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Items: 1 to 20 of 96

1.

IL-36 and IL-1/IL-17 Drive Immunity to Oral Candidiasis via Parallel Mechanisms.

Verma AH, Zafar H, Ponde NO, Hepworth OW, Sihra D, Aggor FEY, Ainscough JS, Ho J, Richardson JP, Coleman BM, Hube B, Stacey M, McGeachy MJ, Naglik JR, Gaffen SL, Moyes DL.

J Immunol. 2018 Jul 15;201(2):627-634. doi: 10.4049/jimmunol.1800515. Epub 2018 Jun 11.

2.

Candidalysin Drives Epithelial Signaling, Neutrophil Recruitment, and Immunopathology at the Vaginal Mucosa.

Richardson JP, Willems HME, Moyes DL, Shoaie S, Barker KS, Tan SL, Palmer GE, Hube B, Naglik JR, Peters BM.

Infect Immun. 2018 Jan 22;86(2). pii: e00645-17. doi: 10.1128/IAI.00645-17. Print 2018 Feb.

3.

Oral epithelial cells orchestrate innate type 17 responses to Candida albicans through the virulence factor candidalysin.

Verma AH, Richardson JP, Zhou C, Coleman BM, Moyes DL, Ho J, Huppler AR, Ramani K, McGeachy MJ, Mufazalov IA, Waisman A, Kane LP, Biswas PS, Hube B, Naglik JR, Gaffen SL.

Sci Immunol. 2017 Nov 3;2(17). pii: eaam8834. doi: 10.1126/sciimmunol.aam8834.

4.

Processing of Candida albicans Ece1p Is Critical for Candidalysin Maturation and Fungal Virulence.

Richardson JP, Mogavero S, Moyes DL, Blagojevic M, Krüger T, Verma AH, Coleman BM, De La Cruz Diaz J, Schulz D, Ponde NO, Carrano G, Kniemeyer O, Wilson D, Bader O, Enoiu SI, Ho J, Kichik N, Gaffen SL, Hube B, Naglik JR.

MBio. 2018 Jan 23;9(1). pii: e02178-17. doi: 10.1128/mBio.02178-17.

5.

Activation of MAPK/c-Fos induced responses in oral epithelial cells is specific to Candida albicans and Candida dubliniensis hyphae.

Moyes DL, Murciano C, Runglall M, Kohli A, Islam A, Naglik JR.

Med Microbiol Immunol. 2012 Feb;201(1):93-101. doi: 10.1007/s00430-011-0209-y. Epub 2011 Jun 25.

6.

Candida albicans yeast and hyphae are discriminated by MAPK signaling in vaginal epithelial cells.

Moyes DL, Murciano C, Runglall M, Islam A, Thavaraj S, Naglik JR.

PLoS One. 2011;6(11):e26580. doi: 10.1371/journal.pone.0026580. Epub 2011 Nov 8.

7.

A biphasic innate immune MAPK response discriminates between the yeast and hyphal forms of Candida albicans in epithelial cells.

Moyes DL, Runglall M, Murciano C, Shen C, Nayar D, Thavaraj S, Kohli A, Islam A, Mora-Montes H, Challacombe SJ, Naglik JR.

Cell Host Microbe. 2010 Sep 16;8(3):225-35. doi: 10.1016/j.chom.2010.08.002.

8.

Candida albicans-epithelial interactions and induction of mucosal innate immunity.

Naglik JR, König A, Hube B, Gaffen SL.

Curr Opin Microbiol. 2017 Dec;40:104-112. doi: 10.1016/j.mib.2017.10.030. Epub 2017 Nov 17. Review.

9.

Probiotic lactobacillus and estrogen effects on vaginal epithelial gene expression responses to Candida albicans.

Wagner RD, Johnson SJ.

J Biomed Sci. 2012 Jun 20;19:58. doi: 10.1186/1423-0127-19-58. Erratum in: J Biomed Sci. 2012;19:84.

10.

Candida albicans cell wall glycosylation may be indirectly required for activation of epithelial cell proinflammatory responses.

