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Items: 1 to 20 of 148

1.

The versatility of Helicobacter pylori CagA effector protein functions: The master key hypothesis.

Backert S, Tegtmeyer N, Selbach M.

Helicobacter. 2010 Jun;15(3):163-76. doi: 10.1111/j.1523-5378.2010.00759.x. Review. Erratum in: Helicobacter. 2010 Oct;15(5):483.

PMID:
20557357
2.

Role of Abl and Src family kinases in actin-cytoskeletal rearrangements induced by the Helicobacter pylori CagA protein.

Tegtmeyer N, Backert S.

Eur J Cell Biol. 2011 Nov;90(11):880-90. doi: 10.1016/j.ejcb.2010.11.006. Epub 2011 Jan 17. Review.

PMID:
21247656
3.

Role of type IV secretion in Helicobacter pylori pathogenesis.

Backert S, Selbach M.

Cell Microbiol. 2008 Aug;10(8):1573-81. doi: 10.1111/j.1462-5822.2008.01156.x. Epub 2008 Apr 8. Review.

PMID:
18410539
4.

Host cell interactome of tyrosine-phosphorylated bacterial proteins.

Selbach M, Paul FE, Brandt S, Guye P, Daumke O, Backert S, Dehio C, Mann M.

Cell Host Microbe. 2009 Apr 23;5(4):397-403. doi: 10.1016/j.chom.2009.03.004.

5.

EPIYA motif is a membrane-targeting signal of Helicobacter pylori virulence factor CagA in mammalian cells.

Higashi H, Yokoyama K, Fujii Y, Ren S, Yuasa H, Saadat I, Murata-Kamiya N, Azuma T, Hatakeyama M.

J Biol Chem. 2005 Jun 17;280(24):23130-7. Epub 2005 Apr 13.

6.

The Helicobacter pylori CagA protein induces cortactin dephosphorylation and actin rearrangement by c-Src inactivation.

Selbach M, Moese S, Hurwitz R, Hauck CR, Meyer TF, Backert S.

EMBO J. 2003 Feb 3;22(3):515-28.

7.

Activation of Abl by Helicobacter pylori: a novel kinase for CagA and crucial mediator of host cell scattering.

Tammer I, Brandt S, Hartig R, König W, Backert S.

Gastroenterology. 2007 Apr;132(4):1309-19. Epub 2007 Feb 1.

PMID:
17408661
8.

c-Src and c-Abl kinases control hierarchic phosphorylation and function of the CagA effector protein in Western and East Asian Helicobacter pylori strains.

Mueller D, Tegtmeyer N, Brandt S, Yamaoka Y, De Poire E, Sgouras D, Wessler S, Torres J, Smolka A, Backert S.

J Clin Invest. 2012 Apr;122(4):1553-66. doi: 10.1172/JCI61143. Epub 2012 Mar 1.

9.

Dual infection system identifies a crucial role for PKA-mediated serine phosphorylation of the EPEC-Tir-injected effector protein in regulating Rac1 function.

Brandt S, Kenny B, Rohde M, Martinez-Quiles N, Backert S.

Cell Microbiol. 2009 Aug;11(8):1254-71. doi: 10.1111/j.1462-5822.2009.01330.x. Epub 2009 Apr 30.

PMID:
19438518
10.

Biological activity of the Helicobacter pylori virulence factor CagA is determined by variation in the tyrosine phosphorylation sites.

Higashi H, Tsutsumi R, Fujita A, Yamazaki S, Asaka M, Azuma T, Hatakeyama M.

Proc Natl Acad Sci U S A. 2002 Oct 29;99(22):14428-33. Epub 2002 Oct 21.

11.

c-Src/Lyn kinases activate Helicobacter pylori CagA through tyrosine phosphorylation of the EPIYA motifs.

Stein M, Bagnoli F, Halenbeck R, Rappuoli R, Fantl WJ, Covacci A.

Mol Microbiol. 2002 Feb;43(4):971-80.

13.

Phosphorylation-independent effects of CagA during interaction between Helicobacter pylori and T84 polarized monolayers.

El-Etr SH, Mueller A, Tompkins LS, Falkow S, Merrell DS.

J Infect Dis. 2004 Oct 15;190(8):1516-23. Epub 2004 Sep 9.

14.

Subversion of host kinases: a key network in cellular signaling hijacked by Helicobacter pylori CagA.

Tegtmeyer N, Neddermann M, Asche CI, Backert S.

Mol Microbiol. 2017 Aug;105(3):358-372. doi: 10.1111/mmi.13707. Epub 2017 May 30. Review.

PMID:
28508421
15.

CagA phosphorylation-dependent MMP-9 expression in gastric epithelial cells.

Nam YH, Ryu E, Lee D, Shim HJ, Lee YC, Lee ST.

Helicobacter. 2011 Aug;16(4):276-83. doi: 10.1111/j.1523-5378.2011.00851.x.

PMID:
21762266
17.

Structural basis and functional consequence of Helicobacter pylori CagA multimerization in cells.

Ren S, Higashi H, Lu H, Azuma T, Hatakeyama M.

J Biol Chem. 2006 Oct 27;281(43):32344-52. Epub 2006 Sep 5.

18.

Interaction between the Helicobacter pylori CagA and alpha-Pix in gastric epithelial AGS cells.

Baek HY, Lim JW, Kim H.

Ann N Y Acad Sci. 2007 Jan;1096:18-23.

PMID:
17405911
19.

Helicobacter pylori CagA containing ITAM-like sequences localized to lipid rafts negatively regulates VacA-induced signaling in vivo.

Asahi M, Tanaka Y, Izumi T, Ito Y, Naiki H, Kersulyte D, Tsujikawa K, Saito M, Sada K, Yanagi S, Fujikawa A, Noda M, Itokawa Y.

Helicobacter. 2003 Feb;8(1):1-14.

PMID:
12603611
20.

Influence of EPIYA-repeat polymorphism on the phosphorylation-dependent biological activity of Helicobacter pylori CagA.

Naito M, Yamazaki T, Tsutsumi R, Higashi H, Onoe K, Yamazaki S, Azuma T, Hatakeyama M.

Gastroenterology. 2006 Apr;130(4):1181-90.

PMID:
16618412

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