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Items: 1 to 20 of 119

1.

Activated renal tubular Wnt/β-catenin signaling triggers renal inflammation during overload proteinuria.

Wong DWL, Yiu WH, Chan KW, Li Y, Li B, Lok SWY, Taketo MM, Igarashi P, Chan LYY, Leung JCK, Lai KN, Tang SCW.

Kidney Int. 2018 Jun;93(6):1367-1383. doi: 10.1016/j.kint.2017.12.017. Epub 2018 Mar 28.

PMID:
29605095
2.

Downregulation of renal tubular Wnt/β-catenin signaling by Dickkopf-3 induces tubular cell death in proteinuric nephropathy.

Wong DW, Yiu WH, Wu HJ, Li RX, Liu Y, Chan KW, Leung JC, Chan LY, Lai KN, Tang SC.

Cell Death Dis. 2016 Mar 24;7:e2155. doi: 10.1038/cddis.2016.62.

3.
4.

Multiple genes of the renin-angiotensin system are novel targets of Wnt/β-catenin signaling.

Zhou L, Li Y, Hao S, Zhou D, Tan RJ, Nie J, Hou FF, Kahn M, Liu Y.

J Am Soc Nephrol. 2015 Jan;26(1):107-20. doi: 10.1681/ASN.2014010085. Epub 2014 Jul 10.

5.

NLRP3 inflammasome mediates albumin-induced renal tubular injury through impaired mitochondrial function.

Zhuang Y, Ding G, Zhao M, Bai M, Yang L, Ni J, Wang R, Jia Z, Huang S, Zhang A.

J Biol Chem. 2014 Sep 5;289(36):25101-11. doi: 10.1074/jbc.M114.578260. Epub 2014 Jul 24.

6.

Enalapril inhibits tubulointerstitial inflammation and NLRP3 inflammasome expression in BSA-overload nephropathy of rats.

Ding LH, Liu D, Xu M, Liu H, Wu M, Tang RN, Lv LL, Ma KL, Liu BC.

Acta Pharmacol Sin. 2014 Oct;35(10):1293-301. doi: 10.1038/aps.2014.66. Epub 2014 Aug 25.

7.

Kidneys with heavy proteinuria show fibrosis, inflammation, and oxidative stress, but no tubular phenotypic change.

Kuusniemi AM, Lapatto R, Holmberg C, Karikoski R, Rapola J, Jalanko H.

Kidney Int. 2005 Jul;68(1):121-32.

8.

Leucine rich α-2 glycoprotein is a potential urinary biomarker for renal tubular injury.

Lee H, Fujimoto M, Ohkawara T, Honda H, Serada S, Terada Y, Naka T.

Biochem Biophys Res Commun. 2018 Apr 15;498(4):1045-1051. doi: 10.1016/j.bbrc.2018.03.111. Epub 2018 Mar 17.

PMID:
29550485
9.

Blockade of Wnt/β-catenin signaling by paricalcitol ameliorates proteinuria and kidney injury.

He W, Kang YS, Dai C, Liu Y.

J Am Soc Nephrol. 2011 Jan;22(1):90-103. doi: 10.1681/ASN.2009121236. Epub 2010 Oct 28.

10.

Aberrant activation of the WNT/β-catenin signaling pathway in lupus nephritis.

Wang XD, Huang XF, Yan QR, Bao CD.

PLoS One. 2014 Jan 21;9(1):e84852. doi: 10.1371/journal.pone.0084852. eCollection 2014.

11.

Hyperuricemia-induced NLRP3 activation of macrophages contributes to the progression of diabetic nephropathy.

Kim SM, Lee SH, Kim YG, Kim SY, Seo JW, Choi YW, Kim DJ, Jeong KH, Lee TW, Ihm CG, Won KY, Moon JY.

Am J Physiol Renal Physiol. 2015 May 1;308(9):F993-F1003. doi: 10.1152/ajprenal.00637.2014. Epub 2015 Jan 28.

12.

Uric acid induces renal inflammation via activating tubular NF-κB signaling pathway.

Zhou Y, Fang L, Jiang L, Wen P, Cao H, He W, Dai C, Yang J.

PLoS One. 2012;7(6):e39738. doi: 10.1371/journal.pone.0039738. Epub 2012 Jun 25.

13.

Diverse roles of TGF-β receptor II in renal fibrosis and inflammation in vivo and in vitro.

Meng XM, Huang XR, Xiao J, Chen HY, Zhong X, Chung AC, Lan HY.

J Pathol. 2012 Jun;227(2):175-88. doi: 10.1002/path.3976. Epub 2012 Feb 22.

PMID:
22190171
14.

Implication of dysregulation of the canonical wingless-type MMTV integration site (WNT) pathway in diabetic nephropathy.

Zhou T, He X, Cheng R, Zhang B, Zhang RR, Chen Y, Takahashi Y, Murray AR, Lee K, Gao G, Ma JX.

Diabetologia. 2012 Jan;55(1):255-66. doi: 10.1007/s00125-011-2314-2. Epub 2011 Oct 21.

PMID:
22016045
15.

AMPKα2 reduces renal epithelial transdifferentiation and inflammation after injury through interaction with CK2β.

Qiu S, Xiao Z, Piao C, Zhang J, Dong Y, Cui W, Liu X, Zhang Y, Du J.

J Pathol. 2015 Nov;237(3):330-42. doi: 10.1002/path.4579. Epub 2015 Jul 23.

PMID:
26108355
16.

Role of RAS/Wnt/β-catenin axis activation in the pathogenesis of podocyte injury and tubulo-interstitial nephropathy.

Chen L, Chen DQ, Wang M, Liu D, Chen H, Dou F, Vaziri ND, Zhao YY.

Chem Biol Interact. 2017 Aug 1;273:56-72. doi: 10.1016/j.cbi.2017.05.025. Epub 2017 Jun 2.

PMID:
28578904
17.

Activation of the Nlrp3 inflammasome by mitochondrial reactive oxygen species: a novel mechanism of albumin-induced tubulointerstitial inflammation.

Liu D, Xu M, Ding LH, Lv LL, Liu H, Ma KL, Zhang AH, Crowley SD, Liu BC.

Int J Biochem Cell Biol. 2014 Dec;57:7-19. doi: 10.1016/j.biocel.2014.09.018. Epub 2014 Oct 2.

18.

Tubule-specific ablation of endogenous β-catenin aggravates acute kidney injury in mice.

Zhou D, Li Y, Lin L, Zhou L, Igarashi P, Liu Y.

Kidney Int. 2012 Sep;82(5):537-47. doi: 10.1038/ki.2012.173. Epub 2012 May 23. Erratum in: Kidney Int. 2013 Sep;84(3):626.

19.

VDR Activation Reduces Proteinuria and High-Glucose-Induced Injury of Kidneys and Podocytes by Regulating Wnt Signaling Pathway.

Guo J, Lu C, Zhang F, Yu H, Zhou M, He M, Wang C, Zhao Z, Liu Z.

Cell Physiol Biochem. 2017;43(1):39-51. doi: 10.1159/000480315. Epub 2017 Aug 24.

20.

Mitochondrial dysfunction confers albumin-induced NLRP3 inflammasome activation and renal tubular injury.

Zhuang Y, Yasinta M, Hu C, Zhao M, Ding G, Bai M, Yang L, Ni J, Wang R, Jia Z, Huang S, Zhang A.

Am J Physiol Renal Physiol. 2015 Apr 15;308(8):F857-66. doi: 10.1152/ajprenal.00203.2014. Epub 2015 Feb 18.

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