especially BLM) in DNA recombination and joining.

Kaneko H, Fukao T, Kondo N.

Adv Biophys. 2004;38:45-64. Review.

PMID:
15493327
2.

The function of RecQ helicase gene family (especially BLM) in DNA recombination and joining.

Kaneko H, Fukao T, Kondo N.

Adv Biophys. 2004;38(Complete):45-64.

PMID:
15476892
3.

Potential role for the BLM helicase in recombinational repair via a conserved interaction with RAD51.

Wu L, Davies SL, Levitt NC, Hickson ID.

J Biol Chem. 2001 Jun 1;276(22):19375-81.

4.

Expression of BLM (the causative gene for Bloom syndrome) and screening of Bloom syndrome.

Morimoto W, Kaneko H, Isogai K, Kasahara K, Kondo N.

Int J Mol Med. 2002 Jul;10(1):95-9.

PMID:
12060858
5.

The Bloom's syndrome gene product promotes branch migration of holliday junctions.

Karow JK, Constantinou A, Li JL, West SC, Hickson ID.

Proc Natl Acad Sci U S A. 2000 Jun 6;97(12):6504-8.

6.

[Functional analysis of yeast homologue gene associated with human DNA helicase causative syndromes].

Miyajima A.

Kokuritsu Iyakuhin Shokuhin Eisei Kenkyusho Hokoku. 2002;(120):53-74. Review. Japanese.

PMID:
12638184
7.

Clinical features of Bloom syndrome and function of the causative gene, BLM helicase.

Kaneko H, Kondo N.

Expert Rev Mol Diagn. 2004 May;4(3):393-401. Review.

PMID:
15137905
8.

Expression of the BLM gene in human haematopoietic cells.

Kaneko H, Matsui E, Fukao T, Kasahara K, Morimoto W, Kondo N.

Clin Exp Immunol. 1999 Nov;118(2):285-9.

9.

Werner and Bloom helicases are involved in DNA repair in a complementary fashion.

Imamura O, Fujita K, Itoh C, Takeda S, Furuichi Y, Matsumoto T.

Oncogene. 2002 Jan 31;21(6):954-63.

10.
11.

BLM (the causative gene of Bloom syndrome) protein translocation into the nucleus by a nuclear localization signal.

Kaneko H, Orii KO, Matsui E, Shimozawa N, Fukao T, Matsumoto T, Shimamoto A, Furuichi Y, Hayakawa S, Kasahara K, Kondo N.

Biochem Biophys Res Commun. 1997 Nov 17;240(2):348-53.

PMID:
9388480
12.

Vertebrate WRNIP1 and BLM are required for efficient maintenance of genome stability.

Hayashi T, Seki M, Inoue E, Yoshimura A, Kusa Y, Tada S, Enomoto T.

Genes Genet Syst. 2008 Feb;83(1):95-100.

13.

Two Japanese siblings with Bloom syndrome gene mutation and B-cell lymphoma.

Kaneko H, Inoue R, Fukao T, Kasahara K, Tashita H, Teramoto T, Kondo N.

Leuk Lymphoma. 1997 Nov;27(5-6):539-42.

PMID:
9477137
14.

Binding and melting of D-loops by the Bloom syndrome helicase.

van Brabant AJ, Ye T, Sanz M, German III JL, Ellis NA, Holloman WK.

Biochemistry. 2000 Nov 28;39(47):14617-25.

PMID:
11087418
15.

DNA helicase deficiencies associated with cancer predisposition and premature ageing disorders.

Mohaghegh P, Hickson ID.

Hum Mol Genet. 2001 Apr;10(7):741-6. Review.

PMID:
11257107
16.

Structural basis of Bloom syndrome (BS) causing mutations in the BLM helicase domain.

Rong SB, Väliaho J, Vihinen M.

Mol Med. 2000 Mar;6(3):155-64.

17.

Bloom helicase is involved in DNA surveillance in early S phase in vertebrate cells.

Imamura O, Fujita K, Shimamoto A, Tanabe H, Takeda S, Furuichi Y, Matsumoto T.

Oncogene. 2001 Mar 8;20(10):1143-51.

18.

Relatively common mutations of the Bloom syndrome gene in the Japanese population.

Kaneko H, Isogai K, Fukao T, Matsui E, Kasahara K, Yachie A, Seki H, Koizumi S, Arai M, Utunomiya J, Miki Y, Kondo N.

Int J Mol Med. 2004 Sep;14(3):439-42.

PMID:
15289897
19.

[Bloom syndrome].

Kondo N.

Nihon Rinsho. 2000 Jul;58(7):1460-6. Review. Japanese.

PMID:
10921324
20.

BLM helicase-dependent transport of p53 to sites of stalled DNA replication forks modulates homologous recombination.

Sengupta S, Linke SP, Pedeux R, Yang Q, Farnsworth J, Garfield SH, Valerie K, Shay JW, Ellis NA, Wasylyk B, Harris CC.

EMBO J. 2003 Mar 3;22(5):1210-22.

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