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Enhanced O-GlcNAc protein modification is associated with insulin resistance in GLUT1-overexpressing muscles.

Buse MG, Robinson KA, Marshall BA, Hresko RC, Mueckler MM.

Am J Physiol Endocrinol Metab. 2002 Aug;283(2):E241-50.


Differential effects of GLUT1 or GLUT4 overexpression on hexosamine biosynthesis by muscles of transgenic mice.

Buse MG, Robinson KA, Marshall BA, Mueckler M.

J Biol Chem. 1996 Sep 20;271(38):23197-202.


Dissociation of GLUT4 translocation and insulin-stimulated glucose transport in transgenic mice overexpressing GLUT1 in skeletal muscle.

Hansen PA, Wang W, Marshall BA, Holloszy JO, Mueckler M.

J Biol Chem. 1998 Jul 17;273(29):18173-9.


Insulin-sensitive regulation of glucose transport and GLUT4 translocation in skeletal muscle of GLUT1 transgenic mice.

Etgen GJ Jr, Zavadoski WJ, Holman GD, Gibbs EM.

Biochem J. 1999 Jan 1;337 ( Pt 1):51-7.


Adipose-specific overexpression of GLUT4 reverses insulin resistance and diabetes in mice lacking GLUT4 selectively in muscle.

Carvalho E, Kotani K, Peroni OD, Kahn BB.

Am J Physiol Endocrinol Metab. 2005 Oct;289(4):E551-61. Epub 2005 May 31.


High glucose and insulin promote O-GlcNAc modification of proteins, including alpha-tubulin.

Walgren JL, Vincent TS, Schey KL, Buse MG.

Am J Physiol Endocrinol Metab. 2003 Feb;284(2):E424-34. Epub 2002 Oct 22.


Altered GLUT1 and GLUT3 gene expression and subcellular redistribution of GLUT4: protein in muscle from patients with acanthosis nigricans and severe insulin resistance.

Stuart CA, Wen G, Williamson ME, Jiang J, Gilkison CR, Blackwell SJ, Nagamani M, Ferrando AA.

Metabolism. 2001 Jul;50(7):771-7.


Expression of the glucose transporter GLUT4 in the muscular dystrophic mdx mouse.

Olichon-Berthe C, Gautier N, Van Obberghen E, Le Marchand-Brustel Y.

Biochem J. 1993 Apr 1;291 ( Pt 1):257-61.


Calpain system regulates muscle mass and glucose transporter GLUT4 turnover.

Otani K, Han DH, Ford EL, Garcia-Roves PM, Ye H, Horikawa Y, Bell GI, Holloszy JO, Polonsky KS.

J Biol Chem. 2004 May 14;279(20):20915-20. Epub 2004 Mar 9.


GLUT4 overexpression in db/db mice dose-dependently ameliorates diabetes but is not a lifelong cure.

Brozinick JT Jr, McCoid SC, Reynolds TH, Nardone NA, Hargrove DM, Stevenson RW, Cushman SW, Gibbs EM.

Diabetes. 2001 Mar;50(3):593-600.


Insulin-independent glucose transport regulates insulin sensitivity.

Ebeling P, Koistinen HA, Koivisto VA.

FEBS Lett. 1998 Oct 9;436(3):301-3. Review.


Glucosamine-induced insulin resistance is coupled to O-linked glycosylation of Munc18c.

Chen G, Liu P, Thurmond DC, Elmendorf JS.

FEBS Lett. 2003 Jan 16;534(1-3):54-60.


C-terminal mutations that alter the turnover number for 3-O-methylglucose transport by GLUT1 and GLUT4.

Dauterive R, Laroux S, Bunn RC, Chaisson A, Sanson T, Reed BC.

J Biol Chem. 1996 May 10;271(19):11414-21.


Mechanisms of insulin-dependent glucose transport into porcine and bovine skeletal muscle.

Duhlmeier R, Hacker A, Widdel A, von Engelhardt W, Sallmann HP.

Am J Physiol Regul Integr Comp Physiol. 2005 Jul;289(1):R187-97. Epub 2005 Apr 7.


The tissue concentration of UDP-N-acetylglucosamine modulates the stimulatory effect of insulin on skeletal muscle glucose uptake.

Hawkins M, Angelov I, Liu R, Barzilai N, Rossetti L.

J Biol Chem. 1997 Feb 21;272(8):4889-95.


Indinavir uncovers different contributions of GLUT4 and GLUT1 towards glucose uptake in muscle and fat cells and tissues.

Rudich A, Konrad D, Török D, Ben-Romano R, Huang C, Niu W, Garg RR, Wijesekara N, Germinario RJ, Bilan PJ, Klip A.

Diabetologia. 2003 May;46(5):649-58. Epub 2003 Apr 24.


Mechanisms regulating GLUT4 glucose transporter expression and glucose transport in skeletal muscle.

Zorzano A, Palacín M, Gumà A.

Acta Physiol Scand. 2005 Jan;183(1):43-58. Review.


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