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Items: 1 to 20 of 102

1.
2.

Clinical utility of fluorescence in situ hybridization (FISH) in morphologically ambiguous gliomas with hybrid oligodendroglial/astrocytic features.

Fuller CE, Schmidt RE, Roth KA, Burger PC, Scheithauer BW, Banerjee R, Trinkaus K, Lytle R, Perry A.

J Neuropathol Exp Neurol. 2003 Nov;62(11):1118-28.

3.

PTEN protein expression correlates with PTEN gene molecular changes but not with VEGF expression in astrocytomas.

Idoate MA, Soria E, Lozano MD, Sola JJ, Panizo A, de Alava E, Manrique M, Pardo-Mindán FJ.

Diagn Mol Pathol. 2003 Sep;12(3):160-5.

PMID:
12960698
4.

PTEN, DMBT1, and p16 alterations in diffusely infiltrating astrocytomas.

Fan X, Muñoz J, Sanko SG, Castresana JS.

Int J Oncol. 2002 Sep;21(3):667-74.

PMID:
12168116
6.

Analysis of 1p, 19q, 9p, and 10q as prognostic markers for high-grade astrocytomas using fluorescence in situ hybridization on tissue microarrays from Radiation Therapy Oncology Group trials.

Brat DJ, Seiferheld WF, Perry A, Hammond EH, Murray KJ, Schulsinger AR, Mehta MP, Curran WJ; Radiation Therapy Oncology Group..

Neuro Oncol. 2004 Apr;6(2):96-103.

7.

Genetic alterations in pediatric high-grade astrocytomas.

Cheng Y, Ng HK, Zhang SF, Ding M, Pang JC, Zheng J, Poon WS.

Hum Pathol. 1999 Nov;30(11):1284-90.

PMID:
10571506
8.

Molecular pathogenesis of malignant gliomas.

Rasheed BK, Wiltshire RN, Bigner SH, Bigner DD.

Curr Opin Oncol. 1999 May;11(3):162-7. Review.

PMID:
10328589
9.

Utility of EGFR and PTEN numerical aberrations in the evaluation of diffusely infiltrating astrocytomas. Laboratory investigation.

Mott RT, Turner KC, Bigner DD, McLendon RE.

J Neurosurg. 2008 Feb;108(2):330-5. doi: 10.3171/JNS/2008/108/2/0330.

PMID:
18240930
10.

Fluorescence in situ hybridization for 1p, 19q status in a cohort of glial neoplasms.

Singh VY, Chacko G, Chacko AG, Rajshekhar V.

Neurol India. 2014 Jan-Feb;62(1):32-6. doi: 10.4103/0028-3886.128275. Erratum in: Neurol India. 2014 Mar-Apr;62(2):191.

11.

Roles of the functional loss of p53 and other genes in astrocytoma tumorigenesis and progression.

Nozaki M, Tada M, Kobayashi H, Zhang CL, Sawamura Y, Abe H, Ishii N, Van Meir EG.

Neuro Oncol. 1999 Apr;1(2):124-37. Review.

12.

Pilocytic astrocytomas do not show most of the genetic changes commonly seen in diffuse astrocytomas.

Cheng Y, Pang JC, Ng HK, Ding M, Zhang SF, Zheng J, Liu DG, Poon WS.

Histopathology. 2000 Nov;37(5):437-44.

PMID:
11119125
13.

Homozygous deletion and expression of PTEN and DMBT1 in human primary neuroblastoma and cell lines.

Muñoz J, Lázcoz P, Inda MM, Nistal M, Pestaña A, Encío IJ, Castresana JS.

Int J Cancer. 2004 May 1;109(5):673-9.

14.

PTEN mutation, EGFR amplification, and outcome in patients with anaplastic astrocytoma and glioblastoma multiforme.

Smith JS, Tachibana I, Passe SM, Huntley BK, Borell TJ, Iturria N, O'Fallon JR, Schaefer PL, Scheithauer BW, James CD, Buckner JC, Jenkins RB.

J Natl Cancer Inst. 2001 Aug 15;93(16):1246-56.

15.

Acquisition of the glioblastoma phenotype during astrocytoma progression is associated with loss of heterozygosity on 10q25-qter.

Fujisawa H, Kurrer M, Reis RM, Yonekawa Y, Kleihues P, Ohgaki H.

Am J Pathol. 1999 Aug;155(2):387-94.

16.

Molecular genetic alterations in glioblastomas with oligodendroglial component.

Kraus JA, Lamszus K, Glesmann N, Beck M, Wolter M, Sabel M, Krex D, Klockgether T, Reifenberger G, Schlegel U.

Acta Neuropathol. 2001 Apr;101(4):311-20.

PMID:
11355302
17.

PTEN and DMBT1 homozygous deletion and expression in medulloblastomas and supratentorial primitive neuroectodermal tumors.

Inda MM, Mercapide J, Muñoz J, Coullin P, Danglot G, Tuñon T, Martínez-Peñuela JM, Rivera JM, Burgos JJ, Bernheim A, Castresana JS.

Oncol Rep. 2004 Dec;12(6):1341-7.

PMID:
15547761
18.

Genomic alterations in low-grade, anaplastic astrocytomas and glioblastomas.

Arslantas A, Artan S, Oner U, Müslümanoglu MH, Ozdemir M, Durmaz R, Arslantas D, Vural M, Cosan E, Atasoy MA.

Pathol Oncol Res. 2007;13(1):39-46.

19.

Correlation of histology and molecular genetic analysis of 1p, 19q, 10q, TP53, EGFR, CDK4, and CDKN2A in 91 astrocytic and oligodendroglial tumors.

Ueki K, Nishikawa R, Nakazato Y, Hirose T, Hirato J, Funada N, Fujimaki T, Hojo S, Kubo O, Ide T, Usui M, Ochiai C, Ito S, Takahashi H, Mukasa A, Asai A, Kirino T.

Clin Cancer Res. 2002 Jan;8(1):196-201.

20.

Assessment of 1p/19q status by fluorescence in situ hybridization assay: A comparative study in oligodendroglial, mixed oligoastrocytic and astrocytic tumors.

Shukla B, Agarwal S, Suri V, Pathak P, Sharma MC, Gupta D, Sharma BS, Suri A, Halder A, Sarkar C.

Neurol India. 2009 Sep-Oct;57(5):559-66. doi: 10.4103/0028-3886.57795.

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