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Items: 1 to 20 of 257

1.

Differential expression of SAP and EAT-2-binding leukocyte cell-surface molecules CD84, CD150 (SLAM), CD229 (Ly9) and CD244 (2B4).

Romero X, Benítez D, March S, Vilella R, Miralpeix M, Engel P.

Tissue Antigens. 2004 Aug;64(2):132-44.

PMID:
15245368
2.

Identification and characterization of SF2000 and SF2001, two new members of the immune receptor SLAM/CD2 family.

Fraser CC, Howie D, Morra M, Qiu Y, Murphy C, Shen Q, Gutierrez-Ramos JC, Coyle A, Kingsbury GA, Terhorst C.

Immunogenetics. 2002 Feb;53(10-11):843-50.

PMID:
11862385
3.

Characterization of mouse CD229 (Ly9), a leukocyte cell surface molecule of the CD150 (SLAM) family.

Sintes J, Vidal-Laliena M, Romero X, Tovar V, Engel P.

Tissue Antigens. 2007 Nov;70(5):355-62.

PMID:
17919264
4.

Mouse novel Ly9: a new member of the expanding CD150 (SLAM) family of leukocyte cell-surface receptors.

Tovar V, del Valle J, Zapater N, Martin M, Romero X, Pizcueta P, Bosch J, Terhorst C, Engel P.

Immunogenetics. 2002 Sep;54(6):394-402.

PMID:
12242590
5.

CD150 is a member of a family of genes that encode glycoproteins on the surface of hematopoietic cells.

Wang N, Morra M, Wu C, Gullo C, Howie D, Coyle T, Engel P, Terhorst C.

Immunogenetics. 2001 Jul;53(5):382-94.

PMID:
11486275
6.

Expression of SLAM (CD150) cell-surface receptors on human B-cell subsets: from pro-B to plasma cells.

De Salort J, Sintes J, Llinàs L, Matesanz-Isabel J, Engel P.

Immunol Lett. 2011 Jan 30;134(2):129-36. doi: 10.1016/j.imlet.2010.09.021.

PMID:
20933013
7.

Analysis of immune activation and clinical events in acute infectious mononucleosis.

Williams H, Macsween K, McAulay K, Higgins C, Harrison N, Swerdlow A, Britton K, Crawford D.

J Infect Dis. 2004 Jul 1;190(1):63-71.

PMID:
15195244
9.
10.

Structural basis for the interaction of the free SH2 domain EAT-2 with SLAM receptors in hematopoietic cells.

Morra M, Lu J, Poy F, Martin M, Sayos J, Calpe S, Gullo C, Howie D, Rietdijk S, Thompson A, Coyle AJ, Denny C, Yaffe MB, Engel P, Eck MJ, Terhorst C.

EMBO J. 2001 Nov 1;20(21):5840-52.

11.

Differential expression of CD150 (SLAM) family receptors by human hematopoietic stem and progenitor cells.

Sintes J, Romero X, Marin P, Terhorst C, Engel P.

Exp Hematol. 2008 Sep;36(9):1199-204. doi: 10.1016/j.exphem.2008.03.015.

12.

Ly9 (CD229)-deficient mice exhibit T cell defects yet do not share several phenotypic characteristics associated with SLAM- and SAP-deficient mice.

Graham DB, Bell MP, McCausland MM, Huntoon CJ, van Deursen J, Faubion WA, Crotty S, McKean DJ.

J Immunol. 2006 Jan 1;176(1):291-300. Erratum in: J Immunol. 2006 Mar 15;176(6):3841.

13.

Potential pathways for regulation of NK and T cell responses: differential X-linked lymphoproliferative syndrome gene product SAP interactions with SLAM and 2B4.

Sayós J, Nguyen KB, Wu C, Stepp SE, Howie D, Schatzle JD, Kumar V, Biron CA, Terhorst C.

Int Immunol. 2000 Dec;12(12):1749-57.

PMID:
11099315
14.

Distinct interactions of the X-linked lymphoproliferative syndrome gene product SAP with cytoplasmic domains of members of the CD2 receptor family.

Lewis J, Eiben LJ, Nelson DL, Cohen JI, Nichols KE, Ochs HD, Notarangelo LD, Duckett CS.

Clin Immunol. 2001 Jul;100(1):15-23.

PMID:
11414741
15.

Molecular and cellular pathogenesis of X-linked lymphoproliferative disease.

Nichols KE, Ma CS, Cannons JL, Schwartzberg PL, Tangye SG.

Immunol Rev. 2005 Feb;203:180-99. Review.

PMID:
15661030
16.

Molecular basis for positive and negative signaling by the natural killer cell receptor 2B4 (CD244).

Eissmann P, Beauchamp L, Wooters J, Tilton JC, Long EO, Watzl C.

Blood. 2005 Jun 15;105(12):4722-9.

17.

Molecular dissection of 2B4 signaling: implications for signal transduction by SLAM-related receptors.

Chen R, Relouzat F, Roncagalli R, Aoukaty A, Tan R, Latour S, Veillette A.

Mol Cell Biol. 2004 Jun;24(12):5144-56.

18.

2B4 (CD244) is expressed and functional on human eosinophils.

Munitz A, Bachelet I, Fraenkel S, Katz G, Mandelboim O, Simon HU, Moretta L, Colonna M, Levi-Schaffer F.

J Immunol. 2005 Jan 1;174(1):110-8.

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