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Items: 1 to 20 of 117

1.

P21Waf1/Cip1 dysfunction in neuroblastoma: a novel mechanism of attenuating G0-G1 cell cycle arrest.

McKenzie PP, Danks MK, Kriwacki RW, Harris LC.

Cancer Res. 2003 Jul 1;63(13):3840-4.

2.

F9 embryonal carcinoma cells fail to stop at G1/S boundary of the cell cycle after gamma-irradiation due to p21WAF1/CIP1 degradation.

Malashicheva AB, Kislyakova TV, Aksenov ND, Osipov KA, Pospelov VA.

Oncogene. 2000 Aug 10;19(34):3858-65.

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Molecular mechanisms underlying interferon-alpha-induced G0/G1 arrest: CKI-mediated regulation of G1 Cdk-complexes and activation of pocket proteins.

Sangfelt O, Erickson S, Castro J, Heiden T, Gustafsson A, Einhorn S, Grandér D.

Oncogene. 1999 May 6;18(18):2798-810.

6.

Induction of p21/WAF1 and G1 cell-cycle arrest by the chemopreventive agent apigenin.

Lepley DM, Pelling JC.

Mol Carcinog. 1997 Jun;19(2):74-82.

PMID:
9210954
8.

Cell cycle exit during terminal erythroid differentiation is associated with accumulation of p27(Kip1) and inactivation of cdk2 kinase.

Hsieh FF, Barnett LA, Green WF, Freedman K, Matushansky I, Skoultchi AI, Kelley LL.

Blood. 2000 Oct 15;96(8):2746-54.

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Cyclin E/Cdk2 activity is controlled by different mechanisms in the G0 and G1 phases of the cell cycle.

Bresnahan WA, Boldogh I, Ma T, Albrecht T, Thompson EA.

Cell Growth Differ. 1996 Oct;7(10):1283-90.

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Kip/Cip and Ink4 Cdk inhibitors cooperate to induce cell cycle arrest in response to TGF-beta.

Reynisdóttir I, Polyak K, Iavarone A, Massagué J.

Genes Dev. 1995 Aug 1;9(15):1831-45.

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Structural studies of p21Waf1/Cip1/Sdi1 in the free and Cdk2-bound state: conformational disorder mediates binding diversity.

Kriwacki RW, Hengst L, Tennant L, Reed SI, Wright PE.

Proc Natl Acad Sci U S A. 1996 Oct 15;93(21):11504-9.

20.

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