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Items: 1 to 20 of 98

1.

Thyroid hormone controls the gene expression of HSV-1 LAT and ICP0 in neuronal cells.

Bedadala GR, Pinnoji RC, Palem JR, Hsia SC.

Cell Res. 2010 May;20(5):587-98. doi: 10.1038/cr.2010.50. Epub 2010 Apr 13.

2.

Regulation of herpes simplex virus type 1 thymidine kinase gene expression by thyroid hormone receptor in cultured neuronal cells.

Hsia SC, Pinnoji RC, Bedadala GR, Hill JM, Palem JR.

J Neurovirol. 2010 Feb;16(1):13-24. doi: 10.3109/13550280903552412.

3.

Increased neurovirulence and reactivation of the herpes simplex virus type 1 latency-associated transcript (LAT)-negative mutant dLAT2903 with a disrupted LAT miR-H2.

Jiang X, Brown D, Osorio N, Hsiang C, BenMohamed L, Wechsler SL.

J Neurovirol. 2016 Feb;22(1):38-49. doi: 10.1007/s13365-015-0362-y. Epub 2015 Jun 12.

4.

CTCF-dependent chromatin boundary element between the latency-associated transcript and ICP0 promoters in the herpes simplex virus type 1 genome.

Chen Q, Lin L, Smith S, Huang J, Berger SL, Zhou J.

J Virol. 2007 May;81(10):5192-201. Epub 2007 Jan 31.

5.

Herpes Simplex Virus 2 Latency-Associated Transcript (LAT) Region Mutations Do Not Identify a Role for LAT-Associated MicroRNAs in Viral Reactivation in Guinea Pig Genital Models.

Kawamura Y, Bosch-Marce M, Tang S, Patel A, Krause PR.

J Virol. 2018 Jun 29;92(14). pii: e00642-18. doi: 10.1128/JVI.00642-18. Print 2018 Jul 15.

PMID:
29720520
6.
9.

The HSV-1 Latency-Associated Transcript Functions to Repress Latent Phase Lytic Gene Expression and Suppress Virus Reactivation from Latently Infected Neurons.

Nicoll MP, Hann W, Shivkumar M, Harman LE, Connor V, Coleman HM, Proença JT, Efstathiou S.

PLoS Pathog. 2016 Apr 7;12(4):e1005539. doi: 10.1371/journal.ppat.1005539. eCollection 2016 Apr.

10.

Thyroid hormone-dependent epigenetic suppression of herpes simplex virus-1 gene expression and viral replication in differentiated neuroendocrine cells.

Figliozzi RW, Chen F, Balish M, Ajavon A, Hsia SV.

J Neurol Sci. 2014 Nov 15;346(1-2):164-73. doi: 10.1016/j.jns.2014.08.017. Epub 2014 Aug 20.

11.

The region of the herpes simplex virus type 1 LAT gene involved in spontaneous reactivation does not encode a functional protein.

Drolet BS, Perng GC, Cohen J, Slanina SM, Yukht A, Nesburn AB, Wechsler SL.

Virology. 1998 Mar 1;242(1):221-32.

12.

A novel mechanism of thyroid hormone-dependent negative regulation by thyroid hormone receptor, nuclear receptor corepressor (NCoR), and GAGA-binding factor on the rat cD44 promoter.

Kim SW, Ho SC, Hong SJ, Kim KM, So EC, Christoffolete M, Harney JW.

J Biol Chem. 2005 Apr 15;280(15):14545-55. Epub 2005 Jan 27.

13.

Epigenetic regulation of latent HSV-1 gene expression.

Bloom DC, Giordani NV, Kwiatkowski DL.

Biochim Biophys Acta. 2010 Mar-Apr;1799(3-4):246-56. doi: 10.1016/j.bbagrm.2009.12.001. Epub 2010 Jan 4. Review.

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The latency-associated transcript gene of herpes simplex virus type 1 (HSV-1) is required for efficient in vivo spontaneous reactivation of HSV-1 from latency.

Perng GC, Dunkel EC, Geary PA, Slanina SM, Ghiasi H, Kaiwar R, Nesburn AB, Wechsler SL.

J Virol. 1994 Dec;68(12):8045-55.

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