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Items: 4

1.

Shotgun brain proteomics reveals early molecular signature in presymptomatic mouse model of Alzheimer's disease.

Yang H, Wittnam JL, Zubarev RA, Bayer TA.

J Alzheimers Dis. 2013;37(2):297-308. doi: 10.3233/JAD-130476.

PMID:
24018289
2.

N-truncated amyloid β (Aβ) 4-42 forms stable aggregates and induces acute and long-lasting behavioral deficits.

Bouter Y, Dietrich K, Wittnam JL, Rezaei-Ghaleh N, Pillot T, Papot-Couturier S, Lefebvre T, Sprenger F, Wirths O, Zweckstetter M, Bayer TA.

Acta Neuropathol. 2013 Aug;126(2):189-205. doi: 10.1007/s00401-013-1129-2. Epub 2013 May 18.

3.

Pyroglutamate amyloid β (Aβ) aggravates behavioral deficits in transgenic amyloid mouse model for Alzheimer disease.

Wittnam JL, Portelius E, Zetterberg H, Gustavsson MK, Schilling S, Koch B, Demuth HU, Blennow K, Wirths O, Bayer TA.

J Biol Chem. 2012 Mar 9;287(11):8154-62. doi: 10.1074/jbc.M111.308601. Epub 2012 Jan 20.

4.

Altered histone acetylation is associated with age-dependent memory impairment in mice.

Peleg S, Sananbenesi F, Zovoilis A, Burkhardt S, Bahari-Javan S, Agis-Balboa RC, Cota P, Wittnam JL, Gogol-Doering A, Opitz L, Salinas-Riester G, Dettenhofer M, Kang H, Farinelli L, Chen W, Fischer A.

Science. 2010 May 7;328(5979):753-6. doi: 10.1126/science.1186088. Erratum in: Science. 2010 Jun 25;328(5986):1634.

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