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Items: 1 to 20 of 163

1.

Regulatory interplay of Cockayne syndrome B ATPase and stress-response gene ATF3 following genotoxic stress.

Kristensen U, Epanchintsev A, Rauschendorf MA, Laugel V, Stevnsner T, Bohr VA, Coin F, Egly JM.

Proc Natl Acad Sci U S A. 2013 Jun 18;110(25):E2261-70. doi: 10.1073/pnas.1220071110. Epub 2013 Jun 3.

2.

Cockayne's Syndrome A and B Proteins Regulate Transcription Arrest after Genotoxic Stress by Promoting ATF3 Degradation.

Epanchintsev A, Costanzo F, Rauschendorf MA, Caputo M, Ye T, Donnio LM, Proietti-de-Santis L, Coin F, Laugel V, Egly JM.

Mol Cell. 2017 Dec 21;68(6):1054-1066.e6. doi: 10.1016/j.molcel.2017.11.009. Epub 2017 Dec 7.

3.

Cockayne syndrome B protein regulates the transcriptional program after UV irradiation.

Proietti-De-Santis L, Drané P, Egly JM.

EMBO J. 2006 May 3;25(9):1915-23. Epub 2006 Apr 6.

4.

Differential requirement for the ATPase domain of the Cockayne syndrome group B gene in the processing of UV-induced DNA damage and 8-oxoguanine lesions in human cells.

Selzer RR, Nyaga S, Tuo J, May A, Muftuoglu M, Christiansen M, Citterio E, Brosh RM Jr, Bohr VA.

Nucleic Acids Res. 2002 Feb 1;30(3):782-93.

5.

The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repair.

Brosh RM Jr, Balajee AS, Selzer RR, Sunesen M, Proietti De Santis L, Bohr VA.

Mol Biol Cell. 1999 Nov;10(11):3583-94.

6.

The conserved Cockayne syndrome B-piggyBac fusion protein (CSB-PGBD3) affects DNA repair and induces both interferon-like and innate antiviral responses in CSB-null cells.

Bailey AD, Gray LT, Pavelitz T, Newman JC, Horibata K, Tanaka K, Weiner AM.

DNA Repair (Amst). 2012 May 1;11(5):488-501. doi: 10.1016/j.dnarep.2012.02.004. Epub 2012 Apr 6.

7.

The C-terminal Region and SUMOylation of Cockayne Syndrome Group B Protein Play Critical Roles in Transcription-coupled Nucleotide Excision Repair.

Sin Y, Tanaka K, Saijo M.

J Biol Chem. 2016 Jan 15;291(3):1387-97. doi: 10.1074/jbc.M115.683235. Epub 2015 Nov 30.

8.
9.

A ubiquitin-binding domain in Cockayne syndrome B required for transcription-coupled nucleotide excision repair.

Anindya R, Mari PO, Kristensen U, Kool H, Giglia-Mari G, Mullenders LH, Fousteri M, Vermeulen W, Egly JM, Svejstrup JQ.

Mol Cell. 2010 Jun 11;38(5):637-48. doi: 10.1016/j.molcel.2010.04.017.

10.

Truncated Cockayne syndrome B protein represses elongation by RNA polymerase I.

Lebedev A, Scharffetter-Kochanek K, Iben S.

J Mol Biol. 2008 Oct 3;382(2):266-74. doi: 10.1016/j.jmb.2008.07.018. Epub 2008 Jul 16.

PMID:
18656484
11.

Role of the ATPase domain of the Cockayne syndrome group B protein in UV induced apoptosis.

Balajee AS, Proietti De Santis L, Brosh RM Jr, Selzer R, Bohr VA.

Oncogene. 2000 Jan 27;19(4):477-89.

12.

The transcriptional response after oxidative stress is defective in Cockayne syndrome group B cells.

Kyng KJ, May A, Brosh RM Jr, Cheng WH, Chen C, Becker KG, Bohr VA.

Oncogene. 2003 Feb 27;22(8):1135-49.

PMID:
12606941
13.

The Cockayne syndrome B protein, involved in transcription-coupled DNA repair, resides in an RNA polymerase II-containing complex.

van Gool AJ, Citterio E, Rademakers S, van Os R, Vermeulen W, Constantinou A, Egly JM, Bootsma D, Hoeijmakers JH.

EMBO J. 1997 Oct 1;16(19):5955-65.

14.

RNA interference against transcription elongation factor SII does not support its role in transcription-coupled nucleotide excision repair.

Mackinnon-Roy C, Stubbert LJ, McKay BC.

Mutat Res. 2011 Jan 10;706(1-2):53-8. doi: 10.1016/j.mrfmmm.2010.10.010. Epub 2010 Nov 9.

PMID:
21070792
15.

Cockayne syndrome exhibits dysregulation of p21 and other gene products that may be independent of transcription-coupled repair.

Cleaver JE, Hefner E, Laposa RR, Karentz D, Marti T.

Neuroscience. 2007 Apr 14;145(4):1300-8. Epub 2006 Oct 19.

16.

Deletion of 5' sequences of the CSB gene provides insight into the pathophysiology of Cockayne syndrome.

Laugel V, Dalloz C, Stary A, Cormier-Daire V, Desguerre I, Renouil M, Fourmaintraux A, Velez-Cruz R, Egly JM, Sarasin A, Dollfus H.

Eur J Hum Genet. 2008 Mar;16(3):320-7. doi: 10.1038/sj.ejhg.5201991. Epub 2008 Jan 9.

17.

Early host cell reactivation of an oxidatively damaged adenovirus-encoded reporter gene requires the Cockayne syndrome proteins CSA and CSB.

Leach DM, Rainbow AJ.

Mutagenesis. 2011 Mar;26(2):315-21. doi: 10.1093/mutage/geq096. Epub 2010 Nov 8.

18.

Dysregulation of gene expression as a cause of Cockayne syndrome neurological disease.

Wang Y, Chakravarty P, Ranes M, Kelly G, Brooks PJ, Neilan E, Stewart A, Schiavo G, Svejstrup JQ.

Proc Natl Acad Sci U S A. 2014 Oct 7;111(40):14454-9. doi: 10.1073/pnas.1412569111. Epub 2014 Sep 23.

19.
20.

The role of CSA and CSB protein in the oxidative stress response.

D'Errico M, Pascucci B, Iorio E, Van Houten B, Dogliotti E.

Mech Ageing Dev. 2013 May-Jun;134(5-6):261-9. doi: 10.1016/j.mad.2013.03.006. Epub 2013 Apr 3. Review.

PMID:
23562424

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