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Items: 1 to 20 of 75

1.

Infection with Toxoplasma gondii increases atherosclerotic lesion in ApoE-deficient mice.

Portugal LR, Fernandes LR, Cesar GC, Santiago HC, Oliveira DR, Silva NM, Silva AA, Lannes-Vieira J, Arantes RM, Gazzinelli RT, Alvarez-Leite JI.

Infect Immun. 2004 Jun;72(6):3571-6.

2.

Murine cytomegalovirus infection increases aortic expression of proatherosclerotic genes.

Burnett MS, Durrani S, Stabile E, Saji M, Lee CW, Kinnaird TD, Hoffman EP, Epstein SE.

Circulation. 2004 Feb 24;109(7):893-7. Epub 2004 Feb 2.

PMID:
14757699
5.
6.
7.

Interleukin-18 enhances atherosclerosis in apolipoprotein E(-/-) mice through release of interferon-gamma.

Whitman SC, Ravisankar P, Daugherty A.

Circ Res. 2002 Feb 8;90(2):E34-8.

PMID:
11834721
8.
10.

Impact of foetus and mother on IFN-gamma-induced indoleamine 2,3-dioxygenase and inducible nitric oxide synthase expression in murine placenta following Toxoplasma gondii infection.

Pfaff AW, Mousli M, Sénégas A, Marcellin L, Takikawa O, Klein JP, Candolfi E.

Int J Parasitol. 2008 Feb;38(2):249-58. Epub 2007 Jul 26.

PMID:
17822706
11.

Aorta of ApoE-deficient mice responds to atherogenic stimuli by a prelesional increase and subsequent decrease in the expression of antioxidant enzymes.

't Hoen PA, Van der Lans CA, Van Eck M, Bijsterbosch MK, Van Berkel TJ, Twisk J.

Circ Res. 2003 Aug 8;93(3):262-9. Epub 2003 Jun 26.

PMID:
12829615
12.

Deficiency in inducible nitric oxide synthase results in reduced atherosclerosis in apolipoprotein E-deficient mice.

Detmers PA, Hernandez M, Mudgett J, Hassing H, Burton C, Mundt S, Chun S, Fletcher D, Card DJ, Lisnock J, Weikel R, Bergstrom JD, Shevell DE, Hermanowski-Vosatka A, Sparrow CP, Chao YS, Rader DJ, Wright SD, Puré E.

J Immunol. 2000 Sep 15;165(6):3430-5.

14.

Overexpression of endothelial nitric oxide synthase accelerates atherosclerotic lesion formation in apoE-deficient mice.

Ozaki M, Kawashima S, Yamashita T, Hirase T, Namiki M, Inoue N, Hirata K, Yasui H, Sakurai H, Yoshida Y, Masada M, Yokoyama M.

J Clin Invest. 2002 Aug;110(3):331-40.

16.

Production of nitric oxide (NO) is not essential for protection against acute Toxoplasma gondii infection in IRF-1-/- mice.

Khan IA, Matsuura T, Fonseka S, Kasper LH.

J Immunol. 1996 Jan 15;156(2):636-43.

PMID:
8543815
17.

The role of IFN-gamma and Toll-like receptors in nephropathy induced by Toxoplasma gondii infection.

Kudo M, Aosai F, Mun HS, Norose K, Akira S, Iwakura Y, Yano A.

Microbiol Immunol. 2004;48(8):617-28.

18.

In the absence of endogenous IL-10, mice acutely infected with Toxoplasma gondii succumb to a lethal immune response dependent on CD4+ T cells and accompanied by overproduction of IL-12, IFN-gamma and TNF-alpha.

Gazzinelli RT, Wysocka M, Hieny S, Scharton-Kersten T, Cheever A, Kühn R, Müller W, Trinchieri G, Sher A.

J Immunol. 1996 Jul 15;157(2):798-805.

PMID:
8752931
19.

Interferon-gamma receptor-deficiency renders mice highly susceptible to toxoplasmosis by decreased macrophage activation.

Deckert-Schlüter M, Rang A, Weiner D, Huang S, Wiestler OD, Hof H, Schlüter D.

Lab Invest. 1996 Dec;75(6):827-41.

PMID:
8973478
20.

Toxoplasma gondii infection in the brain inhibits neuronal degeneration and learning and memory impairments in a murine model of Alzheimer's disease.

Jung BK, Pyo KH, Shin KY, Hwang YS, Lim H, Lee SJ, Moon JH, Lee SH, Suh YH, Chai JY, Shin EH.

PLoS One. 2012;7(3):e33312. doi: 10.1371/journal.pone.0033312. Epub 2012 Mar 21.

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