Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 51

1.

AP-1 as a regulator of cell life and death.

Shaulian E, Karin M.

Nat Cell Biol. 2002 May;4(5):E131-6. Review.

PMID:
11988758
2.

AP-1 in cell proliferation and survival.

Shaulian E, Karin M.

Oncogene. 2001 Apr 30;20(19):2390-400. Review.

4.
5.

AP-1--The Jun proteins: Oncogenes or tumor suppressors in disguise?

Shaulian E.

Cell Signal. 2010 Jun;22(6):894-9. doi: 10.1016/j.cellsig.2009.12.008. Epub 2010 Jan 11. Review.

PMID:
20060892
6.

MAPK pathways mediate hyperoxia-induced oncotic cell death in lung epithelial cells.

Romashko J 3rd, Horowitz S, Franek WR, Palaia T, Miller EJ, Lin A, Birrer MJ, Scott W, Mantell LL.

Free Radic Biol Med. 2003 Oct 15;35(8):978-93.

PMID:
14556862
7.

PACAP and C2-ceramide generate different AP-1 complexes through a MAP-kinase-dependent pathway: involvement of c-Fos in PACAP-induced Bcl-2 expression.

Aubert N, Falluel-Morel A, Vaudry D, Xifro X, Rodriguez-Alvarez J, Fisch C, de Jouffrey S, Lebigot JF, Fournier A, Vaudry H, Gonzalez BJ.

J Neurochem. 2006 Nov;99(4):1237-50. Epub 2006 Oct 5.

8.

Mesothelial cell transformation requires increased AP-1 binding activity and ERK-dependent Fra-1 expression.

Ramos-Nino ME, Timblin CR, Mossman BT.

Cancer Res. 2002 Nov 1;62(21):6065-9.

10.

DEP-induced fra-1 expression correlates with a distinct activation of AP-1-dependent gene transcription in the lung.

Zhang Q, Kleeberger SR, Reddy SP.

Am J Physiol Lung Cell Mol Physiol. 2004 Feb;286(2):L427-36. Epub 2003 Oct 17.

PMID:
14565943
11.

Aberrantly expressed c-Jun and JunB are a hallmark of Hodgkin lymphoma cells, stimulate proliferation and synergize with NF-kappa B.

Mathas S, Hinz M, Anagnostopoulos I, Krappmann D, Lietz A, Jundt F, Bommert K, Mechta-Grigoriou F, Stein H, Dörken B, Scheidereit C.

EMBO J. 2002 Aug 1;21(15):4104-13.

12.

p38 isoforms have opposite effects on AP-1-dependent transcription through regulation of c-Jun. The determinant roles of the isoforms in the p38 MAPK signal specificity.

Pramanik R, Qi X, Borowicz S, Choubey D, Schultz RM, Han J, Chen G.

J Biol Chem. 2003 Feb 14;278(7):4831-9. Epub 2002 Dec 9.

13.

From JNK to pay dirt: jun kinases, their biochemistry, physiology and clinical importance.

Karin M, Gallagher E.

IUBMB Life. 2005 Apr-May;57(4-5):283-95. Review.

14.

Disruption of the c-JUN-JNK complex by a cell-permeable peptide containing the c-JUN delta domain induces apoptosis and affects a distinct set of interleukin-1-induced inflammatory genes.

Holzberg D, Knight CG, Dittrich-Breiholz O, Schneider H, Dörrie A, Hoffmann E, Resch K, Kracht M.

J Biol Chem. 2003 Oct 10;278(41):40213-23. Epub 2003 Jun 27.

17.

Role of c-Jun concentration in neuronal cell death.

Rössler OG, Steinmüller L, Giehl KM, Thiel G.

J Neurosci Res. 2002 Dec 1;70(5):655-64.

PMID:
12424733
18.
19.

Activation of the JNK-AP-1 signal transduction pathway is associated with pathogenesis and progression of human osteosarcomas.

Papachristou DJ, Batistatou A, Sykiotis GP, Varakis I, Papavassiliou AG.

Bone. 2003 Apr;32(4):364-71.

PMID:
12689679
20.

MAP kinase-dependent, NF-kappaB-independent regulation of inhibitor of apoptosis protein genes by TNF-alpha.

Furusu A, Nakayama K, Xu Q, Konta T, Kitamura M.

J Cell Physiol. 2007 Mar;210(3):703-10.

PMID:
17133355

Supplemental Content

Support Center