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Items: 1 to 20 of 137

1.

Porphyromonas gingivalis SerB-mediated dephosphorylation of host cell cofilin modulates invasion efficiency.

Moffatt CE, Inaba H, Hirano T, Lamont RJ.

Cell Microbiol. 2012 Apr;14(4):577-88. doi: 10.1111/j.1462-5822.2011.01743.x. Epub 2012 Feb 2.

2.

Role of Porphyromonas gingivalis SerB in gingival epithelial cell cytoskeletal remodeling and cytokine production.

Hasegawa Y, Tribble GD, Baker HV, Mans JJ, Handfield M, Lamont RJ.

Infect Immun. 2008 Jun;76(6):2420-7. doi: 10.1128/IAI.00156-08. Epub 2008 Apr 7.

3.

Role of Porphyromonas gingivalis phosphoserine phosphatase enzyme SerB in inflammation, immune response, and induction of alveolar bone resorption in rats.

Bainbridge B, Verma RK, Eastman C, Yehia B, Rivera M, Moffatt C, Bhattacharyya I, Lamont RJ, Kesavalu L.

Infect Immun. 2010 Nov;78(11):4560-9. doi: 10.1128/IAI.00703-10. Epub 2010 Aug 30.

4.

The serine phosphatase SerB of Porphyromonas gingivalis suppresses IL-8 production by dephosphorylation of NF-κB RelA/p65.

Takeuchi H, Hirano T, Whitmore SE, Morisaki I, Amano A, Lamont RJ.

PLoS Pathog. 2013;9(4):e1003326. doi: 10.1371/journal.ppat.1003326. Epub 2013 Apr 18.

5.

Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.

Roberts JS, Atanasova KR, Lee J, Diamond G, Deguzman J, Hee Choi C, Yilmaz Ö.

Front Cell Infect Microbiol. 2017 Jul 4;7:291. doi: 10.3389/fcimb.2017.00291. eCollection 2017.

6.

A novel kinase function of a nucleoside-diphosphate-kinase homologue in Porphyromonas gingivalis is critical in subversion of host cell apoptosis by targeting heat-shock protein 27.

Lee J, Roberts JS, Atanasova KR, Chowdhury N, Yilmaz Ö.

Cell Microbiol. 2018 May;20(5):e12825. doi: 10.1111/cmi.12825. Epub 2018 Feb 7.

PMID:
29359393
7.

Porphyromonas gingivalis initiates a mesenchymal-like transition through ZEB1 in gingival epithelial cells.

Sztukowska MN, Ojo A, Ahmed S, Carenbauer AL, Wang Q, Shumway B, Jenkinson HF, Wang H, Darling DS, Lamont RJ.

Cell Microbiol. 2016 Jun;18(6):844-58. doi: 10.1111/cmi.12554. Epub 2016 Jan 13.

8.

Porphyromonas gingivalis entry into gingival epithelial cells modulated by Fusobacterium nucleatum is dependent on lipid rafts.

Saito A, Kokubu E, Inagaki S, Imamura K, Kita D, Lamont RJ, Ishihara K.

Microb Pathog. 2012 Nov-Dec;53(5-6):234-42. doi: 10.1016/j.micpath.2012.08.005. Epub 2012 Aug 29.

9.

LptO (PG0027) Is Required for Lipid A 1-Phosphatase Activity in Porphyromonas gingivalis W50.

Rangarajan M, Aduse-Opoku J, Hashim A, McPhail G, Luklinska Z, Haurat MF, Feldman MF, Curtis MA.

J Bacteriol. 2017 May 9;199(11). pii: e00751-16. doi: 10.1128/JB.00751-16. Print 2017 Jun 1.

10.

Induction of B7-H1 receptor by bacterial cells fractions of Porphyromonas gingivalis on human oral epithelial cells: B7-H1 induction by Porphyromonas gingivalis fractions.

Groeger S, Jarzina F, Mamat U, Meyle J.

Immunobiology. 2017 Feb;222(2):137-147. doi: 10.1016/j.imbio.2016.10.011. Epub 2016 Oct 17.

PMID:
28164807
11.

Efficient Salmonella entry requires activity cycles of host ADF and cofilin.

Dai S, Sarmiere PD, Wiggan O, Bamburg JR, Zhou D.

Cell Microbiol. 2004 May;6(5):459-71.

PMID:
15056216
12.

Cell cycle-associated changes in Slingshot phosphatase activity and roles in cytokinesis in animal cells.

Kaji N, Ohashi K, Shuin M, Niwa R, Uemura T, Mizuno K.

J Biol Chem. 2003 Aug 29;278(35):33450-5. Epub 2003 Jun 14.

13.

Dynamic cofilin phosphorylation in the control of lamellipodial actin homeostasis.

Jovceva E, Larsen MR, Waterfield MD, Baum B, Timms JF.

J Cell Sci. 2007 Jun 1;120(Pt 11):1888-97. Epub 2007 May 15.

14.

Phosphoinositide 3-kinase-mediated activation of cofilin phosphatase Slingshot and its role for insulin-induced membrane protrusion.

Nishita M, Wang Y, Tomizawa C, Suzuki A, Niwa R, Uemura T, Mizuno K.

J Biol Chem. 2004 Feb 20;279(8):7193-8. Epub 2003 Nov 26.

15.

Porphyromonas gingivalis-induced IL-33 down-regulates hCAP-18/LL-37 production in human gingival epithelial cells.

Tada H, Shimizu T, Matsushita K, Takada H.

Biomed Res. 2017;38(3):167-173. doi: 10.2220/biomedres.38.167.

16.

Gingipain-dependent degradation of mammalian target of rapamycin pathway proteins by the periodontal pathogen Porphyromonas gingivalis during invasion.

Stafford P, Higham J, Pinnock A, Murdoch C, Douglas CW, Stafford GP, Lambert DW.

Mol Oral Microbiol. 2013 Oct;28(5):366-78. doi: 10.1111/omi.12030. Epub 2013 May 29.

17.

HmuY is an important virulence factor for Porphyromonas gingivalis growth in the heme-limited host environment and infection of macrophages.

Olczak T, Sosicka P, Olczak M.

Biochem Biophys Res Commun. 2015 Nov 27;467(4):748-53. doi: 10.1016/j.bbrc.2015.10.070. Epub 2015 Oct 19.

PMID:
26482851
18.

Nucleoside-Diphosphate-Kinase of P. gingivalis is Secreted from Epithelial Cells In the Absence of a Leader Sequence Through a Pannexin-1 Interactome.

Atanasova K, Lee J, Roberts J, Lee K, Ojcius DM, Yilmaz Ö.

Sci Rep. 2016 Nov 24;6:37643. doi: 10.1038/srep37643.

19.

Protein kinase D regulates cofilin activity through p21-activated kinase 4.

Spratley SJ, Bastea LI, Döppler H, Mizuno K, Storz P.

J Biol Chem. 2011 Sep 30;286(39):34254-61. doi: 10.1074/jbc.M111.259424. Epub 2011 Aug 9.

20.

Intracellular periodontal pathogen exploits recycling pathway to exit from infected cells.

Takeuchi H, Takada A, Kuboniwa M, Amano A.

Cell Microbiol. 2016 Jul;18(7):928-48. doi: 10.1111/cmi.12551. Epub 2016 Feb 8.

PMID:
26617273

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