Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 168

1.

ATP-dependent activation of an inflammasome in primary gingival epithelial cells infected by Porphyromonas gingivalis.

Yilmaz O, Sater AA, Yao L, Koutouzis T, Pettengill M, Ojcius DM.

Cell Microbiol. 2010 Feb;12(2):188-98. doi: 10.1111/j.1462-5822.2009.01390.x. Epub 2009 Oct 6.

2.

P2X4 assembles with P2X7 and pannexin-1 in gingival epithelial cells and modulates ATP-induced reactive oxygen species production and inflammasome activation.

Hung SC, Choi CH, Said-Sadier N, Johnson L, Atanasova KR, Sellami H, Yilmaz Ö, Ojcius DM.

PLoS One. 2013 Jul 25;8(7):e70210. doi: 10.1371/journal.pone.0070210. Print 2013.

3.

Induction of heme oxygenase-1 attenuates lipopolysaccharide-induced inflammasome activation in human gingival epithelial cells.

Li H, Zhou X, Zhang J.

Int J Mol Med. 2014 Oct;34(4):1039-44. doi: 10.3892/ijmm.2014.1865. Epub 2014 Jul 25.

PMID:
25069505
5.

Fusobacterium nucleatum infection of gingival epithelial cells leads to NLRP3 inflammasome-dependent secretion of IL-1β and the danger signals ASC and HMGB1.

Bui FQ, Johnson L, Roberts J, Hung SC, Lee J, Atanasova KR, Huang PR, Yilmaz Ö, Ojcius DM.

Cell Microbiol. 2016 Jul;18(7):970-81. doi: 10.1111/cmi.12560. Epub 2016 Feb 5.

6.

Losartan inhibits LPS + ATP-induced IL-1beta secretion from mouse primary macrophages by suppressing NALP3 inflammasome.

Wang F, Huang L, Peng ZZ, Tang YT, Lu MM, Peng Y, Mel WJ, Wu L, Mo ZH, Meng J, Tao LJ.

Pharmazie. 2014 Sep;69(9):680-4.

PMID:
25272939
7.

Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.

Park E, Na HS, Song YR, Shin SY, Kim YM, Chung J.

Infect Immun. 2014 Jan;82(1):112-23. doi: 10.1128/IAI.00862-13. Epub 2013 Oct 14.

8.

Porphyromonas gingivalis attenuates ATP-mediated inflammasome activation and HMGB1 release through expression of a nucleoside-diphosphate kinase.

Johnson L, Atanasova KR, Bui PQ, Lee J, Hung SC, Yilmaz Ö, Ojcius DM.

Microbes Infect. 2015 May;17(5):369-77. doi: 10.1016/j.micinf.2015.03.010. Epub 2015 Mar 27.

9.

[Study on the role of NALP3 inflammasome in Porphyromonas gingivalis lipopolysaccharide induced RAW264.7].

Yang F, Chen MY, Hu YY, Wang CN.

Zhonghua Kou Qiang Yi Xue Za Zhi. 2017 May 9;52(5):289-293. doi: 10.3760/cma.j.issn.1002-0098.2017.05.006. Chinese.

PMID:
28482444
10.

Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

Bostanci N, Emingil G, Saygan B, Turkoglu O, Atilla G, Curtis MA, Belibasakis GN.

Clin Exp Immunol. 2009 Sep;157(3):415-22. doi: 10.1111/j.1365-2249.2009.03972.x.

11.

Atrial natriuretic peptide down-regulates LPS/ATP-mediated IL-1β release by inhibiting NF-kB, NLRP3 inflammasome and caspase-1 activation in THP-1 cells.

Mezzasoma L, Antognelli C, Talesa VN.

Immunol Res. 2016 Feb;64(1):303-12. doi: 10.1007/s12026-015-8751-0.

PMID:
26616294
12.

Porphyromonas gingivalis infection sequesters pro-apoptotic Bad through Akt in primary gingival epithelial cells.

Yao L, Jermanus C, Barbetta B, Choi C, Verbeke P, Ojcius DM, Yilmaz O.

Mol Oral Microbiol. 2010 Apr;25(2):89-101. doi: 10.1111/j.2041-1014.2010.00569.x.

13.

Porphyromonas gingivalis induction of microRNA-203 expression controls suppressor of cytokine signaling 3 in gingival epithelial cells.

Moffatt CE, Lamont RJ.

Infect Immun. 2011 Jul;79(7):2632-7. doi: 10.1128/IAI.00082-11. Epub 2011 May 2.

14.

Cryopyrin activates the inflammasome in response to toxins and ATP.

Mariathasan S, Weiss DS, Newton K, McBride J, O'Rourke K, Roose-Girma M, Lee WP, Weinrauch Y, Monack DM, Dixit VM.

Nature. 2006 Mar 9;440(7081):228-32. Epub 2006 Jan 11.

PMID:
16407890
15.

Porphyromonas gingivalis-Derived Lipopolysaccharide Combines Hypoxia to Induce Caspase-1 Activation in Periodontitis.

Cheng R, Liu W, Zhang R, Feng Y, Bhowmick NA, Hu T.

Front Cell Infect Microbiol. 2017 Nov 14;7:474. doi: 10.3389/fcimb.2017.00474. eCollection 2017.

16.

Porphyromonas gingivalis lipopolysaccharide induces shedding of syndecan-1 expressed by gingival epithelial cells.

Andrian E, Grenier D, Rouabhia M.

J Cell Physiol. 2005 Jul;204(1):178-83.

PMID:
15648090
17.

Soluble uric acid increases NALP3 inflammasome and interleukin-1β expression in human primary renal proximal tubule epithelial cells through the Toll-like receptor 4-mediated pathway.

Xiao J, Zhang XL, Fu C, Han R, Chen W, Lu Y, Ye Z.

Int J Mol Med. 2015 May;35(5):1347-54. doi: 10.3892/ijmm.2015.2148. Epub 2015 Mar 18.

PMID:
25813103
18.

A Dual Role for P2X7 Receptor during Porphyromonas gingivalis Infection.

Ramos-Junior ES, Morandini AC, Almeida-da-Silva CL, Franco EJ, Potempa J, Nguyen KA, Oliveira AC, Zamboni DS, Ojcius DM, Scharfstein J, Coutinho-Silva R.

J Dent Res. 2015 Sep;94(9):1233-42. doi: 10.1177/0022034515593465. Epub 2015 Jul 7.

19.

Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.

Roberts JS, Atanasova KR, Lee J, Diamond G, Deguzman J, Hee Choi C, Yilmaz Ö.

Front Cell Infect Microbiol. 2017 Jul 4;7:291. doi: 10.3389/fcimb.2017.00291. eCollection 2017.

20.

Differential inflammasome activation by Porphyromonas gingivalis and cholesterol crystals in human macrophages and coronary artery endothelial cells.

Champaiboon C, Poolgesorn M, Wisitrasameewong W, Sa-Ard-Iam N, Rerkyen P, Mahanonda R.

Atherosclerosis. 2014 Jul;235(1):38-44. doi: 10.1016/j.atherosclerosis.2014.04.007. Epub 2014 Apr 25.

PMID:
24814648

Supplemental Content

Support Center