Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 661

1.

Roles for SR proteins and hnRNP A1 in the regulation of c-src exon N1.

Rooke N, Markovtsov V, Cagavi E, Black DL.

Mol Cell Biol. 2003 Mar;23(6):1874-84.

2.

hnRNP A1 and the SR proteins ASF/SF2 and SC35 have antagonistic functions in splicing of beta-tropomyosin exon 6B.

Expert-Bezançon A, Sureau A, Durosay P, Salesse R, Groeneveld H, Lecaer JP, Marie J.

J Biol Chem. 2004 Sep 10;279(37):38249-59. Epub 2004 Jun 18.

3.

Modulation of exon skipping and inclusion by heterogeneous nuclear ribonucleoprotein A1 and pre-mRNA splicing factor SF2/ASF.

Mayeda A, Helfman DM, Krainer AR.

Mol Cell Biol. 1993 May;13(5):2993-3001. Erratum in: Mol Cell Biol 1993 Jul;13(7):4458.

5.

hnRNP A1 functions with specificity in repression of SMN2 exon 7 splicing.

Kashima T, Rao N, David CJ, Manley JL.

Hum Mol Genet. 2007 Dec 15;16(24):3149-59. Epub 2007 Sep 19.

PMID:
17884807
7.

Selection of alternative 5' splice sites: role of U1 snRNP and models for the antagonistic effects of SF2/ASF and hnRNP A1.

Eperon IC, Makarova OV, Mayeda A, Munroe SH, Cáceres JF, Hayward DG, Krainer AR.

Mol Cell Biol. 2000 Nov;20(22):8303-18.

8.

HnRNP A1/A2 and SF2/ASF regulate alternative splicing of interferon regulatory factor-3 and affect immunomodulatory functions in human non-small cell lung cancer cells.

Guo R, Li Y, Ning J, Sun D, Lin L, Liu X.

PLoS One. 2013 Apr 29;8(4):e62729. doi: 10.1371/journal.pone.0062729. Print 2013.

9.
10.

Relative amounts of antagonistic splicing factors, hnRNP A1 and ASF/SF2, change during neoplastic lung growth: implications for pre-mRNA processing.

Zerbe LK, Pino I, Pio R, Cosper PF, Dwyer-Nield LD, Meyer AM, Port JD, Montuenga LM, Malkinson AM.

Mol Carcinog. 2004 Dec;41(4):187-96.

PMID:
15390079
11.
12.
13.

Cooperative assembly of an hnRNP complex induced by a tissue-specific homolog of polypyrimidine tract binding protein.

Markovtsov V, Nikolic JM, Goldman JA, Turck CW, Chou MY, Black DL.

Mol Cell Biol. 2000 Oct;20(20):7463-79.

14.

hnRNP A1 and hnRNP F modulate the alternative splicing of exon 11 of the insulin receptor gene.

Talukdar I, Sen S, Urbano R, Thompson J, Yates JR 3rd, Webster NJ.

PLoS One. 2011;6(11):e27869. doi: 10.1371/journal.pone.0027869. Epub 2011 Nov 23.

15.

hnRNP H and hnRNP F complex with Fox2 to silence fibroblast growth factor receptor 2 exon IIIc.

Mauger DM, Lin C, Garcia-Blanco MA.

Mol Cell Biol. 2008 Sep;28(17):5403-19. doi: 10.1128/MCB.00739-08. Epub 2008 Jun 23.

17.
19.
20.

An exon skipping-associated nonsense mutation in the dystrophin gene uncovers a complex interplay between multiple antagonistic splicing elements.

Disset A, Bourgeois CF, Benmalek N, Claustres M, Stevenin J, Tuffery-Giraud S.

Hum Mol Genet. 2006 Mar 15;15(6):999-1013. Epub 2006 Feb 6.

PMID:
16461336

Supplemental Content

Support Center