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Items: 1 to 20 of 208


Distinct MHC class I-dependent NK cell-activating receptors control cytomegalovirus infection in different mouse strains.

Pyzik M, Charbonneau B, Gendron-Pontbriand EM, Babić M, Krmpotić A, Jonjić S, Vidal SM.

J Exp Med. 2011 May 9;208(5):1105-17. doi: 10.1084/jem.20101831. Epub 2011 Apr 25.


Viral MHC class I-like molecule allows evasion of NK cell effector responses in vivo.

Pyzik M, Dumaine A, Charbonneau B, Fodil-Cornu N, Jonjic S, Vidal SM.

J Immunol. 2014 Dec 15;193(12):6061-9. doi: 10.4049/jimmunol.1401386. Epub 2014 Nov 12. Erratum in: J Immunol. 2015 Jun 15;194(12):6192. Dumaine, Anne A [Corrected to Dumaine, Anne].


NK cell receptor/H2-Dk-dependent host resistance to viral infection is quantitatively modulated by H2q inhibitory signals.

Fodil-Cornu N, Loredo-Osti JC, Vidal SM.

PLoS Genet. 2011 Apr;7(4):e1001368. doi: 10.1371/journal.pgen.1001368. Epub 2011 Apr 21.


Multiparametric analysis of host response to murine cytomegalovirus in MHC class I-disparate mice reveals primacy of Dk-licensed Ly49G2+ NK cells in viral control.

Prince J, Lundgren A, Stadnisky MD, Nash WT, Beeber A, Turner SD, Brown MG.

J Immunol. 2013 Nov 1;191(9):4709-19. doi: 10.4049/jimmunol.1301388. Epub 2013 Sep 25.


Cmv1-independent antiviral role of NK cells revealed in murine cytomegalovirus-infected New Zealand White mice.

Rodriguez M, Sabastian P, Clark P, Brown MG.

J Immunol. 2004 Nov 15;173(10):6312-8.


Ly49h-deficient C57BL/6 mice: a new mouse cytomegalovirus-susceptible model remains resistant to unrelated pathogens controlled by the NK gene complex.

Fodil-Cornu N, Lee SH, Belanger S, Makrigiannis AP, Biron CA, Buller RM, Vidal SM.

J Immunol. 2008 Nov 1;181(9):6394-405.


Viral infection transiently reverses activation receptor-mediated NK cell hyporesponsiveness in an MHC class I-independent mechanism.

Mazumdar B, Bolanos FD, Tripathy SK.

Eur J Immunol. 2013 May;43(5):1345-55. doi: 10.1002/eji.201243215. Epub 2013 Apr 9.


Direct recognition of cytomegalovirus by activating and inhibitory NK cell receptors.

Arase H, Mocarski ES, Campbell AE, Hill AB, Lanier LL.

Science. 2002 May 17;296(5571):1323-6. Epub 2002 Apr 11.


Natural Killer Cell Sensing of Infected Cells Compensates for MyD88 Deficiency but Not IFN-I Activity in Resistance to Mouse Cytomegalovirus.

Cocita C, Guiton R, Bessou G, Chasson L, Boyron M, Crozat K, Dalod M.

PLoS Pathog. 2015 May 8;11(5):e1004897. doi: 10.1371/journal.ppat.1004897. eCollection 2015 May.


Functional consequences of natural sequence variation of murine cytomegalovirus m157 for Ly49 receptor specificity and NK cell activation.

Corbett AJ, Coudert JD, Forbes CA, Scalzo AA.

J Immunol. 2011 Feb 1;186(3):1713-22. doi: 10.4049/jimmunol.1003308. Epub 2010 Dec 27.


MHC class I D(k) expression in hematopoietic and nonhematopoietic cells confers natural killer cell resistance to murine cytomegalovirus.

Xie X, Stadnisky MD, Coats ER, Ahmed Rahim MM, Lundgren A, Xu W, Makrigiannis AP, Brown MG.

Proc Natl Acad Sci U S A. 2010 May 11;107(19):8754-9. doi: 10.1073/pnas.0913126107. Epub 2010 Apr 26.


Glycosylation contributes to variability in expression of murine cytomegalovirus m157 and enhances stability of interaction with the NK-cell receptor Ly49H.

Guseva NV, Fullenkamp CA, Naumann PW, Shey MR, Ballas ZK, Houtman JC, Forbes CA, Scalzo AA, Heusel JW.

Eur J Immunol. 2010 Sep;40(9):2618-31. doi: 10.1002/eji.200940134.


Ly49P recognition of cytomegalovirus-infected cells expressing H2-Dk and CMV-encoded m04 correlates with the NK cell antiviral response.

Kielczewska A, Pyzik M, Sun T, Krmpotic A, Lodoen MB, Munks MW, Babic M, Hill AB, Koszinowski UH, Jonjic S, Lanier LL, Vidal SM.

J Exp Med. 2009 Mar 16;206(3):515-23. doi: 10.1084/jem.20080954. Epub 2009 Mar 2.


Ly49C-dependent control of MCMV Infection by NK cells is cis-regulated by MHC Class I molecules.

Forbes CA, Scalzo AA, Degli-Esposti MA, Coudert JD.

PLoS Pathog. 2014 May 29;10(5):e1004161. doi: 10.1371/journal.ppat.1004161. eCollection 2014 May.


Targeting of a natural killer cell receptor family by a viral immunoevasin.

Berry R, Ng N, Saunders PM, Vivian JP, Lin J, Deuss FA, Corbett AJ, Forbes CA, Widjaja JM, Sullivan LC, McAlister AD, Perugini MA, Call MJ, Scalzo AA, Degli-Esposti MA, Coudert JD, Beddoe T, Brooks AG, Rossjohn J.

Nat Immunol. 2013 Jul;14(7):699-705. doi: 10.1038/ni.2605. Epub 2013 May 12.


Ly49C Impairs NK Cell Memory in Mouse Cytomegalovirus Infection.

Forbes CA, Scalzo AA, Degli-Esposti MA, Coudert JD.

J Immunol. 2016 Jul 1;197(1):128-40. doi: 10.4049/jimmunol.1600199. Epub 2016 May 27.


The activating Ly49W and inhibitory Ly49G NK cell receptors display similar affinities for identical MHC class I ligands.

Ma BJ, Craveiro Salvado CM, Kane KP.

Immunogenetics. 2014 Aug;66(7-8):467-77. doi: 10.1007/s00251-014-0777-2. Epub 2014 May 7.


Transgenic expression of the activating natural killer receptor Ly49H confers resistance to cytomegalovirus in genetically susceptible mice.

Lee SH, Zafer A, de Repentigny Y, Kothary R, Tremblay ML, Gros P, Duplay P, Webb JR, Vidal SM.

J Exp Med. 2003 Feb 17;197(4):515-26.


Overcoming innate host resistance to vaccination: employing a genetically distinct strain of murine cytomegalovirus avoids vector-mediated resistance to virally vectored immunocontraception.

Nikolovski S, Lloyd ML, Harvey N, Hardy CM, Shellam GR, Redwood AJ.

Vaccine. 2009 Aug 20;27(38):5226-32. doi: 10.1016/j.vaccine.2009.06.064. Epub 2009 Jul 8.


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