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Items: 1 to 20 of 77

1.

Association of the LILRA3 deletion with B-NHL and functional characterization of the immunostimulatory molecule.

Low HZ, Reuter S, Topperwien M, Dankenbrink N, Peest D, Kabalak G, Stripecke R, Schmidt RE, Matthias T, Witte T.

PLoS One. 2013 Dec 9;8(12):e81360. doi: 10.1371/journal.pone.0081360. eCollection 2013.

2.

Distribution of LILRA3 (ILT6/LIR4) deletion in psoriatic patients and healthy controls.

Wiśniewski A, Łuszczek W, Mańczak M, Jasek M, Kubicka W, Cislo M, Kuśnierczyk P.

Hum Immunol. 2003 Apr;64(4):458-61.

PMID:
12651072
3.

TLR8 regulation of LILRA3 in monocytes is abrogated in human immunodeficiency virus infection and correlates to CD4 counts and virus loads.

Low HZ, Ahrenstorf G, Pommerenke C, Habermann N, Schughart K, Ordóñez D, Stripecke R, Wilk E, Witte T.

Retrovirology. 2016 Mar 12;13:15. doi: 10.1186/s12977-016-0248-y.

4.
5.

Analysis of candidate genes on chromosome 19 in coeliac disease: an association study of the KIR and LILR gene clusters.

Moodie SJ, Norman PJ, King AL, Fraser JS, Curtis D, Ellis HJ, Vaughan RW, Ciclitira PJ.

Eur J Immunogenet. 2002 Aug;29(4):287-91.

PMID:
12121272
6.

Soluble LILRA3, a potential natural antiinflammatory protein, is increased in patients with rheumatoid arthritis and is tightly regulated by interleukin 10, tumor necrosis factor-alpha, and interferon-gamma.

An H, Chandra V, Piraino B, Borges L, Geczy C, McNeil HP, Bryant K, Tedla N.

J Rheumatol. 2010 Aug 1;37(8):1596-606. doi: 10.3899/jrheum.091119. Epub 2010 Jul 1.

PMID:
20595277
7.

6.7-kbp deletion in LILRA3 (ILT6) gene is associated with later onset of the multiple sclerosis in a Polish population.

Wiśniewski A, Wagner M, Nowak I, Bilińska M, Pokryszko-Dragan A, Jasek M, Kuśnierczyk P.

Hum Immunol. 2013 Mar;74(3):353-7. doi: 10.1016/j.humimm.2012.12.006. Epub 2012 Dec 10.

PMID:
23238213
8.

Glycosylation in a mammalian expression system is critical for the production of functionally active leukocyte immunoglobulin-like receptor A3 protein.

Lee TH, Mitchell A, Liu Lau S, An H, Rajeaskariah P, Wasinger V, Raftery M, Bryant K, Tedla N.

J Biol Chem. 2013 Nov 15;288(46):32873-85. doi: 10.1074/jbc.M113.478578. Epub 2013 Sep 30.

9.

Multiple sclerosis associates with LILRA3 deletion in Spanish patients.

Ordóñez D, Sánchez AJ, Martínez-Rodríguez JE, Cisneros E, Ramil E, Romo N, Moraru M, Munteis E, López-Botet M, Roquer J, García-Merino A, Vilches C.

Genes Immun. 2009 Sep;10(6):579-85. doi: 10.1038/gene.2009.34. Epub 2009 May 7.

PMID:
19421224
11.

LILRA3 deletion is a genetic risk factor of HIV infection.

Ahrenstorf G, Low HZ, Kniesch K, Ordonez D, Meyer-Olson D, Ahmad F, Kücherer C, Gunsenheimer-Bartmeyer B, Stoll M, Matthias T, Schmidt RE, Witte T.

AIDS. 2017 Jan 2;31(1):25-34.

PMID:
27755104
12.

Monocyte-derived dendritic cells generated after a short-term culture with IFN-alpha and granulocyte-macrophage colony-stimulating factor stimulate a potent Epstein-Barr virus-specific CD8+ T cell response.

Santodonato L, D'Agostino G, Nisini R, Mariotti S, Monque DM, Spada M, Lattanzi L, Perrone MP, Andreotti M, Belardelli F, Ferrantini M.

J Immunol. 2003 May 15;170(10):5195-202.

13.

CD28, IL-2-independent costimulatory pathways for CD8 T lymphocyte activation.

Sepulveda H, Cerwenka A, Morgan T, Dutton RW.

J Immunol. 1999 Aug 1;163(3):1133-42.

14.

CpG-A oligonucleotides induce a monocyte-derived dendritic cell-like phenotype that preferentially activates CD8 T cells.

Krug A, Rothenfusser S, Selinger S, Bock C, Kerkmann M, Battiany J, Sarris A, Giese T, Speiser D, Endres S, Hartmann G.

J Immunol. 2003 Apr 1;170(7):3468-77.

15.

Fluorescent target array T helper assay: a multiplex flow cytometry assay to measure antigen-specific CD4+ T cell-mediated B cell help in vivo.

Quah BJ, Wijesundara DK, Ranasinghe C, Parish CR.

J Immunol Methods. 2013 Jan 31;387(1-2):181-90. doi: 10.1016/j.jim.2012.10.013. Epub 2012 Oct 30.

PMID:
23123200
16.

Association of immunoglobulin-like transcript 6 deficiency with Sjögren's syndrome.

Kabalak G, Dobberstein SB, Matthias T, Reuter S, The YH, Dörner T, Schmidt RE, Witte T.

Arthritis Rheum. 2009 Oct;60(10):2923-5. doi: 10.1002/art.24804.

17.

Human resting B lymphocytes can serve as accessory cells for anti-CD2-induced T cell activation.

Hirokawa M, Gray JD, Takahashi T, Horwitz DA.

J Immunol. 1992 Sep 15;149(6):1859-66.

PMID:
1355500
18.

CD4+ and CD8+ mediated cellular immune response to recombinant influenza nucleoprotein.

Gschoesser C, Almanzar G, Hainz U, Ortin J, Schonitzer D, Schild H, Saurwein-Teissl M, Grubeck-Loebenstein B.

Vaccine. 2002 Nov 1;20(31-32):3731-8.

PMID:
12399202
19.

NK cells regulate CD8+ T cell effector function in response to an intracellular pathogen.

Vankayalapati R, Klucar P, Wizel B, Weis SE, Samten B, Safi H, Shams H, Barnes PF.

J Immunol. 2004 Jan 1;172(1):130-7.

20.

A soluble form of lymphocyte activation gene-3 (IMP321) induces activation of a large range of human effector cytotoxic cells.

Brignone C, Grygar C, Marcu M, Schäkel K, Triebel F.

J Immunol. 2007 Sep 15;179(6):4202-11.

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