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Items: 14


Complex Regulation of the Pericellular Proteolytic Microenvironment during Tumor Progression and Wound Repair: Functional Interactions between the Serine Protease and Matrix Metalloproteinase Cascades.

Wilkins-Port CE, Higgins SP, Higgins CE, Kobori-Hotchkiss I, Higgins PJ.

Biochem Res Int. 2012;2012:454368. doi: 10.1155/2012/454368. Epub 2012 Feb 20.


PAI-1: An Integrator of Cell Signaling and Migration.

Czekay RP, Wilkins-Port CE, Higgins SP, Freytag J, Overstreet JM, Klein RM, Higgins CE, Samarakoon R, Higgins PJ.

Int J Cell Biol. 2011;2011:562481. doi: 10.1155/2011/562481. Epub 2011 Aug 3.


PAI-1 mediates the TGF-beta1+EGF-induced "scatter" response in transformed human keratinocytes.

Freytag J, Wilkins-Port CE, Higgins CE, Higgins SP, Samarakoon R, Higgins PJ.

J Invest Dermatol. 2010 Sep;130(9):2179-90. doi: 10.1038/jid.2010.106. Epub 2010 Apr 29.


TGF-beta1 + EGF-initiated invasive potential in transformed human keratinocytes is coupled to a plasmin/MMP-10/MMP-1-dependent collagen remodeling axis: role for PAI-1.

Wilkins-Port CE, Ye Q, Mazurkiewicz JE, Higgins PJ.

Cancer Res. 2009 May 1;69(9):4081-91. doi: 10.1158/0008-5472.CAN-09-0043. Epub 2009 Apr 21.


TGF-β1-Induced Expression of the Anti-Apoptotic PAI-1 Protein Requires EGFR Signaling.

Higgins SP, Samarakoon R, Higgins CE, Freytag J, Wilkins-Port CE, Higgins PJ.

Cell Commun Insights. 2009;2:1-11.


PAI-1 Regulates the Invasive Phenotype in Human Cutaneous Squamous Cell Carcinoma.

Freytag J, Wilkins-Port CE, Higgins CE, Carlson JA, Noel A, Foidart JM, Higgins SP, Samarakoon R, Higgins PJ.

J Oncol. 2009;2009:963209. doi: 10.1155/2009/963209. Epub 2010 Mar 1.


SERPINE1 (PAI-1) is deposited into keratinocyte migration "trails" and required for optimal monolayer wound repair.

Providence KM, Higgins SP, Mullen A, Battista A, Samarakoon R, Higgins CE, Wilkins-Port CE, Higgins PJ.

Arch Dermatol Res. 2008 Jul;300(6):303-10. doi: 10.1007/s00403-008-0845-2. Epub 2008 Apr 2.


SERPINE1 (PAI-1) is a prominent member of the early G0 --> G1 transition "wound repair" transcriptome in p53 mutant human keratinocytes.

Qi L, Higgins SP, Lu Q, Samarakoon R, Wilkins-Port CE, Ye Q, Higgins CE, Staiano-Coico L, Higgins PJ.

J Invest Dermatol. 2008 Mar;128(3):749-53. Epub 2007 Sep 20. No abstract available.


PAI-1 is a Critical Upstream Regulator of the TGF-beta1/EGF-Induced Invasive Phenotype in Mutant p53 Human Cutaneous Squamous Cell Carcinoma.

Wilkins-Port CE, Higgins CE, Freytag J, Higgins SP, Carlson JA, Higgins PJ.

J Biomed Biotechnol. 2007;2007(2):85208. Epub 2007 Mar 20.


Vitronectin's basic domain is a syndecan ligand which functions in trans to regulate vitronectin turnover.

Wilkins-Port CE, Sanderson RD, Tominna-Sebald E, McKeown-Longo PJ.

Cell Commun Adhes. 2003 Mar-Apr;10(2):85-103.


Localization of urokinase type plasminogen activator to focal adhesions requires ligation of vitronectin integrin receptors.

Wilcox-Adelman SA, Wilkins-Port CE, McKeown-Longo PJ.

Cell Adhes Commun. 2000;7(6):477-90.


Degradation of distinct forms of multimeric vitronectin by human fibroblasts.

Wilkins-Port CE, McKeown-Longo PJ.

Biochim Biophys Acta. 1998 Sep 16;1404(3):353-66.


Heparan sulfate proteoglycans function in the binding and degradation of vitronectin by fibroblast monolayers.

Wilkins-Port CE, McKeown-Longo PJ.

Biochem Cell Biol. 1996;74(6):887-97.


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