Format
Sort by
Items per page

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 101

1.

Regulation of ICP0-null mutant herpes simplex virus type 1 infection by ND10 components ATRX and hDaxx.

Lukashchuk V, Everett RD.

J Virol. 2010 Apr;84(8):4026-40. doi: 10.1128/JVI.02597-09. Epub 2010 Feb 10.

3.
4.

Components of promyelocytic leukemia nuclear bodies (ND10) act cooperatively to repress herpesvirus infection.

Glass M, Everett RD.

J Virol. 2013 Feb;87(4):2174-85. doi: 10.1128/JVI.02950-12. Epub 2012 Dec 5.

5.

Herpes simplex virus is equipped with RNA- and protein-based mechanisms to repress expression of ATRX, an effector of intrinsic immunity.

Jurak I, Silverstein LB, Sharma M, Coen DM.

J Virol. 2012 Sep;86(18):10093-102. doi: 10.1128/JVI.00930-12. Epub 2012 Jul 11.

6.

PML contributes to a cellular mechanism of repression of herpes simplex virus type 1 infection that is inactivated by ICP0.

Everett RD, Rechter S, Papior P, Tavalai N, Stamminger T, Orr A.

J Virol. 2006 Aug;80(16):7995-8005.

7.

Replication of ICP0-null mutant herpes simplex virus type 1 is restricted by both PML and Sp100.

Everett RD, Parada C, Gripon P, Sirma H, Orr A.

J Virol. 2008 Mar;82(6):2661-72. Epub 2007 Dec 26.

8.

Human cytomegalovirus protein pp71 displaces the chromatin-associated factor ATRX from nuclear domain 10 at early stages of infection.

Lukashchuk V, McFarlane S, Everett RD, Preston CM.

J Virol. 2008 Dec;82(24):12543-54. doi: 10.1128/JVI.01215-08. Epub 2008 Oct 15.

9.

MORC3, a Component of PML Nuclear Bodies, Has a Role in Restricting Herpes Simplex Virus 1 and Human Cytomegalovirus.

Sloan E, Orr A, Everett RD.

J Virol. 2016 Sep 12;90(19):8621-33. doi: 10.1128/JVI.00621-16. Print 2016 Oct 1.

10.

Dynamic Response of IFI16 and Promyelocytic Leukemia Nuclear Body Components to Herpes Simplex Virus 1 Infection.

Everett RD.

J Virol. 2015 Oct 14;90(1):167-79. doi: 10.1128/JVI.02249-15. Print 2016 Jan 1.

11.
12.
13.

The viral ubiquitin ligase ICP0 is neither sufficient nor necessary for degradation of the cellular DNA sensor IFI16 during herpes simplex virus 1 infection.

Cuchet-Lourenço D, Anderson G, Sloan E, Orr A, Everett RD.

J Virol. 2013 Dec;87(24):13422-32. doi: 10.1128/JVI.02474-13. Epub 2013 Oct 2.

14.
15.
16.

Analysis of the functional interchange between the IE1 and pp71 proteins of human cytomegalovirus and ICP0 of herpes simplex virus 1.

Lu Y, Everett RD.

J Virol. 2015 Mar;89(6):3062-75. doi: 10.1128/JVI.03480-14. Epub 2014 Dec 31.

17.

Human Daxx-mediated repression of human cytomegalovirus gene expression correlates with a repressive chromatin structure around the major immediate early promoter.

Woodhall DL, Groves IJ, Reeves MB, Wilkinson G, Sinclair JH.

J Biol Chem. 2006 Dec 8;281(49):37652-60. Epub 2006 Oct 11.

18.

Kaposi's sarcoma associated herpesvirus tegument protein ORF75 is essential for viral lytic replication and plays a critical role in the antagonization of ND10-instituted intrinsic immunity.

Full F, Jungnickl D, Reuter N, Bogner E, Brulois K, Scholz B, Stürzl M, Myoung J, Jung JU, Stamminger T, Ensser A.

PLoS Pathog. 2014 Jan;10(1):e1003863. doi: 10.1371/journal.ppat.1003863. Epub 2014 Jan 16.

19.

Identification of three redundant segments responsible for herpes simplex virus 1 ICP0 to fuse with ND10 nuclear bodies.

Zheng Y, Gu H.

J Virol. 2015 Apr;89(8):4214-26. doi: 10.1128/JVI.03658-14. Epub 2015 Jan 28.

20.

Mechanisms of Host IFI16, PML, and Daxx Protein Restriction of Herpes Simplex Virus 1 Replication.

Merkl PE, Orzalli MH, Knipe DM.

J Virol. 2018 Apr 27;92(10). pii: e00057-18. doi: 10.1128/JVI.00057-18. Print 2018 May 15.

Supplemental Content

Support Center