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Murine cytomegalovirus is regulated by a discrete subset of natural killer cells reactive with monoclonal antibody to Ly49H.

Daniels KA, Devora G, Lai WC, O'Donnell CL, Bennett M, Welsh RM.

J Exp Med. 2001 Jul 2;194(1):29-44.


The role of LY49 NK cell subsets in the regulation of murine cytomegalovirus infections.

Tay CH, Yu LY, Kumar V, Mason L, Ortaldo JR, Welsh RM.

J Immunol. 1999 Jan 15;162(2):718-26.


Cmv1-independent antiviral role of NK cells revealed in murine cytomegalovirus-infected New Zealand White mice.

Rodriguez M, Sabastian P, Clark P, Brown MG.

J Immunol. 2004 Nov 15;173(10):6312-8.


Specific and nonspecific NK cell activation during virus infection.

Dokun AO, Kim S, Smith HR, Kang HS, Chu DT, Yokoyama WM.

Nat Immunol. 2001 Oct;2(10):951-6.


Coordinate expression of cytokines and chemokines by NK cells during murine cytomegalovirus infection.

Dorner BG, Smith HR, French AR, Kim S, Poursine-Laurent J, Beckman DL, Pingel JT, Kroczek RA, Yokoyama WM.

J Immunol. 2004 Mar 1;172(5):3119-31.


Assessment of Cmv1 candidates by genetic mapping and in vivo antibody depletion of NK cell subsets.

Depatie C, Chalifour A, Paré C, Lee SH, Vidal SM, Lemieux S.

Int Immunol. 1999 Sep;11(9):1541-51.


Continuous engagement of a self-specific activation receptor induces NK cell tolerance.

Tripathy SK, Keyel PA, Yang L, Pingel JT, Cheng TP, Schneeberger A, Yokoyama WM.

J Exp Med. 2008 Aug 4;205(8):1829-41. doi: 10.1084/jem.20072446. Epub 2008 Jul 7.


Cytokine-Mediated Activation of NK Cells during Viral Infection.

Freeman BE, Raué HP, Hill AB, Slifka MK.

J Virol. 2015 Aug;89(15):7922-31. doi: 10.1128/JVI.00199-15. Epub 2015 May 20.


Gain of virulence caused by loss of a gene in murine cytomegalovirus.

Bubić I, Wagner M, Krmpotić A, Saulig T, Kim S, Yokoyama WM, Jonjić S, Koszinowski UH.

J Virol. 2004 Jul;78(14):7536-44.


Direct recognition of cytomegalovirus by activating and inhibitory NK cell receptors.

Arase H, Mocarski ES, Campbell AE, Hill AB, Lanier LL.

Science. 2002 May 17;296(5571):1323-6. Epub 2002 Apr 11.


Ly49h-deficient C57BL/6 mice: a new mouse cytomegalovirus-susceptible model remains resistant to unrelated pathogens controlled by the NK gene complex.

Fodil-Cornu N, Lee SH, Belanger S, Makrigiannis AP, Biron CA, Buller RM, Vidal SM.

J Immunol. 2008 Nov 1;181(9):6394-405.


Transgenic expression of the activating natural killer receptor Ly49H confers resistance to cytomegalovirus in genetically susceptible mice.

Lee SH, Zafer A, de Repentigny Y, Kothary R, Tremblay ML, Gros P, Duplay P, Webb JR, Vidal SM.

J Exp Med. 2003 Feb 17;197(4):515-26.


Natural killer cell depletion enhances virus synthesis and virus-induced hepatitis in vivo.

Bukowski JF, Woda BA, Habu S, Okumura K, Welsh RM.

J Immunol. 1983 Sep;131(3):1531-8.


Susceptibility to mouse cytomegalovirus is associated with deletion of an activating natural killer cell receptor of the C-type lectin superfamily.

Lee SH, Girard S, Macina D, Busà M, Zafer A, Belouchi A, Gros P, Vidal SM.

Nat Genet. 2001 May;28(1):42-5.


Natural Killer Cell Sensing of Infected Cells Compensates for MyD88 Deficiency but Not IFN-I Activity in Resistance to Mouse Cytomegalovirus.

Cocita C, Guiton R, Bessou G, Chasson L, Boyron M, Crozat K, Dalod M.

PLoS Pathog. 2015 May 8;11(5):e1004897. doi: 10.1371/journal.ppat.1004897. eCollection 2015 May.


Ly49H engagement compensates for the absence of type I interferon signaling in stimulating NK cell proliferation during murine cytomegalovirus infection.

Geurs TL, Zhao YM, Hill EB, French AR.

J Immunol. 2009 Nov 1;183(9):5830-6. doi: 10.4049/jimmunol.0901520. Epub 2009 Oct 14.

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