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Items: 1 to 20 of 99

1.

Overexpression of Helicard, a CARD-containing helicase cleaved during apoptosis, accelerates DNA degradation.

Kovacsovics M, Martinon F, Micheau O, Bodmer JL, Hofmann K, Tschopp J.

Curr Biol. 2002 May 14;12(10):838-43. Erratum in: Curr Biol. 2002 Sep 17;12(18):1633..

2.

Acinus is a caspase-3-activated protein required for apoptotic chromatin condensation.

Sahara S, Aoto M, Eguchi Y, Imamoto N, Yoneda Y, Tsujimoto Y.

Nature. 1999 Sep 9;401(6749):168-73.

PMID:
10490026
3.

Caspase-activated DNase/DNA fragmentation factor 40 mediates apoptotic DNA fragmentation in transient cerebral ischemia and in neuronal cultures.

Cao G, Pei W, Lan J, Stetler RA, Luo Y, Nagayama T, Graham SH, Yin XM, Simon RP, Chen J.

J Neurosci. 2001 Jul 1;21(13):4678-90.

5.

Involvement of caspase 3-activated DNase in internucleosomal DNA cleavage induced by diverse apoptotic stimuli.

McIlroy D, Sakahira H, Talanian RV, Nagata S.

Oncogene. 1999 Aug 5;18(31):4401-8.

6.

A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD.

Enari M, Sakahira H, Yokoyama H, Okawa K, Iwamatsu A, Nagata S.

Nature. 1998 Jan 1;391(6662):43-50. Erratum in: Nature 1998 May 28;393(6683):396.

PMID:
9422506
7.

Apoptotic nuclear morphological change without DNA fragmentation.

Sakahira H, Enari M, Ohsawa Y, Uchiyama Y, Nagata S.

Curr Biol. 1999 May 20;9(10):543-6.

8.

Auxin-induced rapid degradation of inhibitor of caspase-activated DNase (ICAD) induces apoptotic DNA fragmentation, caspase activation, and cell death: a cell suicide module.

Samejima K, Ogawa H, Ageichik AV, Peterson KL, Kaufmann SH, Kanemaki MT, Earnshaw WC.

J Biol Chem. 2014 Nov 7;289(45):31617-23. doi: 10.1074/jbc.M114.583542. Epub 2014 Sep 23.

9.

Cleavage of the Bloom's syndrome gene product during apoptosis by caspase-3 results in an impaired interaction with topoisomerase IIIalpha.

Freire R, d'Adda Di Fagagna F, Wu L, Pedrazzi G, Stagljar I, Hickson ID, Jackson SP.

Nucleic Acids Res. 2001 Aug 1;29(15):3172-80.

10.

Apoptotic DNA fragmentation.

Nagata S.

Exp Cell Res. 2000 Apr 10;256(1):12-8. Review.

PMID:
10739646
11.

BRCC2, a novel BH3-like domain-containing protein, induces apoptosis in a caspase-dependent manner.

Broustas CG, Gokhale PC, Rahman A, Dritschilo A, Ahmad I, Kasid U.

J Biol Chem. 2004 Jun 18;279(25):26780-8. Epub 2004 Apr 6.

12.

MST, a physiological caspase substrate, highly sensitizes apoptosis both upstream and downstream of caspase activation.

Lee KK, Ohyama T, Yajima N, Tsubuki S, Yonehara S.

J Biol Chem. 2001 Jun 1;276(22):19276-85. Epub 2001 Mar 7.

13.

Chromatin collapse during caspase-dependent apoptotic cell death requires DNA fragmentation factor, 40-kDa subunit-/caspase-activated deoxyribonuclease-mediated 3'-OH single-strand DNA breaks.

Iglesias-Guimarais V, Gil-Guiñon E, Sánchez-Osuna M, Casanelles E, García-Belinchón M, Comella JX, Yuste VJ.

J Biol Chem. 2013 Mar 29;288(13):9200-15. doi: 10.1074/jbc.M112.411371. Epub 2013 Feb 21.

14.

Caspase-activated DNase is necessary and sufficient for oligonucleosomal DNA breakdown, but not for chromatin disassembly during caspase-dependent apoptosis of LN-18 glioblastoma cells.

Sánchez-Osuna M, Garcia-Belinchón M, Iglesias-Guimarais V, Gil-Guiñón E, Casanelles E, Yuste VJ.

J Biol Chem. 2014 Jul 4;289(27):18752-69. doi: 10.1074/jbc.M114.550020. Epub 2014 May 17.

15.

The NS3 protease and helicase domains of Japanese encephalitis virus trigger cell death via caspase‑dependent and ‑independent pathways.

Yiang GT, Chen YH, Chou PL, Chang WJ, Wei CW, Yu YL.

Mol Med Rep. 2013 Mar;7(3):826-30. doi: 10.3892/mmr.2013.1261. Epub 2013 Jan 3.

PMID:
23291778
17.

Endonuclease G is an apoptotic DNase when released from mitochondria.

Li LY, Luo X, Wang X.

Nature. 2001 Jul 5;412(6842):95-9.

PMID:
11452314
18.

Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosis.

Sakahira H, Enari M, Nagata S.

Nature. 1998 Jan 1;391(6662):96-9. Erratum in: Nature. 2015 Oct 29;526(7575):728.

PMID:
9422513
19.

Correlation between nucleocytoplasmic transport and caspase-3-dependent dismantling of nuclear pores during apoptosis.

Kihlmark M, Rustum C, Eriksson C, Beckman M, Iverfeldt K, Hallberg E.

Exp Cell Res. 2004 Feb 15;293(2):346-56.

PMID:
14729472
20.

Oligomerization state of the DNA fragmentation factor in normal and apoptotic cells.

Lechardeur D, Dougaparsad S, Nemes C, Lukacs GL.

J Biol Chem. 2005 Dec 2;280(48):40216-25. Epub 2005 Oct 3.

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