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A proinflammatory peptide from Helicobacter pylori activates monocytes to induce lymphocyte dysfunction and apoptosis.

Betten A, Bylund J, Christophe T, Boulay F, Romero A, Hellstrand K, Dahlgren C.

J Clin Invest. 2001 Oct;108(8):1221-8. Erratum in: J Clin Invest. 2006 May;116(5):1457. Cristophe, T [corrected to Christophe, T].


Proinflammatory activity of a cecropin-like antibacterial peptide from Helicobacter pylori.

Bylund J, Christophe T, Boulay F, Nyström T, Karlsson A, Dahlgren C.

Antimicrob Agents Chemother. 2001 Jun;45(6):1700-4.


Basophils infiltrate human gastric mucosa at sites of Helicobacter pylori infection, and exhibit chemotaxis in response to H. pylori-derived peptide Hp(2-20).

de Paulis A, Prevete N, Fiorentino I, Walls AF, Curto M, Petraroli A, Castaldo V, Ceppa P, Fiocca R, Marone G.

J Immunol. 2004 Jun 15;172(12):7734-43.


A proinflammatory peptide from herpes simplex virus type 2 glycoprotein G affects neutrophil, monocyte, and NK cell functions.

Bellner L, Thorén F, Nygren E, Liljeqvist JA, Karlsson A, Eriksson K.

J Immunol. 2005 Feb 15;174(4):2235-41.


Helicobacter pylori Hp(2-20) promotes migration and proliferation of gastric epithelial cells by interacting with formyl peptide receptors in vitro and accelerates gastric mucosal healing in vivo.

de Paulis A, Prevete N, Rossi FW, Rivellese F, Salerno F, Delfino G, Liccardo B, Avilla E, Montuori N, Mascolo M, Staibano S, Melillo RM, D'Argenio G, Ricci V, Romano M, Marone G.

J Immunol. 2009 Sep 15;183(6):3761-9. doi: 10.4049/jimmunol.0900863. Epub 2009 Aug 19.


Natural killer cell receptor T-lymphocytes in normal and Helicobacter pylori-infected human gastric mucosa.

O'Keeffe J, Gately CM, O'Donoghue Y, Zulquernain SA, Stevens FM, Moran AP.

Helicobacter. 2008 Dec;13(6):500-5. doi: 10.1111/j.1523-5378.2008.00641.x.


Soluble surface proteins from Helicobacter pylori activate monocytes/macrophages by lipopolysaccharide-independent mechanism.

Mai UE, Perez-Perez GI, Wahl LM, Wahl SM, Blaser MJ, Smith PD.

J Clin Invest. 1991 Mar;87(3):894-900.


The effect of Helicobacter pylori infection on expression of macrophage migration inhibitory factor by T cells and macrophages in gastric mucosa.

He XX, Yang J, Zheng XL, Ding YW, Shen QY, Liu W, Zhao YH.

Chin Med J (Engl). 2005 Jul 20;118(14):1201-5.


[What are the gastric modifications induced by acute and chronic Helicobacter pylori infection?].

Lamarque D, Tran Van Nhieu J, Breban M.

Gastroenterol Clin Biol. 2003 Mar;27(3 Pt 2):391-400. Review. French.


The neutrophil-activating protein (HP-NAP) of Helicobacter pylori is a protective antigen and a major virulence factor.

Satin B, Del Giudice G, Della Bianca V, Dusi S, Laudanna C, Tonello F, Kelleher D, Rappuoli R, Montecucco C, Rossi F.

J Exp Med. 2000 May 1;191(9):1467-76.


Gene expression profiling in human gastric mucosa infected with Helicobacter pylori.

Hofman VJ, Moreilhon C, Brest PD, Lassalle S, Le Brigand K, Sicard D, Raymond J, Lamarque D, Hébuterne XA, Mari B, Barbry PJ, Hofman PM.

Mod Pathol. 2007 Sep;20(9):974-89. Epub 2007 Jul 20.


Activation of the chemotactic peptide receptor FPRL1 in monocytes phosphorylates the chemokine receptor CCR5 and attenuates cell responses to selected chemokines.

Shen W, Proost P, Li B, Gong W, Le Y, Sargeant R, Murphy PM, Van Damme J, Wang JM.

Biochem Biophys Res Commun. 2000 May 27;272(1):276-83.


The neutrophil-activating protein of Helicobacter pylori.

Dundon WG, Nishioka H, Polenghi A, Papinutto E, Zanotti G, Montemurro P, Del GG, Rappuoli R, Montecucco C.

Int J Med Microbiol. 2002 Feb;291(6-7):545-50. Review.


gammadelta T cells increase with gastric mucosal interleukin (IL)-7, IL-1beta, and Helicobacter pylori urease specific immunoglobulin levels via CCR2 upregulation in Helicobacter pylori gastritis.

Futagami S, Hiratsuka T, Suzuki K, Kusunoki M, Wada K, Miyake K, Ohashi K, Shimizu M, Takahashi H, Gudis K, Kato S, Tsukui T, Sakamoto C.

J Gastroenterol Hepatol. 2006 Jan;21(1 Pt 1):32-40.


Additive effects of Helicobacter pylori lipopolysaccharide and proteins in monocyte inflammatory responses.

Hansen PS, Petersen SB, Varning K, Nielsen H.

Scand J Gastroenterol. 2002 Jul;37(7):765-71.


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