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Items: 1 to 20 of 100

1.

A novel kinase function of a nucleoside-diphosphate-kinase homologue in Porphyromonas gingivalis is critical in subversion of host cell apoptosis by targeting heat-shock protein 27.

Lee J, Roberts JS, Atanasova KR, Chowdhury N, Yilmaz Ö.

Cell Microbiol. 2018 May;20(5):e12825. doi: 10.1111/cmi.12825. Epub 2018 Feb 7.

PMID:
29359393
2.

Nucleoside-Diphosphate-Kinase of P. gingivalis is Secreted from Epithelial Cells In the Absence of a Leader Sequence Through a Pannexin-1 Interactome.

Atanasova K, Lee J, Roberts J, Lee K, Ojcius DM, Yilmaz Ö.

Sci Rep. 2016 Nov 24;6:37643. doi: 10.1038/srep37643.

3.

Porphyromonas gingivalis-nucleoside-diphosphate-kinase inhibits ATP-induced reactive-oxygen-species via P2X7 receptor/NADPH-oxidase signalling and contributes to persistence.

Choi CH, Spooner R, DeGuzman J, Koutouzis T, Ojcius DM, Yilmaz Ö.

Cell Microbiol. 2013 Jun;15(6):961-76. doi: 10.1111/cmi.12089. Epub 2013 Jan 9.

4.

Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.

Roberts JS, Atanasova KR, Lee J, Diamond G, Deguzman J, Hee Choi C, Yilmaz Ö.

Front Cell Infect Microbiol. 2017 Jul 4;7:291. doi: 10.3389/fcimb.2017.00291. eCollection 2017.

5.

Porphyromonas gingivalis attenuates ATP-mediated inflammasome activation and HMGB1 release through expression of a nucleoside-diphosphate kinase.

Johnson L, Atanasova KR, Bui PQ, Lee J, Hung SC, Yilmaz Ö, Ojcius DM.

Microbes Infect. 2015 May;17(5):369-77. doi: 10.1016/j.micinf.2015.03.010. Epub 2015 Mar 27.

6.

Porphyromonas gingivalis infection sequesters pro-apoptotic Bad through Akt in primary gingival epithelial cells.

Yao L, Jermanus C, Barbetta B, Choi C, Verbeke P, Ojcius DM, Yilmaz O.

Mol Oral Microbiol. 2010 Apr;25(2):89-101. doi: 10.1111/j.2041-1014.2010.00569.x.

8.

Porphyromonas gingivalis traffics into endoplasmic reticulum-rich-autophagosomes for successful survival in human gingival epithelial cells.

Lee K, Roberts JS, Choi CH, Atanasova KR, Yilmaz Ö.

Virulence. 2018 Dec 31;9(1):845-859. doi: 10.1080/21505594.2018.1454171.

9.

ATP scavenging by the intracellular pathogen Porphyromonas gingivalis inhibits P2X7-mediated host-cell apoptosis.

Yilmaz O, Yao L, Maeda K, Rose TM, Lewis EL, Duman M, Lamont RJ, Ojcius DM.

Cell Microbiol. 2008 Apr;10(4):863-75. Epub 2007 Nov 13.

10.

Nucleoside diphosphate kinase (Ndk): A pleiotropic effector manipulating bacterial virulence and adaptive responses.

Yu H, Rao X, Zhang K.

Microbiol Res. 2017 Dec;205:125-134. doi: 10.1016/j.micres.2017.09.001. Epub 2017 Sep 8. Review.

11.

Porphyromonas gingivalis SerB-mediated dephosphorylation of host cell cofilin modulates invasion efficiency.

Moffatt CE, Inaba H, Hirano T, Lamont RJ.

Cell Microbiol. 2012 Apr;14(4):577-88. doi: 10.1111/j.1462-5822.2011.01743.x. Epub 2012 Feb 2.

12.

Cell Cycle Arrest and Apoptosis Induced by Porphyromonas gingivalis Require Jun N-Terminal Protein Kinase- and p53-Mediated p38 Activation in Human Trophoblasts.

Inaba H, Amano A, Lamont RJ, Murakami Y, Matsumoto-Nakano M.

Infect Immun. 2018 Mar 22;86(4). pii: e00923-17. doi: 10.1128/IAI.00923-17. Print 2018 Apr.

13.

Intrinsic apoptotic pathways of gingival epithelial cells modulated by Porphyromonas gingivalis.

Mao S, Park Y, Hasegawa Y, Tribble GD, James CE, Handfield M, Stavropoulos MF, Yilmaz O, Lamont RJ.

Cell Microbiol. 2007 Aug;9(8):1997-2007. Epub 2007 Apr 5.

14.

Genetic transformation of an obligate anaerobe, P. gingivalis for FMN-green fluorescent protein expression in studying host-microbe interaction.

Choi CH, DeGuzman JV, Lamont RJ, Yilmaz Ö.

PLoS One. 2011 Apr 15;6(4):e18499. doi: 10.1371/journal.pone.0018499.

15.

Noncanonical activation of β-catenin by Porphyromonas gingivalis.

Zhou Y, Sztukowska M, Wang Q, Inaba H, Potempa J, Scott DA, Wang H, Lamont RJ.

Infect Immun. 2015 Aug;83(8):3195-203. doi: 10.1128/IAI.00302-15. Epub 2015 Jun 1.

16.

Inhibition of epithelial cell apoptosis by Porphyromonas gingivalis.

Nakhjiri SF, Park Y, Yilmaz O, Chung WO, Watanabe K, El-Sabaeny A, Park K, Lamont RJ.

FEMS Microbiol Lett. 2001 Jun 25;200(2):145-9.

17.

Mycobacterium tuberculosis nucleoside diphosphate kinase inactivates small GTPases leading to evasion of innate immunity.

Sun J, Singh V, Lau A, Stokes RW, Obregón-Henao A, Orme IM, Wong D, Av-Gay Y, Hmama Z.

PLoS Pathog. 2013;9(7):e1003499. doi: 10.1371/journal.ppat.1003499. Epub 2013 Jul 18.

19.

Human Primary Epithelial Cells Acquire an Epithelial-Mesenchymal-Transition Phenotype during Long-Term Infection by the Oral Opportunistic Pathogen, Porphyromonas gingivalis.

Lee J, Roberts JS, Atanasova KR, Chowdhury N, Han K, Yilmaz Ö.

Front Cell Infect Microbiol. 2017 Dec 1;7:493. doi: 10.3389/fcimb.2017.00493. eCollection 2017.

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