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Items: 1 to 20 of 96

1.

Modulation of tumor necrosis factor-induced increase in renal (LLC-PK1) transepithelial permeability.

Mullin JM, Laughlin KV, Marano CW, Russo LM, Soler AP.

Am J Physiol. 1992 Nov;263(5 Pt 2):F915-24.

PMID:
1279987
2.

Different size limitations for increased transepithelial paracellular solute flux across phorbol ester and tumor necrosis factor-treated epithelial cell sheets.

Mullin JM, Marano CW, Laughlin KV, Nuciglio M, Stevenson BR, Soler P.

J Cell Physiol. 1997 May;171(2):226-33.

PMID:
9130471
3.

Long-term effects of tumor necrosis factor on LLC-PK1 transepithelial resistance.

Marano CW, Laughlin KV, Russo LM, Peralta Soler A, Mullin JM.

J Cell Physiol. 1993 Dec;157(3):519-27.

PMID:
8253863
4.

Activation of NF-kappaB is necessary for the restoration of the barrier function of an epithelium undergoing TNF-alpha-induced apoptosis.

Soler AP, Marano CW, Bryans M, Miller RD, Garulacan LA, Mauldin SK, Stamato TD, Mullin JM.

Eur J Cell Biol. 1999 Jan;78(1):56-66.

PMID:
10082424
5.
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7.

cAMP modulates transepithelial resistance response of LLC-PK1 renal epithelia to tumor necrosis factor.

Marano CW, Laughlin KV, Russo LM, Short TH, Mullin JM.

Am J Physiol. 1995 Feb;268(2 Pt 2):F315-22.

PMID:
7864172
8.

Tumor necrosis factor-α induces a biphasic change in claudin-2 expression in tubular epithelial cells: role in barrier functions.

Amoozadeh Y, Dan Q, Xiao J, Waheed F, Szászi K.

Am J Physiol Cell Physiol. 2015 Jul 1;309(1):C38-50. doi: 10.1152/ajpcell.00388.2014. Epub 2015 May 6.

9.

Chronic exposure of LLC-PK1 epithelia to the phorbol ester TPA produces polyp-like foci with leaky tight junctions and altered protein kinase C-alpha expression and localization.

Mullin JM, Soler AP, Laughlin KV, Kampherstein JA, Russo LM, Saladik DT, George K, Shurina RD, O'Brien TG.

Exp Cell Res. 1996 Aug 25;227(1):12-22.

PMID:
8806446
10.
11.

The protein kinase C inhibitor, bisindolylmaleimide, inhibits the TPA-induced but not the TNF-induced increase in LLC-PK1 transepithelial permeability.

Marano CW, Laughlin KV, Russo LM, Mullin JM.

Biochem Biophys Res Commun. 1995 Apr 17;209(2):669-76.

PMID:
7733936
12.

Transepithelial paracellular leakiness induced by chronic phorbol ester exposure correlates with polyp-like foci and redistribution of protein kinase C-alpha.

Mullin JM, Kampherstein JA, Laughlin KV, Saladik DT, Soler AP.

Carcinogenesis. 1997 Dec;18(12):2339-45.

PMID:
9450479
13.

Effects of acute vs. chronic phorbol ester exposure on transepithelial permeability and epithelial morphology.

Mullin JM, Snock KV, Shurina RD, Noe J, George K, Misner L, Imaizumi S, O'Brien TG.

J Cell Physiol. 1992 Jul;152(1):35-47.

PMID:
1618921
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17.

Overexpression of protein kinase C-delta increases tight junction permeability in LLC-PK1 epithelia.

Mullin JM, Kampherstein JA, Laughlin KV, Clarkin CE, Miller RD, Szallasi Z, Kachar B, Soler AP, Rosson D.

Am J Physiol. 1998 Aug;275(2 Pt 1):C544-54.

PMID:
9688609
18.

Protein kinase C-alpha activity modulates transepithelial permeability and cell junctions in the LLC-PK1 epithelial cell line.

Rosson D, O'Brien TG, Kampherstein JA, Szallasi Z, Bogi K, Blumberg PM, Mullin JM.

J Biol Chem. 1997 Jun 6;272(23):14950-3.

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20.

Mechanism of TNF-{alpha} modulation of Caco-2 intestinal epithelial tight junction barrier: role of myosin light-chain kinase protein expression.

Ma TY, Boivin MA, Ye D, Pedram A, Said HM.

Am J Physiol Gastrointest Liver Physiol. 2005 Mar;288(3):G422-30.

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