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Items: 1 to 20 of 695

1.

A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage.

Paull TT, Rogakou EP, Yamazaki V, Kirchgessner CU, Gellert M, Bonner WM.

Curr Biol. 2000 Jul 27-Aug 10;10(15):886-95.

2.

Phosphorylation of histone H2AX and activation of Mre11, Rad50, and Nbs1 in response to replication-dependent DNA double-strand breaks induced by mammalian DNA topoisomerase I cleavage complexes.

Furuta T, Takemura H, Liao ZY, Aune GJ, Redon C, Sedelnikova OA, Pilch DR, Rogakou EP, Celeste A, Chen HT, Nussenzweig A, Aladjem MI, Bonner WM, Pommier Y.

J Biol Chem. 2003 May 30;278(22):20303-12. Epub 2003 Mar 25.

3.
4.

Sub-nuclear localization of Rad51 in response to DNA damage.

Mladenov E, Anachkova B, Tsaneva I.

Genes Cells. 2006 May;11(5):513-24.

5.

DNA double-strand breaks and gamma-H2AX signaling in the testis.

Hamer G, Roepers-Gajadien HL, van Duyn-Goedhart A, Gademan IS, Kal HB, van Buul PP, de Rooij DG.

Biol Reprod. 2003 Feb;68(2):628-34.

PMID:
12533428
6.

DNA damage-induced RPA focalization is independent of gamma-H2AX and RPA hyper-phosphorylation.

Liu JS, Kuo SR, Melendy T.

J Cell Biochem. 2006 Dec 1;99(5):1452-62.

PMID:
16927366
7.

Identification of sequences that target BRCA1 to nuclear foci following alkylative DNA damage.

Au WW, Henderson BR.

Cell Signal. 2007 Sep;19(9):1879-92. Epub 2007 May 1.

PMID:
17531442
8.

Collaborative roles of gammaH2AX and the Rad51 paralog Xrcc3 in homologous recombinational repair.

Sonoda E, Zhao GY, Kohzaki M, Dhar PK, Kikuchi K, Redon C, Pilch DR, Bonner WM, Nakano A, Watanabe M, Nakayama T, Takeda S, Takami Y.

DNA Repair (Amst). 2007 Mar 1;6(3):280-92. Epub 2006 Nov 22.

PMID:
17123873
9.

Long-term in vivo effects of cisplatin on gamma-H2AX foci signaling in peripheral lymphocytes of tumor patients after irradiation.

Sak A, Grehl S, Engelhard M, Wierlemann A, Kaelberlah HP, Erichsen P, Pöttgen C, Groneberg M, Stuschke M.

Clin Cancer Res. 2009 Apr 15;15(8):2927-34. doi: 10.1158/1078-0432.CCR-08-0650. Epub 2009 Mar 31.

10.

Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks.

Celeste A, Fernandez-Capetillo O, Kruhlak MJ, Pilch DR, Staudt DW, Lee A, Bonner RF, Bonner WM, Nussenzweig A.

Nat Cell Biol. 2003 Jul;5(7):675-9.

PMID:
12792649
11.

Ionizing radiation induces DNA double-strand breaks in bystander primary human fibroblasts.

Sokolov MV, Smilenov LB, Hall EJ, Panyutin IG, Bonner WM, Sedelnikova OA.

Oncogene. 2005 Nov 10;24(49):7257-65.

PMID:
16170376
13.

Histone H2AX phosphorylation in normal human cells irradiated with focused ultrasoft X rays: evidence for chromatin movement during repair.

Hamada N, Schettino G, Kashino G, Vaid M, Suzuki K, Kodama S, Vojnovic B, Folkard M, Watanabe M, Michael BD, Prise KM.

Radiat Res. 2006 Jul;166(1 Pt 1):31-8.

PMID:
16808616
14.

End-joining of reconstituted histone H2AX-containing chromatin in vitro by soluble nuclear proteins from human cells.

Siino JS, Nazarov IB, Zalenskaya IA, Yau PM, Bradbury EM, Tomilin NV.

FEBS Lett. 2002 Sep 11;527(1-3):105-8.

15.

Kinetics of gamma-H2AX focus formation upon treatment of cells with UV light and alkylating agents.

Staszewski O, Nikolova T, Kaina B.

Environ Mol Mutagen. 2008 Dec;49(9):734-40. doi: 10.1002/em.20430.

PMID:
18800352
16.

Histone H2AX is phosphorylated in an ATR-dependent manner in response to replicational stress.

Ward IM, Chen J.

J Biol Chem. 2001 Dec 21;276(51):47759-62. Epub 2001 Oct 22.

17.

RAD51 foci formation in response to DNA damage is modulated by TIP49.

Gospodinov A, Tsaneva I, Anachkova B.

Int J Biochem Cell Biol. 2009 Apr;41(4):925-33. doi: 10.1016/j.biocel.2008.09.004. Epub 2008 Sep 13.

PMID:
18834951
18.

gamma-H2AX as a therapeutic target for improving the efficacy of radiation therapy.

Kao J, Milano MT, Javaheri A, Garofalo MC, Chmura SJ, Weichselbaum RR, Kron SJ.

Curr Cancer Drug Targets. 2006 May;6(3):197-205. Review.

PMID:
16712457
19.

Gamma-H2AX - a novel biomarker for DNA double-strand breaks.

Kuo LJ, Yang LX.

In Vivo. 2008 May-Jun;22(3):305-9. Review.

20.

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