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Items: 1 to 20 of 166

1.

Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.

Roberts JS, Atanasova KR, Lee J, Diamond G, Deguzman J, Hee Choi C, Yilmaz Ö.

Front Cell Infect Microbiol. 2017 Jul 4;7:291. doi: 10.3389/fcimb.2017.00291. eCollection 2017.

2.

Porphyromonas gingivalis-nucleoside-diphosphate-kinase inhibits ATP-induced reactive-oxygen-species via P2X7 receptor/NADPH-oxidase signalling and contributes to persistence.

Choi CH, Spooner R, DeGuzman J, Koutouzis T, Ojcius DM, Yilmaz Ö.

Cell Microbiol. 2013 Jun;15(6):961-76. doi: 10.1111/cmi.12089. Epub 2013 Jan 9.

3.

A novel kinase function of a nucleoside-diphosphate-kinase homologue in Porphyromonas gingivalis is critical in subversion of host cell apoptosis by targeting heat-shock protein 27.

Lee J, Roberts JS, Atanasova KR, Chowdhury N, Yilmaz Ö.

Cell Microbiol. 2018 May;20(5):e12825. doi: 10.1111/cmi.12825. Epub 2018 Feb 7.

PMID:
29359393
4.

Nucleoside-Diphosphate-Kinase of P. gingivalis is Secreted from Epithelial Cells In the Absence of a Leader Sequence Through a Pannexin-1 Interactome.

Atanasova K, Lee J, Roberts J, Lee K, Ojcius DM, Yilmaz Ö.

Sci Rep. 2016 Nov 24;6:37643. doi: 10.1038/srep37643.

5.

NOX1/2 activation in human gingival fibroblasts by Fusobacterium nucleatum facilitates attachment of Porphyromonas gingivalis.

Ahn SH, Song JE, Kim S, Cho SH, Lim YK, Kook JK, Kook MS, Lee TH.

Arch Microbiol. 2016 Aug;198(6):573-83. doi: 10.1007/s00203-016-1223-7. Epub 2016 Apr 12.

PMID:
27071620
6.

Porphyromonas gingivalis traffics into endoplasmic reticulum-rich-autophagosomes for successful survival in human gingival epithelial cells.

Lee K, Roberts JS, Choi CH, Atanasova KR, Yilmaz Ö.

Virulence. 2018 Dec 31;9(1):845-859. doi: 10.1080/21505594.2018.1454171.

7.

Porphyromonas gingivalis infection sequesters pro-apoptotic Bad through Akt in primary gingival epithelial cells.

Yao L, Jermanus C, Barbetta B, Choi C, Verbeke P, Ojcius DM, Yilmaz O.

Mol Oral Microbiol. 2010 Apr;25(2):89-101. doi: 10.1111/j.2041-1014.2010.00569.x.

8.

Porphyromonas gingivalis attenuates ATP-mediated inflammasome activation and HMGB1 release through expression of a nucleoside-diphosphate kinase.

Johnson L, Atanasova KR, Bui PQ, Lee J, Hung SC, Yilmaz Ö, Ojcius DM.

Microbes Infect. 2015 May;17(5):369-77. doi: 10.1016/j.micinf.2015.03.010. Epub 2015 Mar 27.

9.

ATP-dependent activation of an inflammasome in primary gingival epithelial cells infected by Porphyromonas gingivalis.

Yilmaz O, Sater AA, Yao L, Koutouzis T, Pettengill M, Ojcius DM.

Cell Microbiol. 2010 Feb;12(2):188-98. doi: 10.1111/j.1462-5822.2009.01390.x. Epub 2009 Oct 6.

10.

The role of reactive-oxygen-species in microbial persistence and inflammation.

Spooner R, Yilmaz O.

Int J Mol Sci. 2011 Jan 13;12(1):334-52. doi: 10.3390/ijms12010334. Review.

11.

Porphyromonas gingivalis fimbriae dampen P2X7-dependent interleukin-1β secretion.

Morandini AC, Ramos-Junior ES, Potempa J, Nguyen KA, Oliveira AC, Bellio M, Ojcius DM, Scharfstein J, Coutinho-Silva R.

J Innate Immun. 2014;6(6):831-45. doi: 10.1159/000363338. Epub 2014 Jun 6.

13.

Porphyromonas gingivalis SerB-mediated dephosphorylation of host cell cofilin modulates invasion efficiency.

Moffatt CE, Inaba H, Hirano T, Lamont RJ.

Cell Microbiol. 2012 Apr;14(4):577-88. doi: 10.1111/j.1462-5822.2011.01743.x. Epub 2012 Feb 2.

14.

Human Primary Epithelial Cells Acquire an Epithelial-Mesenchymal-Transition Phenotype during Long-Term Infection by the Oral Opportunistic Pathogen, Porphyromonas gingivalis.

Lee J, Roberts JS, Atanasova KR, Chowdhury N, Han K, Yilmaz Ö.

Front Cell Infect Microbiol. 2017 Dec 1;7:493. doi: 10.3389/fcimb.2017.00493. eCollection 2017.

16.

Intercellular spreading of Porphyromonas gingivalis infection in primary gingival epithelial cells.

Yilmaz O, Verbeke P, Lamont RJ, Ojcius DM.

Infect Immun. 2006 Jan;74(1):703-10.

17.

Porphyromonas gingivalis-induced IL-33 down-regulates hCAP-18/LL-37 production in human gingival epithelial cells.

Tada H, Shimizu T, Matsushita K, Takada H.

Biomed Res. 2017;38(3):167-173. doi: 10.2220/biomedres.38.167.

18.

Porphyromonas gingivalis induction of microRNA-203 expression controls suppressor of cytokine signaling 3 in gingival epithelial cells.

Moffatt CE, Lamont RJ.

Infect Immun. 2011 Jul;79(7):2632-7. doi: 10.1128/IAI.00082-11. Epub 2011 May 2.

19.

Noncanonical activation of β-catenin by Porphyromonas gingivalis.

Zhou Y, Sztukowska M, Wang Q, Inaba H, Potempa J, Scott DA, Wang H, Lamont RJ.

Infect Immun. 2015 Aug;83(8):3195-203. doi: 10.1128/IAI.00302-15. Epub 2015 Jun 1.

20.

A Dual Role for P2X7 Receptor during Porphyromonas gingivalis Infection.

Ramos-Junior ES, Morandini AC, Almeida-da-Silva CL, Franco EJ, Potempa J, Nguyen KA, Oliveira AC, Zamboni DS, Ojcius DM, Scharfstein J, Coutinho-Silva R.

J Dent Res. 2015 Sep;94(9):1233-42. doi: 10.1177/0022034515593465. Epub 2015 Jul 7.

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