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Porphyromonas gingivalis regulates TREM-1 in human polymorphonuclear neutrophils via its gingipains.

Bostanci N, Thurnheer T, Aduse-Opoku J, Curtis MA, Zinkernagel AS, Belibasakis GN.

PLoS One. 2013 Oct 4;8(10):e75784. doi: 10.1371/journal.pone.0075784. eCollection 2013.


Role of Porphyromonas gingivalis gingipains in multi-species biofilm formation.

Bao K, Belibasakis GN, Thurnheer T, Aduse-Opoku J, Curtis MA, Bostanci N.

BMC Microbiol. 2014 Oct 2;14:258. doi: 10.1186/s12866-014-0258-7.


Involvement of the TREM-1/DAP12 pathway in the innate immune responses to Porphyromonas gingivalis.

Bostanci N, Thurnheer T, Belibasakis GN.

Mol Immunol. 2011 Oct;49(1-2):387-94. doi: 10.1016/j.molimm.2011.09.012. Epub 2011 Oct 2.


Inactive Gingipains from P. gingivalis Selectively Skews T Cells toward a Th17 Phenotype in an IL-6 Dependent Manner.

Glowczyk I, Wong A, Potempa B, Babyak O, Lech M, Lamont RJ, Potempa J, Koziel J.

Front Cell Infect Microbiol. 2017 Apr 27;7:140. doi: 10.3389/fcimb.2017.00140. eCollection 2017.


Porphyromonas gingivalis gingipains cause defective macrophage migration towards apoptotic cells and inhibit phagocytosis of primary apoptotic neutrophils.

Castro SA, Collighan R, Lambert PA, Dias IH, Chauhan P, Bland CE, Milic I, Milward MR, Cooper PR, Devitt A.

Cell Death Dis. 2017 Mar 2;8(3):e2644. doi: 10.1038/cddis.2016.481.


Cleavage of the human C5A receptor by proteinases derived from Porphyromonas gingivalis: cleavage of leukocyte C5a receptor.

Jagels MA, Ember JA, Travis J, Potempa J, Pike R, Hugli TE.

Adv Exp Med Biol. 1996;389:155-64.


The role of phagocytosis, oxidative burst and neutrophil extracellular traps in the interaction between neutrophils and the periodontal pathogen Porphyromonas gingivalis.

Jayaprakash K, Demirel I, Khalaf H, Bengtsson T.

Mol Oral Microbiol. 2015 Oct;30(5):361-75. doi: 10.1111/omi.12099. Epub 2015 May 4.


Doxycycline inhibits TREM-1 induction by Porphyromonas gingivalis.

Bostanci N, Belibasakis GN.

FEMS Immunol Med Microbiol. 2012 Oct;66(1):37-44. doi: 10.1111/j.1574-695X.2012.00982.x. Epub 2012 May 21.


Expression and regulation of triggering receptor expressed on myeloid cells 1 in periodontal diseases.

Willi M, Belibasakis GN, Bostanci N.

Clin Exp Immunol. 2014 Oct;178(1):190-200. doi: 10.1111/cei.12397.


Suppression of inflammatory responses of human gingival fibroblasts by gingipains from Porphyromonas gingivalis.

Palm E, Khalaf H, Bengtsson T.

Mol Oral Microbiol. 2015 Feb;30(1):74-85. doi: 10.1111/omi.12073. Epub 2014 Sep 19.


Gingipains inactivate a cell surface ligand on Porphyromonas gingivalis that induces TLR2-and TLR4-independent signaling.

Kishimoto M, Yoshimura A, Naito M, Okamoto K, Yamamoto K, Golenbock DT, Hara Y, Nakayama K.

Microbiol Immunol. 2006;50(4):315-25.


Identification of a novel Porphyromonas gingivalis outer membrane protein, PG534, required for the production of active gingipains.

Saiki K, Konishi K.

FEMS Microbiol Lett. 2010 Sep 1;310(2):168-74. doi: 10.1111/j.1574-6968.2010.02059.x. Epub 2010 Jul 12.


Contradictory roles of Porphyromonas gingivalis gingipains in caspase-1 activation.

Jung YJ, Jun HK, Choi BK.

Cell Microbiol. 2015 Sep;17(9):1304-19. doi: 10.1111/cmi.12435. Epub 2015 Apr 16.


Triggering receptor expressed on myeloid cells in the pathogenesis of periodontitis: potential novel treatment strategies.

Rudick CP, Miyamoto T, Lang MS, Agrawal DK.

Expert Rev Clin Immunol. 2017 Dec;13(12):1189-1197. doi: 10.1080/1744666X.2017.1392855. Epub 2017 Oct 20. Review.


Cleavage of protease-activated receptors on an immortalized oral epithelial cell line by Porphyromonas gingivalis gingipains.

Giacaman RA, Asrani AC, Ross KF, Herzberg MC.

Microbiology. 2009 Oct;155(Pt 10):3238-46. doi: 10.1099/mic.0.029132-0. Epub 2009 Jul 16.


Molecular genetics of Porphyromonas gingivalis: gingipains and other virulence factors.

Nakayama K.

Curr Protein Pept Sci. 2003 Dec;4(6):389-95. Review.


Gingipains from Porphyromonas gingivalis synergistically induce the production of proinflammatory cytokines through protease-activated receptors with Toll-like receptor and NOD1/2 ligands in human monocytic cells.

Uehara A, Imamura T, Potempa J, Travis J, Takada H.

Cell Microbiol. 2008 May;10(5):1181-9. doi: 10.1111/j.1462-5822.2008.01119.x. Epub 2008 Jan 7.


Attenuation of the phosphatidylinositol 3-kinase/Akt signaling pathway by Porphyromonas gingivalis gingipains RgpA, RgpB, and Kgp.

Nakayama M, Inoue T, Naito M, Nakayama K, Ohara N.

J Biol Chem. 2015 Feb 20;290(8):5190-202. doi: 10.1074/jbc.M114.591610. Epub 2015 Jan 6.


Gingipains: Critical Factors in the Development of Aspiration Pneumonia Caused by Porphyromonas gingivalis.

Benedyk M, Mydel PM, Delaleu N, PÅ‚aza K, Gawron K, Milewska A, Maresz K, Koziel J, Pyrc K, Potempa J.

J Innate Immun. 2016;8(2):185-98. doi: 10.1159/000441724. Epub 2015 Nov 28.


The host cytokine response to Porphyromonas gingivalis is modified by gingipains.

Stathopoulou PG, Benakanakere MR, Galicia JC, Kinane DF.

Oral Microbiol Immunol. 2009 Feb;24(1):11-7. doi: 10.1111/j.1399-302X.2008.00467.x.

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