Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 193

1.

Role of SOCS2 in modulating heart damage and function in a murine model of acute Chagas disease.

Esper L, Roman-Campos D, Lara A, Brant F, Castro LL, Barroso A, Araujo RR, Vieira LQ, Mukherjee S, Gomes ER, Rocha NN, Ramos IP, Lisanti MP, Campos CF, Arantes RM, Guatimosim S, Weiss LM, Cruz JS, Tanowitz HB, Teixeira MM, Machado FS.

Am J Pathol. 2012 Jul;181(1):130-40. doi: 10.1016/j.ajpath.2012.03.042. Epub 2012 May 29.

2.

The Aryl Hydrocarbon Receptor Modulates Production of Cytokines and Reactive Oxygen Species and Development of Myocarditis during Trypanosoma cruzi Infection.

Barroso A, Gualdrón-López M, Esper L, Brant F, Araújo RR, Carneiro MB, Ávila TV, Souza DG, Vieira LQ, Rachid MA, Tanowitz HB, Teixeira MM, Machado FS.

Infect Immun. 2016 Sep 19;84(10):3071-82. doi: 10.1128/IAI.00575-16. Print 2016 Oct.

3.

Experimental Trypanosoma cruzi infection in platelet-activating factor receptor-deficient mice.

Talvani A, Santana G, Barcelos LS, Ishii S, Shimizu T, Romanha AJ, Silva JS, Soares MB, Teixeira MM.

Microbes Infect. 2003 Jul;5(9):789-96.

PMID:
12850205
4.

Differential susceptibility to acute Trypanosoma cruzi infection in BALB/c and C57BL/6 mice is not associated with a distinct parasite load but cytokine abnormalities.

Roggero E, Perez A, Tamae-Kakazu M, Piazzon I, Nepomnaschy I, Wietzerbin J, Serra E, Revelli S, Bottasso O.

Clin Exp Immunol. 2002 Jun;128(3):421-8.

5.

Kinetics of cytokine gene expression in experimental chagasic cardiomyopathy: tissue parasitism and endogenous IFN-gamma as important determinants of chemokine mRNA expression during infection with Trypanosoma cruzi.

Talvani A, Ribeiro CS, Aliberti JC, Michailowsky V, Santos PV, Murta SM, Romanha AJ, Almeida IC, Farber J, Lannes-Vieira J, Silva JS, Gazzinelli RT.

Microbes Infect. 2000 Jul;2(8):851-66.

PMID:
10962268
6.

5-lipoxygenase is a key determinant of acute myocardial inflammation and mortality during Trypanosoma cruzi infection.

Pavanelli WR, Gutierrez FR, Mariano FS, Prado CM, Ferreira BR, Teixeira MM, Canetti C, Rossi MA, Cunha FQ, Silva JS.

Microbes Infect. 2010 Aug;12(8-9):587-97. doi: 10.1016/j.micinf.2010.03.016. Epub 2010 Apr 8.

PMID:
20381637
7.

Suppressor of cytokine signaling 2 modulates the immune response profile and development of experimental cerebral malaria.

Brant F, Miranda AS, Esper L, Gualdrón-López M, Cisalpino D, de Souza DDG, Rachid MA, Tanowitz HB, Teixeira MM, Teixeira AL, Machado FS.

Brain Behav Immun. 2016 May;54:73-85. doi: 10.1016/j.bbi.2016.01.002. Epub 2016 Jan 4.

PMID:
26765997
8.

NADPH oxidase inhibition ameliorates Trypanosoma cruzi-induced myocarditis during Chagas disease.

Dhiman M, Garg NJ.

J Pathol. 2011 Dec;225(4):583-96. doi: 10.1002/path.2975. Epub 2011 Sep 26.

9.
10.

The receptor Slamf1 on the surface of myeloid lineage cells controls susceptibility to infection by Trypanosoma cruzi.

Calderón J, Maganto-Garcia E, Punzón C, Carrión J, Terhorst C, Fresno M.

PLoS Pathog. 2012;8(7):e1002799. doi: 10.1371/journal.ppat.1002799. Epub 2012 Jul 12.

11.

Increased Trypanosoma cruzi invasion and heart fibrosis associated with high transforming growth factor beta levels in mice deficient in alpha(2)-macroglobulin.

Waghabi MC, Coutinho CM, Soeiro MN, Pereira MC, Feige JJ, Keramidas M, Cosson A, Minoprio P, Van Leuven F, Araújo-Jorge TC.

Infect Immun. 2002 Sep;70(9):5115-23.

12.

Perforin-expressing cytotoxic cells contribute to chronic cardiomyopathy in Trypanosoma cruzi infection.

Silverio JC, de-Oliveira-Pinto LM, da Silva AA, de Oliveira GM, Lannes-Vieira J.

Int J Exp Pathol. 2010 Feb;91(1):72-86. doi: 10.1111/j.1365-2613.2009.00670.x. Epub 2009 Oct 28.

13.
14.

Administration of granulocyte colony-stimulating factor induces immunomodulation, recruitment of T regulatory cells, reduction of myocarditis and decrease of parasite load in a mouse model of chronic Chagas disease cardiomyopathy.

Vasconcelos JF, Souza BS, Lins TF, Garcia LM, Kaneto CM, Sampaio GP, de Alcântara AC, Meira CS, Macambira SG, Ribeiro-dos-Santos R, Soares MB.

FASEB J. 2013 Dec;27(12):4691-702. doi: 10.1096/fj.13-229351. Epub 2013 Aug 20. Erratum in: FASEB J. 2015 Nov;29(11):4756.

PMID:
23964077
16.

IL-17 produced during Trypanosoma cruzi infection plays a central role in regulating parasite-induced myocarditis.

da Matta Guedes PM, Gutierrez FR, Maia FL, Milanezi CM, Silva GK, Pavanelli WR, Silva JS.

PLoS Negl Trop Dis. 2010 Feb 16;4(2):e604. doi: 10.1371/journal.pntd.0000604.

17.

CD8+ T-cells expressing interferon gamma or perforin play antagonistic roles in heart injury in experimental Trypanosoma cruzi-elicited cardiomyopathy.

Silverio JC, Pereira IR, Cipitelli Mda C, Vinagre NF, Rodrigues MM, Gazzinelli RT, Lannes-Vieira J.

PLoS Pathog. 2012;8(4):e1002645. doi: 10.1371/journal.ppat.1002645. Epub 2012 Apr 19.

18.

IFN-gamma, but not nitric oxide or specific IgG, is essential for the in vivo control of low-virulence Sylvio X10/4 Trypanosoma cruzi parasites.

Marinho CR, Nuñez-Apaza LN, Martins-Santos R, Bastos KR, Bombeiro AL, Bucci DZ, Sardinha LR, Lima MR, Alvarez JM.

Scand J Immunol. 2007 Aug-Sep;66(2-3):297-308.

19.

Trypanosoma cruzi-infected cardiomyocytes produce chemokines and cytokines that trigger potent nitric oxide-dependent trypanocidal activity.

Machado FS, Martins GA, Aliberti JC, Mestriner FL, Cunha FQ, Silva JS.

Circulation. 2000 Dec 12;102(24):3003-8.

PMID:
11113053
20.

Granulocyte colony-stimulating factor partially repairs the damage provoked by Trypanosoma cruzi in murine myocardium.

González MN, Dey N, Garg NJ, Postan M.

Int J Cardiol. 2013 Oct 3;168(3):2567-74. doi: 10.1016/j.ijcard.2013.03.049. Epub 2013 Apr 15.

Supplemental Content

Support Center