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Items: 1 to 20 of 247

1.

Regulation of E2F1 activity by acetylation.

Martínez-Balbás MA, Bauer UM, Nielsen SJ, Brehm A, Kouzarides T.

EMBO J. 2000 Feb 15;19(4):662-71.

2.

RbAp48 belongs to the histone deacetylase complex that associates with the retinoblastoma protein.

Nicolas E, Morales V, Magnaghi-Jaulin L, Harel-Bellan A, Richard-Foy H, Trouche D.

J Biol Chem. 2000 Mar 31;275(13):9797-804.

3.

E2F transcriptional activation requires TRRAP and GCN5 cofactors.

Lang SE, McMahon SB, Cole MD, Hearing P.

J Biol Chem. 2001 Aug 31;276(35):32627-34. Epub 2001 Jun 19.

4.

E2F family members are differentially regulated by reversible acetylation.

Marzio G, Wagener C, Gutierrez MI, Cartwright P, Helin K, Giacca M.

J Biol Chem. 2000 Apr 14;275(15):10887-92.

5.

CBP/p300 histone acetyl-transferase activity is important for the G1/S transition.

Ait-Si-Ali S, Polesskaya A, Filleur S, Ferreira R, Duquet A, Robin P, Vervish A, Trouche D, Cabon F, Harel-Bellan A.

Oncogene. 2000 May 11;19(20):2430-7.

6.

A genetic analysis of the E2F1 gene distinguishes regulation by Rb, p107, and adenovirus E4.

Cress WD, Johnson DG, Nevins JR.

Mol Cell Biol. 1993 Oct;13(10):6314-25.

7.

Multiple DNA elements are required for the growth regulation of the mouse E2F1 promoter.

Hsiao KM, McMahon SL, Farnham PJ.

Genes Dev. 1994 Jul 1;8(13):1526-37.

8.

E2F1 and E1A(12S) have a homologous activation domain regulated by RB and CBP.

Trouche D, Kouzarides T.

Proc Natl Acad Sci U S A. 1996 Feb 20;93(4):1439-42.

9.

Specific role for p300/CREB-binding protein-associated factor activity in E2F1 stabilization in response to DNA damage.

Ianari A, Gallo R, Palma M, Alesse E, Gulino A.

J Biol Chem. 2004 Jul 16;279(29):30830-5. Epub 2004 May 3.

10.

Retinoblastoma protein represses transcription by recruiting a histone deacetylase.

Magnaghi-Jaulin L, Groisman R, Naguibneva I, Robin P, Lorain S, Le Villain JP, Troalen F, Trouche D, Harel-Bellan A.

Nature. 1998 Feb 5;391(6667):601-5.

PMID:
9468140
11.
12.

The CBP co-activator stimulates E2F1/DP1 activity.

Trouche D, Cook A, Kouzarides T.

Nucleic Acids Res. 1996 Nov 1;24(21):4139-45.

13.

The three members of the pocket proteins family share the ability to repress E2F activity through recruitment of a histone deacetylase.

Ferreira R, Magnaghi-Jaulin L, Robin P, Harel-Bellan A, Trouche D.

Proc Natl Acad Sci U S A. 1998 Sep 1;95(18):10493-8.

14.

E2F4-RB and E2F4-p107 complexes suppress gene expression by transforming growth factor beta through E2F binding sites.

Li JM, Hu PP, Shen X, Yu Y, Wang XF.

Proc Natl Acad Sci U S A. 1997 May 13;94(10):4948-53.

15.

Retinoblastoma protein recruits histone deacetylase to repress transcription.

Brehm A, Miska EA, McCance DJ, Reid JL, Bannister AJ, Kouzarides T.

Nature. 1998 Feb 5;391(6667):597-601.

PMID:
9468139
16.

Functional interplay between p53 and E2F through co-activator p300.

Lee CW, Sørensen TS, Shikama N, La Thangue NB.

Oncogene. 1998 May 28;16(21):2695-710.

17.

Regulation of the cyclin E gene by transcription factor E2F1.

Ohtani K, DeGregori J, Nevins JR.

Proc Natl Acad Sci U S A. 1995 Dec 19;92(26):12146-50.

19.

A potent transrepression domain in the retinoblastoma protein induces a cell cycle arrest when bound to E2F sites.

Sellers WR, Rodgers JW, Kaelin WG Jr.

Proc Natl Acad Sci U S A. 1995 Dec 5;92(25):11544-8.

20.

Acetylation of the BETA2 transcription factor by p300-associated factor is important in insulin gene expression.

Qiu Y, Guo M, Huang S, Stein R.

J Biol Chem. 2004 Mar 12;279(11):9796-802. Epub 2003 Dec 30.

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