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Items: 1 to 20 of 103

1.

C/EBPβ Promotes Immunity to Oral Candidiasis through Regulation of β-Defensins.

Simpson-Abelson MR, Childs EE, Ferreira MC, Bishu S, Conti HR, Gaffen SL.

PLoS One. 2015 Aug 28;10(8):e0136538. doi: 10.1371/journal.pone.0136538. eCollection 2015.

2.

Interleukin-17-induced protein lipocalin 2 is dispensable for immunity to oral candidiasis.

Ferreira MC, Whibley N, Mamo AJ, Siebenlist U, Chan YR, Gaffen SL.

Infect Immun. 2014 Mar;82(3):1030-5. doi: 10.1128/IAI.01389-13. Epub 2013 Dec 16.

3.

Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis.

Conti HR, Shen F, Nayyar N, Stocum E, Sun JN, Lindemann MJ, Ho AW, Hai JH, Yu JJ, Jung JW, Filler SG, Masso-Welch P, Edgerton M, Gaffen SL.

J Exp Med. 2009 Feb 16;206(2):299-311. doi: 10.1084/jem.20081463. Epub 2009 Feb 9.

4.

Role of neutrophils in IL-17-dependent immunity to mucosal candidiasis.

Huppler AR, Conti HR, Hernández-Santos N, Darville T, Biswas PS, Gaffen SL.

J Immunol. 2014 Feb 15;192(4):1745-52. doi: 10.4049/jimmunol.1302265. Epub 2014 Jan 17. Erratum in: J Immunol. 2015 Feb 1;194(3):1382.

5.

Defective IL-17- and IL-22-dependent mucosal host response to Candida albicans determines susceptibility to oral candidiasis in mice expressing the HIV-1 transgene.

Goupil M, Cousineau-Côté V, Aumont F, Sénéchal S, Gaboury L, Hanna Z, Jolicoeur P, de Repentigny L.

BMC Immunol. 2014 Oct 26;15:49. doi: 10.1186/s12865-014-0049-9.

6.

β-Defensin 1 plays a role in acute mucosal defense against Candida albicans.

Tomalka J, Azodi E, Narra HP, Patel K, O'Neill S, Cardwell C, Hall BA, Wilson JM, Hise AG.

J Immunol. 2015 Feb 15;194(4):1788-95. doi: 10.4049/jimmunol.1203239. Epub 2015 Jan 16.

7.

Antibody blockade of IL-17 family cytokines in immunity to acute murine oral mucosal candidiasis.

Whibley N, Tritto E, Traggiai E, Kolbinger F, Moulin P, Brees D, Coleman BM, Mamo AJ, Garg AV, Jaycox JR, Siebenlist U, Kammüller M, Gaffen SL.

J Leukoc Biol. 2016 Jun;99(6):1153-64. doi: 10.1189/jlb.4A0915-428R. Epub 2016 Jan 4. Erratum in: J Leukoc Biol. 2017 Apr;101(4):1065.

8.

New mechanism of oral immunity to mucosal candidiasis in hyper-IgE syndrome.

Conti HR, Baker O, Freeman AF, Jang WS, Holland SM, Li RA, Edgerton M, Gaffen SL.

Mucosal Immunol. 2011 Jul;4(4):448-55. doi: 10.1038/mi.2011.5. Epub 2011 Feb 23.

9.

The adaptor CARD9 is required for adaptive but not innate immunity to oral mucosal Candida albicans infections.

Bishu S, Hernández-Santos N, Simpson-Abelson MR, Huppler AR, Conti HR, Ghilardi N, Mamo AJ, Gaffen SL.

Infect Immun. 2014 Mar;82(3):1173-80. doi: 10.1128/IAI.01335-13. Epub 2013 Dec 30.

10.

CCAAT/Enhancer-binding protein β promotes pathogenesis of EAE.

Simpson-Abelson MR, Hernandez-Mir G, Childs EE, Cruz JA, Poholek AC, Chattopadhyay A, Gaffen SL, McGeachy MJ.

Cytokine. 2017 Apr;92:24-32. doi: 10.1016/j.cyto.2017.01.005. Epub 2017 Jan 12.

11.

IL-17 Receptor Signaling in Oral Epithelial Cells Is Critical for Protection against Oropharyngeal Candidiasis.

Conti HR, Bruno VM, Childs EE, Daugherty S, Hunter JP, Mengesha BG, Saevig DL, Hendricks MR, Coleman BM, Brane L, Solis N, Cruz JA, Verma AH, Garg AV, Hise AG, Richardson JP, Naglik JR, Filler SG, Kolls JK, Sinha S, Gaffen SL.

Cell Host Microbe. 2016 Nov 9;20(5):606-617. doi: 10.1016/j.chom.2016.10.001. Epub 2016 Oct 27.

12.
13.

Signaling through IL-17C/IL-17RE is dispensable for immunity to systemic, oral and cutaneous candidiasis.

Conti HR, Whibley N, Coleman BM, Garg AV, Jaycox JR, Gaffen SL.

PLoS One. 2015 Apr 7;10(4):e0122807. doi: 10.1371/journal.pone.0122807. eCollection 2015.

14.

Oral-resident natural Th17 cells and γδ T cells control opportunistic Candida albicans infections.

Conti HR, Peterson AC, Brane L, Huppler AR, Hernández-Santos N, Whibley N, Garg AV, Simpson-Abelson MR, Gibson GA, Mamo AJ, Osborne LC, Bishu S, Ghilardi N, Siebenlist U, Watkins SC, Artis D, McGeachy MJ, Gaffen SL.

J Exp Med. 2014 Sep 22;211(10):2075-84. doi: 10.1084/jem.20130877. Epub 2014 Sep 8.

15.

Oral epithelial cells orchestrate innate type 17 responses to Candida albicans through the virulence factor candidalysin.

Verma AH, Richardson JP, Zhou C, Coleman BM, Moyes DL, Ho J, Huppler AR, Ramani K, McGeachy MJ, Mufazalov IA, Waisman A, Kane LP, Biswas PS, Hube B, Naglik JR, Gaffen SL.

Sci Immunol. 2017 Nov 3;2(17). pii: eaam8834. doi: 10.1126/sciimmunol.aam8834.

16.

Expression of C/EBPβ in myeloid progenitors during sepsis promotes immunosuppression.

Dai J, Kumbhare A, Youssef D, Yao ZQ, McCall CE, El Gazzar M.

Mol Immunol. 2017 Nov;91:165-172. doi: 10.1016/j.molimm.2017.09.008. Epub 2017 Sep 19.

17.

Early activation of the interleukin-23-17 axis in a murine model of oropharyngeal candidiasis.

Saunus JM, Wagner SA, Matias MA, Hu Y, Zaini ZM, Farah CS.

Mol Oral Microbiol. 2010 Oct;25(5):343-56. doi: 10.1111/j.2041-1014.2010.00570.x.

PMID:
20883223
18.
19.

Cutting edge: IL-17-secreting innate lymphoid cells are essential for host defense against fungal infection.

Gladiator A, Wangler N, Trautwein-Weidner K, LeibundGut-Landmann S.

J Immunol. 2013 Jan 15;190(2):521-5. doi: 10.4049/jimmunol.1202924. Epub 2012 Dec 19.

20.

Th17 cells confer long-term adaptive immunity to oral mucosal Candida albicans infections.

Hernández-Santos N, Huppler AR, Peterson AC, Khader SA, McKenna KC, Gaffen SL.

Mucosal Immunol. 2013 Sep;6(5):900-10. doi: 10.1038/mi.2012.128. Epub 2012 Dec 19.

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