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Items: 1 to 20 of 362

2.

Porphyromonas gingivalis LPS stimulates the expression of LPS-binding protein in human oral keratinocytes in vitro.

Ding PH, Wang CY, Darveau RP, Jin L.

Innate Immun. 2013 Feb;19(1):66-75. doi: 10.1177/1753425912450348. Epub 2012 Jun 26.

PMID:
22736337
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Role of the phosphatidylinositol 3 kinase-Akt pathway in the regulation of IL-10 and IL-12 by Porphyromonas gingivalis lipopolysaccharide.

Martin M, Schifferle RE, Cuesta N, Vogel SN, Katz J, Michalek SM.

J Immunol. 2003 Jul 15;171(2):717-25.

5.

Lipopolysaccharide (LPS) of Porphyromonas gingivalis induces IL-1beta, TNF-alpha and IL-6 production by THP-1 cells in a way different from that of Escherichia coli LPS.

Diya Zhang, Lili Chen, Shenglai Li, Zhiyuan Gu, Jie Yan.

Innate Immun. 2008 Apr;14(2):99-107. doi: 10.1177/1753425907088244.

PMID:
18713726
6.

3LPS-binding protein and its interactions with P. gingivalis LPS modulate pro-inflammatory response and Toll-like receptor signaling in human oral keratinocytes.

Ding PH, Darveau RP, Wang CY, Jin L.

PLoS One. 2017 Apr 6;12(4):e0173223. doi: 10.1371/journal.pone.0173223. eCollection 2017. Erratum in: PLoS One. 2017 May 1;12 (5):e0176996.

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Female sex hormones modulate Porphyromonas gingivalis lipopolysaccharide-induced Toll-like receptor signaling in primary human monocytes.

Jitprasertwong P, Charadram N, Kumphune S, Pongcharoen S, Sirisinha S.

J Periodontal Res. 2016 Jun;51(3):395-406. doi: 10.1111/jre.12320. Epub 2015 Sep 14.

PMID:
26364725
10.

Lipopolysaccharide-binding protein inhibits toll-like receptor 2 activation by lipoteichoic acid in human odontoblast-like cells.

Carrouel F, Staquet MJ, Keller JF, Baudouin C, Msika P, Bleicher F, Alliot-Licht B, Farges JC.

J Endod. 2013 Aug;39(8):1008-14. doi: 10.1016/j.joen.2013.04.020. Epub 2013 May 16.

PMID:
23880268
11.

The role of lipopolysaccharide-binding protein in innate immunity: a revisit and its relevance to oral/periodontal health.

Ding PH, Jin LJ.

J Periodontal Res. 2014 Feb;49(1):1-9. doi: 10.1111/jre.12081. Epub 2013 Apr 18. Review.

PMID:
23601005
12.

Lipopolysaccharide-binding protein critically regulates lipopolysaccharide-induced IFN-beta signaling pathway in human monocytes.

Kato A, Ogasawara T, Homma T, Saito H, Matsumoto K.

J Immunol. 2004 May 15;172(10):6185-94.

13.

Porphyromonas gingivalis lipopolysaccharide is both agonist and antagonist for p38 mitogen-activated protein kinase activation.

Darveau RP, Arbabi S, Garcia I, Bainbridge B, Maier RV.

Infect Immun. 2002 Apr;70(4):1867-73.

14.

Tormentic acid inhibits LPS-induced inflammatory response in human gingival fibroblasts via inhibition of TLR4-mediated NF-κB and MAPK signalling pathway.

Jian CX, Li MZ, Zheng WY, He Y, Ren Y, Wu ZM, Fan QS, Hu YH, Li CJ.

Arch Oral Biol. 2015 Sep;60(9):1327-32. doi: 10.1016/j.archoralbio.2015.05.005. Epub 2015 May 22.

PMID:
26123747
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16.

Porphorymonas gingivalis induces intracellular adhesion molecule-1 expression in endothelial cells through the nuclear factor-kappaB pathway, but not through the p38 MAPK pathway.

Zhang D, Zheng H, Zhao J, Lin L, Li C, Liu J, Pan Y.

J Periodontal Res. 2011 Feb;46(1):31-8. doi: 10.1111/j.1600-0765.2010.01305.x. Epub 2010 Sep 3.

PMID:
20825555
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18.

Role of JNK and NF-κB pathways in Porphyromonas gingivalis LPS-induced vascular cell adhesion molecule-1 expression in human aortic endothelial cells.

Liu B, Wang J, Cheng L, Liang J.

Mol Med Rep. 2013 Nov;8(5):1594-600. doi: 10.3892/mmr.2013.1685. Epub 2013 Sep 17.

PMID:
24045414

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