Murciano C, Moyes DL, Runglall M, Islam A, Mille C, Fradin C, Poulain D, Gow NA, Naglik JR.

Infect Immun. 2011 Dec;79(12):4902-11. doi: 10.1128/IAI.05591-11. Epub 2011 Sep 19.

11.

The adaptor CARD9 is required for adaptive but not innate immunity to oral mucosal Candida albicans infections.

Bishu S, Hernández-Santos N, Simpson-Abelson MR, Huppler AR, Conti HR, Ghilardi N, Mamo AJ, Gaffen SL.

Infect Immun. 2014 Mar;82(3):1173-80. doi: 10.1128/IAI.01335-13. Epub 2013 Dec 30.

12.

Defective IL-17- and IL-22-dependent mucosal host response to Candida albicans determines susceptibility to oral candidiasis in mice expressing the HIV-1 transgene.

Goupil M, Cousineau-Côté V, Aumont F, Sénéchal S, Gaboury L, Hanna Z, Jolicoeur P, de Repentigny L.

BMC Immunol. 2014 Oct 26;15:49. doi: 10.1186/s12865-014-0049-9.

13.

Immunity to Candida.

Fidel PL Jr.

Oral Dis. 2002;8 Suppl 2:69-75. Review.

PMID:
12164664
14.

β-Defensin 1 plays a role in acute mucosal defense against Candida albicans.

Tomalka J, Azodi E, Narra HP, Patel K, O'Neill S, Cardwell C, Hall BA, Wilson JM, Hise AG.

J Immunol. 2015 Feb 15;194(4):1788-95. doi: 10.4049/jimmunol.1203239. Epub 2015 Jan 16.

15.

Candida albicans β-Glucan-Containing Particles Increase HO-1 Expression in Oral Keratinocytes via a Reactive Oxygen Species/p38 Mitogen-Activated Protein Kinase/Nrf2 Pathway.

Ishida Y, Ohta K, Naruse T, Kato H, Fukui A, Shigeishi H, Nishi H, Tobiume K, Takechi M.

Infect Immun. 2018 Mar 22;86(4). pii: e00575-17. doi: 10.1128/IAI.00575-17. Print 2018 Apr.

16.

Oral candidosis in relation to oral immunity.

Feller L, Khammissa RA, Chandran R, Altini M, Lemmer J.

J Oral Pathol Med. 2014 Sep;43(8):563-9. doi: 10.1111/jop.12120. Epub 2013 Oct 9. Review.

PMID:
24118267
17.

Involvement of interleukin-18 in the inflammatory response against oropharyngeal candidiasis.

Tardif F, Goulet JP, Zakrazewski A, Chauvin P, Rouabhia M.

Med Sci Monit. 2004 Aug;10(8):BR239-49. Epub 2004 Jul 23.

PMID:
15277983
18.

Protection against epithelial damage during Candida albicans infection is mediated by PI3K/Akt and mammalian target of rapamycin signaling.

Moyes DL, Shen C, Murciano C, Runglall M, Richardson JP, Arno M, Aldecoa-Otalora E, Naglik JR.

J Infect Dis. 2014 Jun 1;209(11):1816-26. doi: 10.1093/infdis/jit824. Epub 2013 Dec 19.

19.

Initiation of T-helper cell immunity to Candida albicans by IL-12: the role of neutrophils.

Romani L, Bistoni F, Puccetti P.

Chem Immunol. 1997;68:110-35. Review.

PMID:
9329219
20.

Antibody blockade of IL-17 family cytokines in immunity to acute murine oral mucosal candidiasis.

Whibley N, Tritto E, Traggiai E, Kolbinger F, Moulin P, Brees D, Coleman BM, Mamo AJ, Garg AV, Jaycox JR, Siebenlist U, Kammüller M, Gaffen SL.

J Leukoc Biol. 2016 Jun;99(6):1153-64. doi: 10.1189/jlb.4A0915-428R. Epub 2016 Jan 4. Erratum in: J Leukoc Biol. 2017 Apr;101(4):1065.

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