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Mice lacking the chemokine receptor CCR1 show increased susceptibility to Toxoplasma gondii infection.

Khan IA, Murphy PM, Casciotti L, Schwartzman JD, Collins J, Gao JL, Yeaman GR.

J Immunol. 2001 Feb 1;166(3):1930-7.


Cutting edge: MyD88 is required for resistance to Toxoplasma gondii infection and regulates parasite-induced IL-12 production by dendritic cells.

Scanga CA, Aliberti J, Jankovic D, Tilloy F, Bennouna S, Denkers EY, Medzhitov R, Sher A.

J Immunol. 2002 Jun 15;168(12):5997-6001.


CCR1 and CC chemokine ligand 5 interactions exacerbate innate immune responses during sepsis.

Ness TL, Carpenter KJ, Ewing JL, Gerard CJ, Hogaboam CM, Kunkel SL.

J Immunol. 2004 Dec 1;173(11):6938-48.


A role for inducible costimulator protein in the CD28- independent mechanism of resistance to Toxoplasma gondii.

Villegas EN, Lieberman LA, Mason N, Blass SL, Zediak VP, Peach R, Horan T, Yoshinaga S, Hunter CA.

J Immunol. 2002 Jul 15;169(2):937-43.


IP-10 is critical for effector T cell trafficking and host survival in Toxoplasma gondii infection.

Khan IA, MacLean JA, Lee FS, Casciotti L, DeHaan E, Schwartzman JD, Luster AD.

Immunity. 2000 May;12(5):483-94.


Both lymphotoxin-alpha and TNF are crucial for control of Toxoplasma gondii in the central nervous system.

Schlüter D, Kwok LY, Lütjen S, Soltek S, Hoffmann S, Körner H, Deckert M.

J Immunol. 2003 Jun 15;170(12):6172-82.


In vivo study of toxoplasmic parasitemia using interferon-gamma-deficient mice: absolute cell number of leukocytes, parasite load and cell susceptibility.

Norose K, Naoi K, Fang H, Yano A.

Parasitol Int. 2008 Dec;57(4):447-53. doi: 10.1016/j.parint.2008.05.007. Epub 2008 Jun 8.


Rapid recruitment of neutrophils containing prestored IL-12 during microbial infection.

Bliss SK, Butcher BA, Denkers EY.

J Immunol. 2000 Oct 15;165(8):4515-21.


The C10/CCL6 chemokine and CCR1 play critical roles in the pathogenesis of IL-13-induced inflammation and remodeling.

Ma B, Zhu Z, Homer RJ, Gerard C, Strieter R, Elias JA.

J Immunol. 2004 Feb 1;172(3):1872-81.


Respiratory syncytial virus-induced exaggeration of allergic airway disease is dependent upon CCR1-associated immune responses.

John AE, Gerard CJ, Schaller M, Miller AL, Berlin AA, Humbles AA, Lukacs NW.

Eur J Immunol. 2005 Jan;35(1):108-16. Erratum in: Eur J Immunol. 2005 Oct;35(10):3083.


Chemokine receptors Ccr1, Ccr2, and Ccr5 mediate neutrophil migration to postischemic tissue.

Reichel CA, Khandoga A, Anders HJ, Schlöndorff D, Luckow B, Krombach F.

J Leukoc Biol. 2006 Jan;79(1):114-22. Epub 2005 Nov 7.


Deletion of CCR1 attenuates pathophysiologic responses during respiratory syncytial virus infection.

Miller AL, Gerard C, Schaller M, Gruber AD, Humbles AA, Lukacs NW.

J Immunol. 2006 Feb 15;176(4):2562-7.


The human specific CCR1 antagonist CP-481,715 inhibits cell infiltration and inflammatory responses in human CCR1 transgenic mice.

Gladue RP, Cole SH, Roach ML, Tylaska LA, Nelson RT, Shepard RM, McNeish JD, Ogborne KT, Neote KS.

J Immunol. 2006 Mar 1;176(5):3141-8.

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