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Items: 1 to 20 of 333

1.

Generation of lys-gingipain protease activity in Porphyromonas gingivalis W50 is independent of Arg-gingipain protease activities.

Aduse-Opoku J, Davies NN, Gallagher A, Hashim A, Evans HE, Rangarajan M, Slaney JM, Curtis MA.

Microbiology. 2000 Aug;146 ( Pt 8):1933-40.

PMID:
10931897
2.

Role of RgpA, RgpB, and Kgp proteinases in virulence of Porphyromonas gingivalis W50 in a murine lesion model.

O'Brien-Simpson NM, Paolini RA, Hoffmann B, Slakeski N, Dashper SG, Reynolds EC.

Infect Immun. 2001 Dec;69(12):7527-34.

4.

Expression of Arg-Gingipain RgpB is required for correct glycosylation and stability of monomeric Arg-gingipain RgpA from Porphyromonas gingivalis W50.

Rangarajan M, Hashim A, Aduse-Opoku J, Paramonov N, Hounsell EF, Curtis MA.

Infect Immun. 2005 Aug;73(8):4864-78.

5.
6.

Porphyromonas gingivalis gingipains: the molecular teeth of a microbial vampire.

O-Brien-Simpson NM, Veith PD, Dashper SG, Reynolds EC.

Curr Protein Pept Sci. 2003 Dec;4(6):409-26. Review.

PMID:
14683427
7.

The roles of RgpB and Kgp in late onset gingipain activity in the vimA-defective mutant of Porphyromonas gingivalis W83.

Dou Y, Robles A, Roy F, Aruni AW, Sandberg L, Nothnagel E, Fletcher HM.

Mol Oral Microbiol. 2015 Oct;30(5):347-60. doi: 10.1111/omi.12098. Epub 2015 May 8.

8.

Gingipains from Porphyromonas gingivalis play a significant role in induction and regulation of CXCL8 in THP-1 cells.

Jayaprakash K, Khalaf H, Bengtsson T.

BMC Microbiol. 2014 Jul 18;14:193. doi: 10.1186/1471-2180-14-193.

10.

Attenuation of the virulence of Porphyromonas gingivalis by using a specific synthetic Kgp protease inhibitor.

Curtis MA, Aduse Opoku J, Rangarajan M, Gallagher A, Sterne JA, Reid CR, Evans HE, Samuelsson B.

Infect Immun. 2002 Dec;70(12):6968-75.

11.

Hemoglobin hydrolysis and heme acquisition by Porphyromonas gingivalis.

Dashper SG, Cross KJ, Slakeski N, Lissel P, Aulakh P, Moore C, Reynolds EC.

Oral Microbiol Immunol. 2004 Feb;19(1):50-6.

PMID:
14678474
12.
14.

Roles of Arg- and Lys-gingipains in coaggregation of Porphyromonas gingivalis: identification of its responsible molecules in translation products of rgpA, kgp, and hagA genes.

Abe N, Baba A, Takii R, Nakayama K, Kamaguchi A, Shibata Y, Abiko Y, Okamoto K, Kadowaki T, Yamamoto K.

Biol Chem. 2004 Nov;385(11):1041-7.

PMID:
15576324
15.

Involvement of arginine-specific cysteine proteinase (Arg-gingipain) in fimbriation of Porphyromonas gingivalis.

Nakayama K, Yoshimura F, Kadowaki T, Yamamoto K.

J Bacteriol. 1996 May;178(10):2818-24.

16.

Hemoglobinase activity of the lysine gingipain protease (Kgp) of Porphyromonas gingivalis W83.

Lewis JP, Dawson JA, Hannis JC, Muddiman D, Macrina FL.

J Bacteriol. 1999 Aug;181(16):4905-13.

17.

Structural insights unravel the zymogenic mechanism of the virulence factor gingipain K from Porphyromonas gingivalis, a causative agent of gum disease from the human oral microbiome.

Pomowski A, Usón I, Nowakowska Z, Veillard F, Sztukowska MN, Guevara T, Goulas T, Mizgalska D, Nowak M, Potempa B, Huntington JA, Potempa J, Gomis-Rüth FX.

J Biol Chem. 2017 Apr 7;292(14):5724-5735. doi: 10.1074/jbc.M117.776724. Epub 2017 Feb 14.

18.

The role of gingipains in the pathogenesis of periodontal disease.

Imamura T.

J Periodontol. 2003 Jan;74(1):111-8. Review.

PMID:
12593605
19.

Altered gingipain maturation in vimA- and vimE-defective isogenic mutants of Porphyromonas gingivalis.

Vanterpool E, Roy F, Sandberg L, Fletcher HM.

Infect Immun. 2005 Mar;73(3):1357-66.

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