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Items: 1 to 20 of 187

1.

Porphyromonas gingivalis fimbriae dampen P2X7-dependent interleukin-1β secretion.

Morandini AC, Ramos-Junior ES, Potempa J, Nguyen KA, Oliveira AC, Bellio M, Ojcius DM, Scharfstein J, Coutinho-Silva R.

J Innate Immun. 2014;6(6):831-45. doi: 10.1159/000363338. Epub 2014 Jun 6.

2.

A Dual Role for P2X7 Receptor during Porphyromonas gingivalis Infection.

Ramos-Junior ES, Morandini AC, Almeida-da-Silva CL, Franco EJ, Potempa J, Nguyen KA, Oliveira AC, Zamboni DS, Ojcius DM, Scharfstein J, Coutinho-Silva R.

J Dent Res. 2015 Sep;94(9):1233-42. doi: 10.1177/0022034515593465. Epub 2015 Jul 7.

3.

Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.

Park E, Na HS, Song YR, Shin SY, Kim YM, Chung J.

Infect Immun. 2014 Jan;82(1):112-23. doi: 10.1128/IAI.00862-13. Epub 2013 Oct 14.

4.

Increases in IL-33 production by fimbriae and lipopeptide from Porphyromonas gingivalis in mouse bone marrow-derived dendritic cells via Toll-like receptor 2.

Tada H, Suzuki R, Nemoto E, Shimauchi H, Matsushita K, Takada H.

Biomed Res. 2017;38(3):189-195. doi: 10.2220/biomedres.38.189.

5.

P2X7 receptor-mediated leukocyte recruitment and Porphyromonas gingivalis clearance requires IL-1β production and autocrine IL-1 receptor activation.

Almeida-da-Silva CLC, Ramos-Junior ES, Morandini AC, Rocha GDC, Marinho Y, Tamura AS, de Andrade KQ, Bellio M, Savio LEB, Scharfstein J, Ojcius DM, Coutinho-Silva R.

Immunobiology. 2019 Jan;224(1):50-59. doi: 10.1016/j.imbio.2018.10.008. Epub 2018 Nov 2.

PMID:
30429052
6.

The ATP-P2X7 Signaling Axis Is an Essential Sentinel for Intracellular Clostridium difficile Pathogen-Induced Inflammasome Activation.

Liu YH, Chang YC, Chen LK, Su PA, Ko WC, Tsai YS, Chen YH, Lai HC, Wu CY, Hung YP, Tsai PJ.

Front Cell Infect Microbiol. 2018 Mar 16;8:84. doi: 10.3389/fcimb.2018.00084. eCollection 2018.

7.

Mechanism and implications of CXCR4-mediated integrin activation by Porphyromonas gingivalis.

Hajishengallis G, McIntosh ML, Nishiyama SI, Yoshimura F.

Mol Oral Microbiol. 2013 Aug;28(4):239-49. doi: 10.1111/omi.12021. Epub 2013 Jan 21.

8.

Macrophage-specific TLR2 signaling mediates pathogen-induced TNF-dependent inflammatory oral bone loss.

Papadopoulos G, Weinberg EO, Massari P, Gibson FC 3rd, Wetzler LM, Morgan EF, Genco CA.

J Immunol. 2013 Feb 1;190(3):1148-57. doi: 10.4049/jimmunol.1202511. Epub 2012 Dec 21.

9.

Porphyromonas gingivalis-nucleoside-diphosphate-kinase inhibits ATP-induced reactive-oxygen-species via P2X7 receptor/NADPH-oxidase signalling and contributes to persistence.

Choi CH, Spooner R, DeGuzman J, Koutouzis T, Ojcius DM, Yilmaz Ö.

Cell Microbiol. 2013 Jun;15(6):961-76. doi: 10.1111/cmi.12089. Epub 2013 Jan 9.

10.
11.

Fimbria-dependent activation of pro-inflammatory molecules in Porphyromonas gingivalis infected human aortic endothelial cells.

Takahashi Y, Davey M, Yumoto H, Gibson FC 3rd, Genco CA.

Cell Microbiol. 2006 May;8(5):738-57.

PMID:
16611224
13.

Liver X receptors contribute to periodontal pathogen-elicited inflammation and oral bone loss.

Huang N, Shaik-Dasthagirisaheb YB, LaValley MP, Gibson FC 3rd.

Mol Oral Microbiol. 2015 Dec;30(6):438-50. doi: 10.1111/omi.12103. Epub 2015 Jun 9.

14.

TLR agonists stimulate Nlrp3-dependent IL-1β production independently of the purinergic P2X7 receptor in dendritic cells and in vivo.

He Y, Franchi L, Núñez G.

J Immunol. 2013 Jan 1;190(1):334-9. doi: 10.4049/jimmunol.1202737. Epub 2012 Dec 7.

15.

Porphyromonas gingivalis Stimulates TLR2-PI3K Signaling to Escape Immune Clearance and Induce Bone Resorption Independently of MyD88.

Makkawi H, Hoch S, Burns E, Hosur K, Hajishengallis G, Kirschning CJ, Nussbaum G.

Front Cell Infect Microbiol. 2017 Aug 8;7:359. doi: 10.3389/fcimb.2017.00359. eCollection 2017.

17.

Fimbrial proteins of porphyromonas gingivalis mediate in vivo virulence and exploit TLR2 and complement receptor 3 to persist in macrophages.

Wang M, Shakhatreh MA, James D, Liang S, Nishiyama S, Yoshimura F, Demuth DR, Hajishengallis G.

J Immunol. 2007 Aug 15;179(4):2349-58.

18.

TLR2-dependent modulation of osteoclastogenesis by Porphyromonas gingivalis through differential induction of NFATc1 and NF-kappaB.

Zhang P, Liu J, Xu Q, Harber G, Feng X, Michalek SM, Katz J.

J Biol Chem. 2011 Jul 8;286(27):24159-69. doi: 10.1074/jbc.M110.198085. Epub 2011 May 12.

19.

Aging and contribution of MyD88 and TRIF to expression of TLR pathway-associated genes following stimulation with Porphyromonas gingivalis.

Shaik-Dasthagirisaheb YB, Huang N, Weinberg EO, Shen SS, Genco CA, Gibson FC 3rd.

J Periodontal Res. 2015 Feb;50(1):89-102. doi: 10.1111/jre.12185. Epub 2014 May 24.

20.

Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.

Roberts JS, Atanasova KR, Lee J, Diamond G, Deguzman J, Hee Choi C, Yilmaz Ö.

Front Cell Infect Microbiol. 2017 Jul 4;7:291. doi: 10.3389/fcimb.2017.00291. eCollection 2017.

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