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Items: 1 to 20 of 75

2.

Genotype by random environmental interactions gives an advantage to non-favored minor alleles.

Mahdipour-Shirayeh A, Darooneh AH, Long AD, Komarova NL, Kohandel M.

Sci Rep. 2017 Jul 12;7(1):5193. doi: 10.1038/s41598-017-05375-0.

3.

The Beavis Effect in Next-Generation Mapping Panels in Drosophila melanogaster.

King EG, Long AD.

G3 (Bethesda). 2017 Jun 7;7(6):1643-1652. doi: 10.1534/g3.117.041426.

4.

A Model of Compound Heterozygous, Loss-of-Function Alleles Is Broadly Consistent with Observations from Complex-Disease GWAS Datasets.

Sanjak JS, Long AD, Thornton KR.

PLoS Genet. 2017 Jan 19;13(1):e1006573. doi: 10.1371/journal.pgen.1006573. eCollection 2017 Jan.

5.

Genome-wide analysis of long-term evolutionary domestication in Drosophila melanogaster.

Phillips MA, Long AD, Greenspan ZS, Greer LF, Burke MK, Villeponteau B, Matsagas KC, Rizza CL, Mueller LD, Rose MR.

Sci Rep. 2016 Dec 22;6:39281. doi: 10.1038/srep39281.

6.

Contiguous and accurate de novo assembly of metazoan genomes with modest long read coverage.

Chakraborty M, Baldwin-Brown JG, Long AD, Emerson JJ.

Nucleic Acids Res. 2016 Nov 2;44(19):e147. Epub 2016 Jul 25.

7.

Efficient Software for Multi-marker, Region-Based Analysis of GWAS Data.

Sanjak JS, Long AD, Thornton KR.

G3 (Bethesda). 2016 Apr 7;6(4):1023-30. doi: 10.1534/g3.115.026013.

8.

Identifying Loci Contributing to Natural Variation in Xenobiotic Resistance in Drosophila.

Najarro MA, Hackett JL, Smith BR, Highfill CA, King EG, Long AD, Macdonald SJ.

PLoS Genet. 2015 Nov 30;11(11):e1005663. doi: 10.1371/journal.pgen.1005663. eCollection 2015 Nov. Erratum in: PLoS Genet. 2016 Jan;12(1):e1005830.

9.

Gene expression variation in Drosophila melanogaster due to rare transposable element insertion alleles of large effect.

Cridland JM, Thornton KR, Long AD.

Genetics. 2015 Jan;199(1):85-93. doi: 10.1534/genetics.114.170837. Epub 2014 Oct 21.

10.

Fine-mapping nicotine resistance loci in Drosophila using a multiparent advanced generation inter-cross population.

Marriage TN, King EG, Long AD, Macdonald SJ.

Genetics. 2014 Sep;198(1):45-57. doi: 10.1534/genetics.114.162107.

11.

Using Drosophila melanogaster to identify chemotherapy toxicity genes.

King EG, Kislukhin G, Walters KN, Long AD.

Genetics. 2014 Sep;198(1):31-43. doi: 10.1534/genetics.114.161968.

12.

Dissecting complex traits using the Drosophila Synthetic Population Resource.

Long AD, Macdonald SJ, King EG.

Trends Genet. 2014 Nov;30(11):488-95. doi: 10.1016/j.tig.2014.07.009. Epub 2014 Aug 28. Review.

13.

Standing genetic variation drives repeatable experimental evolution in outcrossing populations of Saccharomyces cerevisiae.

Burke MK, Liti G, Long AD.

Mol Biol Evol. 2014 Dec;31(12):3228-39. doi: 10.1093/molbev/msu256. Epub 2014 Aug 28.

14.

Genetic dissection of the Drosophila melanogaster female head transcriptome reveals widespread allelic heterogeneity.

King EG, Sanderson BJ, McNeil CL, Long AD, Macdonald SJ.

PLoS Genet. 2014 May 8;10(5):e1004322. doi: 10.1371/journal.pgen.1004322. eCollection 2014 May.

15.

The power to detect quantitative trait loci using resequenced, experimentally evolved populations of diploid, sexual organisms.

Baldwin-Brown JG, Long AD, Thornton KR.

Mol Biol Evol. 2014 Apr;31(4):1040-55. doi: 10.1093/molbev/msu048. Epub 2014 Jan 18.

16.

Genome-wide association study of extreme longevity in Drosophila melanogaster.

Burke MK, King EG, Shahrestani P, Rose MR, Long AD.

Genome Biol Evol. 2014 Jan;6(1):1-11. doi: 10.1093/gbe/evt180.

17.

Abundance and distribution of transposable elements in two Drosophila QTL mapping resources.

Cridland JM, Macdonald SJ, Long AD, Thornton KR.

Mol Biol Evol. 2013 Oct;30(10):2311-27. doi: 10.1093/molbev/mst129. Epub 2013 Jul 24.

18.

The genetic architecture of methotrexate toxicity is similar in Drosophila melanogaster and humans.

Kislukhin G, King EG, Walters KN, Macdonald SJ, Long AD.

G3 (Bethesda). 2013 Aug 7;3(8):1301-10. doi: 10.1534/g3.113.006619.

19.

Controlled synthesis and transfer of large-area WS2 sheets: from single layer to few layers.

Elías AL, Perea-López N, Castro-Beltrán A, Berkdemir A, Lv R, Feng S, Long AD, Hayashi T, Kim YA, Endo M, Gutiérrez HR, Pradhan NR, Balicas L, Mallouk TE, López-Urías F, Terrones H, Terrones M.

ACS Nano. 2013 Jun 25;7(6):5235-42. doi: 10.1021/nn400971k. Epub 2013 May 15.

PMID:
23647141
20.

Properties and modeling of GWAS when complex disease risk is due to non-complementing, deleterious mutations in genes of large effect.

Thornton KR, Foran AJ, Long AD.

PLoS Genet. 2013;9(2):e1003258. doi: 10.1371/journal.pgen.1003258. Epub 2013 Feb 21.

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