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Items: 1 to 20 of 105

1.

Monoglyceride lipase-like enzymatic activity is responsible for hydrolysis of 2-arachidonoylglycerol in rat cerebellar membranes.

Saario SM, Savinainen JR, Laitinen JT, Järvinen T, Niemi R.

Biochem Pharmacol. 2004 Apr 1;67(7):1381-7.

PMID:
15013854
2.

Brain monoglyceride lipase participating in endocannabinoid inactivation.

Dinh TP, Carpenter D, Leslie FM, Freund TF, Katona I, Sensi SL, Kathuria S, Piomelli D.

Proc Natl Acad Sci U S A. 2002 Aug 6;99(16):10819-24. Erratum in: Proc Natl Acad Sci U S A 2002 Oct 15;99(21):13961.

3.

Characterization of the sulfhydryl-sensitive site in the enzyme responsible for hydrolysis of 2-arachidonoyl-glycerol in rat cerebellar membranes.

Saario SM, Salo OM, Nevalainen T, Poso A, Laitinen JT, Järvinen T, Niemi R.

Chem Biol. 2005 Jun;12(6):649-56.

4.

Discovery and development of endocannabinoid-hydrolyzing enzyme inhibitors.

Minkkilä A, Saario S, Nevalainen T.

Curr Top Med Chem. 2010;10(8):828-58. Review.

PMID:
20370710
5.

Fatty acid amide hydrolase but not monoacyl glycerol lipase controls cell death induced by the endocannabinoid 2-arachidonoyl glycerol in hepatic cell populations.

Siegmund SV, Wojtalla A, Schlosser M, Zimmer A, Singer MV.

Biochem Biophys Res Commun. 2013 Jul 19;437(1):48-54. doi: 10.1016/j.bbrc.2013.06.033.

PMID:
23806692
6.

The novel endogenous cannabinoid 2-arachidonoylglycerol is inactivated by neuronal- and basophil-like cells: connections with anandamide.

Di Marzo V, Bisogno T, Sugiura T, Melck D, De Petrocellis L.

Biochem J. 1998 Apr 1;331 ( Pt 1):15-9.

7.

Inhibition of monoacylglycerol lipase and fatty acid amide hydrolase by analogues of 2-arachidonoylglycerol.

Ghafouri N, Tiger G, Razdan RK, Mahadevan A, Pertwee RG, Martin BR, Fowler CJ.

Br J Pharmacol. 2004 Nov;143(6):774-84.

9.

Brain regional cannabinoid CB(1) receptor signalling and alternative enzymatic pathways for 2-arachidonoylglycerol generation in brain sections of diacylglycerol lipase deficient mice.

Aaltonen N, Riera Ribas C, Lehtonen M, Savinainen JR, Laitinen JT.

Eur J Pharm Sci. 2014 Jan 23;51:87-95. doi: 10.1016/j.ejps.2013.08.035.

PMID:
24012970
10.

Development of a potent inhibitor of 2-arachidonoylglycerol hydrolysis with antinociceptive activity in vivo.

Bisogno T, Ortar G, Petrosino S, Morera E, Palazzo E, Nalli M, Maione S, Di Marzo V; Endocannabinoid Research Group..

Biochim Biophys Acta. 2009 Jan;1791(1):53-60. doi: 10.1016/j.bbalip.2008.10.007.

PMID:
19027877
12.

Cannabinoid receptor-dependent metabolism of 2-arachidonoylglycerol during aging.

Pascual AC, Gaveglio VL, Giusto NM, Pasquaré SJ.

Exp Gerontol. 2014 Jul;55:134-42. doi: 10.1016/j.exger.2014.04.008.

PMID:
24768821
13.

Selective blockade of 2-arachidonoylglycerol hydrolysis produces cannabinoid behavioral effects.

Long JZ, Li W, Booker L, Burston JJ, Kinsey SG, Schlosburg JE, Pavón FJ, Serrano AM, Selley DE, Parsons LH, Lichtman AH, Cravatt BF.

Nat Chem Biol. 2009 Jan;5(1):37-44. doi: 10.1038/nchembio.129.

14.

Lack of selectivity of URB602 for 2-oleoylglycerol compared to anandamide hydrolysis in vitro.

Vandevoorde S, Jonsson KO, Labar G, Persson E, Lambert DM, Fowler CJ.

Br J Pharmacol. 2007 Jan;150(2):186-91.

15.

Metabolism of 2-acylglycerol in rabbit and human platelets. Involvement of monoacylglycerol lipase and fatty acid amide hydrolase.

Gkini E, Anagnostopoulos D, Mavri-Vavayianni M, Siafaka-Kapadai A.

Platelets. 2009 Sep;20(6):376-85.

PMID:
19811221
16.

A role for monoglyceride lipase in 2-arachidonoylglycerol inactivation.

Dinh TP, Freund TF, Piomelli D.

Chem Phys Lipids. 2002 Dec 31;121(1-2):149-58. Review.

PMID:
12505697
19.

Influence of the degree of unsaturation of the acyl side chain upon the interaction of analogues of 1-arachidonoylglycerol with monoacylglycerol lipase and fatty acid amide hydrolase.

Vandevoorde S, Saha B, Mahadevan A, Razdan RK, Pertwee RG, Martin BR, Fowler CJ.

Biochem Biophys Res Commun. 2005 Nov 11;337(1):104-9.

PMID:
16181610
20.
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